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  1. 2641
    “…Here, we present the first identified CaM and S100A1 binding sites at the N-terminal of TRPM6. We have confirmed the L520-R535 N-terminal TRPM6 domain as a shared binding site for CaM and S100A1 using biophysical and molecular modelling methods. …”
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    TRPM6 N-Terminal CaM- and S100A1-Binding Domains
  2. 2642
    “…[Image: see text] A ligand-promoted iridium-catalyzed transfer hydrogenation of terminal alkynes with ethanol and its application has been developed. …”
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    Ligand-Promoted Iridium-Catalyzed Transfer Hydrogenation of Terminal Alkynes with Ethanol and Its Application
  3. 2643
    “…For 10% of patients with a genetic disease, a nonsense/STOP mutation/premature termination codon (PTC) is the underlying cause of their malady. …”
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    2348 Lafora disease premature termination codons (PTCs) are likely candidates for suppression by aminoglycosides
  4. 2644
    “…Conversely, one study in Israel showed higher termination rates among patients who did not undergo amniocentesis vs. those who did (35.5% vs. 7.3%). …”
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    1780. Congenital Cytomegalovirus: What Are the Rates of Maternal Screening, Diagnostic Amniocentesis, and Elective Termination?
  5. 2645
    “…In the present study, we found that the expression of deubiquitylase USP7 is significantly increased during human terminal erythroid differentiation. We further showed that interfering with USP7 function, either by short hairpin RNA-mediated knockdown or USP7-specific inhibitors, impaired human terminal erythroid differentiation due to decreased GATA1 level and that restoration of GATA1 levels rescued the differentiation defect. …”
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    Deubiquitylase USP7 regulates human terminal erythroid differentiation by stabilizing GATA1
  6. 2646
    “…We found that the control of flower termination was not as robust as classically pictured. …”
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    Paf1c defects challenge the robustness of flower meristem termination in Arabidopsis thaliana
  7. 2647
    “…NUCC-390 was tested for its capability to induce the regeneration of motor axon terminals completely degenerated by the presynaptic neurotoxin α-Latrotoxin. …”
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    An Agonist of the CXCR4 Receptor Strongly Promotes Regeneration of Degenerated Motor Axon Terminals
  8. 2648
    “…The chance of death with a terminal drop decreased: 22% if no CD, 20% if 1 CD, 14% if 2 CD, 11% if >2 CD. …”
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    CHRONIC DISEASE AND TERMINAL DECLINE IN VERY OLD MEN: THE MANITOBA FOLLOW-UP STUDY
  9. 2649
    por Lee, Sungmin
    Publicado 2018
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    Termination of Idiopathic Sustained Monomorphic Ventricular Tachycardia by Synchronized Electrical Cardioversion during Pregnancy
  10. 2650
    por Voegeli, R., Rawlings, A.V., Haftek, M.
    Publicado 2019
    “…RESULT: Plasmin was only observed in the terminally differentiated cell layers of the epidermis and was largely associated with the corneocyte envelopes and to some extent with the intercellular lipid matrix in the stratum corneum. …”
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    Expression and ultrastructural localization of plasmin(ogen) in the terminally differentiated layers of normal human epidermis
  11. 2651
    “…Recent studies of muscle synapses (endplates) show that there are also terminal Schwann cells (tSCs), glial cells associated with motor neurons and their functions, and vacant sites (or vacancies) devoid of tSCs and axons proposing tSCs as key effectors of synapse elimination. …”
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    Terminal Schwann cell and vacant site mediated synapse elimination at developing neuromuscular junctions
  12. 2652
    “…BACKGROUND: The objective of this study was to develop a strategy to optimize medical health surveillance protocols for administrative employees using video display terminals (VDTs). A total of 2453 medical examinations were analysed for VDT users in various sectors. …”
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    A comparative analysis of health surveillance strategies for administrative video display terminal employees
  13. 2653
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    Respecting Autonomy and Promoting the Patient’s Good in the Setting of Serious Terminal and Concurrent Mental Illness
  14. 2654
    “…Here we report that during the final stages of migration, transcription factor Neurogenic Differentiation 2 (Neurod2) contributes to terminal cellular localization within the cortical plate. …”
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    Terminal neuron localization to the upper cortical plate is controlled by the transcription factor NEUROD2
  15. 2655
    “…Here, we investigated the role of the C-terminal region in OaPAC, the length of which is the only notable difference from bPAC. …”
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    The C-terminal region affects the activity of photoactivated adenylyl cyclase from Oscillatoria acuminata
  16. 2656
    “…The allosteric and torpedo models have been used for 30 yr to explain how transcription terminates on protein-coding genes. The former invokes termination via conformational changes in the transcription complex and the latter proposes that degradation of the downstream product of poly(A) signal (PAS) processing is important. …”
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    A unified allosteric/torpedo mechanism for transcriptional termination on human protein-coding genes
  17. 2657
    “…To elucidate the molecular mechanisms underlying the dysfunction of these synapses, we performed an explorative proteomic study of the dentate terminal zone of the perforant path. The outer two-thirds of the molecular layer of the dentate gyrus, where the perforant path synapses are located, was microdissected from five subjects with AD and five controls. …”
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    The Proteome of the Dentate Terminal Zone of the Perforant Path Indicates Presynaptic Impairment in Alzheimer Disease
  18. 2658
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    Antibiotic production in Streptomyces is organized by a division of labor through terminal genomic differentiation
  19. 2659
    “…Despite extensive studies on transcription mechanisms, it is unknown how termination complexes are disassembled, especially in what order the essential components dissociate. …”
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    Transcription reinitiation by recycling RNA polymerase that diffuses on DNA after releasing terminated RNA
  20. 2660
    “…Previous studies have reported that the regulatory function of the different c-Jun N-terminal kinases isoforms (JNK1, JNK2, and JNK3) play an essential role in neurological disorders, such as epilepsy and metabolic-cognitive alterations. …”
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    Role of c-Jun N-Terminal Kinases (JNKs) in Epilepsy and Metabolic Cognitive Impairment
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