“…OBJECTIVE: Stosstherapy has been used since early 19(th) century for treating nutritional rickets. However, there are no clear cut guidelines for the biochemical monitoring of this treatment. …”
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“…We included children aged 0 to 15 years diagnosed with vitamin D deficiency or vitamin D–deficient rickets; those with congenital/secondary rickets and low-birth-weight infants were excluded. …”
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“…The prevalence of nutritional rickets is increasing in high-income countries, largely driven by an influx of immigrant populations. …”
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“…Modern lifestyle limits our exposure to sunlight, which photosynthesizes vitamin D in the skin, and the incidence of nutritional rickets has been resurging. Vitamin D is one of the first hormones; it is photosynthesized in all organism from the phytoplankton to mammals. …”
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“…Since first described almost a century ago, vitamin D preparations have been successfully used as a public health intervention to prevent nutritional rickets. In this manuscript, we document the periods in history when nutritional rickets was described, examine early efforts to understand its etiology and the steps taken to treat and prevent it. …”
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“…Here, we discuss the management of different forms of rickets, including new therapeutic approaches based on recent guidelines. …”
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“…Young rats on a diet low in phosphorus can be protected from rickets by irradiations with sunlight for about 15 minutes daily. …”
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“…A simple diet is presented which regularly induced rickets in young rats. 2. The substitution of 0.4 per cent secondary potassium phosphate for a small part of calcium lactate in this diet completely inhibited the development of rickets. 3. …”
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“…Vitamin D deficiency rickets (VDDR) is a disorder biochemically characterized by elevated serum alkaline phosphatase (ALP) activity, normal or decreased serum calcium (Ca) and inorganic phosphate concentrations, secondary hyperparathyroidism and decreased serum 25−hydroxyvitamin D (25(OH)D) levels. …”
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“…Renal synthesis of 1,25(OH)(2)D from its endogenous precursor, 25-hydroxyvitamin D (25OHD), is the rate-limiting and is catalyzed by the 1α-hydroxylase. Vitamin D dependent rickets type I (VDDR-I), also referred to as vitamin D 1α-hydroxylase deficiency or pseudovitamin D deficiency rickets, is an autosomal recessive disorder characterized clinically by hypotonia, muscle weakness, growth failure, hypocalcemic seizures in early infancy, and radiographic findings of rickets. …”
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“…We wish to report two cases of rickets due to vitamin D deficiency secondary to underlying ichthyotic skin disorder. …”
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“…We report here, a rare case of hypophosphatemic rickets associated with a giant hairy nevus in a 10-year-old boy.…”
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“…We present the case of a preterm 6-month-old African American infant who developed craniosynostosis secondary to rickets. This child developed rickets and macrocephaly by the age of 6 months. …”
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“…BACKGROUND AND OBJECTIVES: Rickets is commonly seen as a sign of malabsorption like celiac disease if it is not treated appropriately with vitamin D and calcium supplements. …”
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