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  1. 1721
    “…mRNAs containing premature termination codons (PTCs) are known to be degraded via nonsense-mediated mRNA decay (NMD). …”
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    Premature termination codons are recognized in the nucleus in a reading-frame-dependent manner
  2. 1722
    por Bryan, Nadezda, Raisch, Kevin P.
    Publicado 2015
    “…Therefore, a series of truncations and point mutations were made to the N-terminal end of HKII to identify the binding site to VDAC. …”
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    Identification of a mitochondrial-binding site on the N-terminal end of hexokinase II
  3. 1723
    “…Termination of protein synthesis is not 100% efficient. …”
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    Translational readthrough potential of natural termination codons in eucaryotes – The impact of RNA sequence
  4. 1724
    “…We show that BC derivatives migrate along peripheral nerves to reach the skin, where they give rise to terminal glia associated with dermal nerve endings. …”
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    Boundary Caps Give Rise to Neurogenic Stem Cells and Terminal Glia in the Skin
  5. 1725
    “…BACKGROUND: Eukaryotic RNA polymerase II contains a C-terminal repeated domain (CTD) consisting of 52 consensus heptad repeats of Y(1)S(2)P(3)T(4)S(5)P(6)S(7) that mediate interactions with many cellular proteins to regulate transcription elongation, RNA processing and chromatin structure. …”
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    Proteomics studies of the interactome of RNA polymerase II C-terminal repeated domain
  6. 1726
    “…By analyzing a series of D2R constructs containing mutations in cysteine residues, we found that palmitoylation of the D2R most likely occurs on the C-terminal cysteine residue (C443) of the polypeptide. …”
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    Effect of C-Terminal S-Palmitoylation on D2 Dopamine Receptor Trafficking and Stability
  7. 1727
    “…As seizures progress, topographical and geometrical changes in network connectivity strengthen and tighten synchronous connectivity near foci—a mechanism that may aid seizure termination. Collectively, our observations implicate distributed cortical structures in seizure generation, propagation and termination, and may have practical significance in determining which circuits to modulate with implantable devices.…”
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    Dynamic Network Drivers of Seizure Generation, Propagation and Termination in Human Neocortical Epilepsy
  8. 1728
    por Buckley, Rachael M., Stubbe, JoAnne
    Publicado 2015
    “…Comparison of the relative rate constants of thiolysis by CoA and elongation by PhaC(Cc), and the size of the PHB polymer generated in vivo, suggests a mechanism for chain termination and reinitiation.…”
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    Chemistry with an Artificial Primer of Polyhydroxybutyrate Synthase Suggests a Mechanism for Chain Termination
  9. 1729
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    Extending computational models of astrocyte-neuron interactions with biochemical mechanisms on the postsynaptic terminal
  10. 1730
    “…BACKGROUND: Long terminal repeat (LTR)-retrotransposons constitute 42.4 % of the genome of the ‘Suli’ pear (Pyrus pyrifolia white pear group), implying that retrotransposons have played important roles in Pyrus evolution. …”
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    Isolation and characterization of putative functional long terminal repeat retrotransposons in the Pyrus genome
  11. 1731
    “…The histone H3 variant (H3.V) and an alternate nucleotide, base J (ß-D-glucosyl-hydroxymethyluracil), are two chromatin marks found at both transcription termination sites (TTSs) and telomeres. Here, we report that the absence of both base J and H3.V result in transcription readthrough and the appearance of antisense transcripts near TTSs. …”
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    Base J and H3.V Regulate Transcriptional Termination in Trypanosoma brucei
  12. 1732
    “…A number of calcium clearance mechanisms are present in central nerve terminals that regulate intracellular free calcium levels both during and after stimulation. …”
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    Mitochondrial Calcium Uptake Modulates Synaptic Vesicle Endocytosis in Central Nerve Terminals
  13. 1733
    por Marcianò, G., Huang, D. T.
    Publicado 2016
    “…FACT is a heterodimeric complex consisting of Spt16 and SSRP1. The N-terminal domain of Spt16 resembles an inactive aminopeptidase. …”
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    Structure of the human histone chaperone FACT Spt16 N-terminal domain
  14. 1734
    “…However, at high NaCl concentrations I-state gains substantial compaction, and the unfolding barrier is strongly affected, revealing the importance of electrostatics in the folding mechanism. The role of the C-terminal region (CTR), the key determinant of frataxin stability, was also studied. …”
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    Human Frataxin Folds Via an Intermediate State. Role of the C-Terminal Region
  15. 1735
    por Newell-Litwa, Karen A.
    Publicado 2016
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    Breaking down to build up: Neuroligin’s C-terminal domain strengthens the synapse
  16. 1736
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    Cytoskeletal Linker Protein Dystonin Is Not Critical to Terminal Oligodendrocyte Differentiation or CNS Myelination
  17. 1737
    “…Mechanistically, these studies reveal that a proteolytically released fragment of this collagen, termed a matricryptin, promotes the assembly of inhibitory nerve terminals through integrin receptors. Collectively, these studies not only identify roles for collagen-derived matricryptins in cortical circuit formation, but they also reveal a novel paracrine mechanism that regulates the assembly of these synapses.…”
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    Collagen-derived matricryptins promote inhibitory nerve terminal formation in the developing neocortex
  18. 1738
    “…In E. coli respiratory mutants, both O(2)-consumption and aerobic growth proved to be severely impaired by sulfide when respiration was sustained by the bo(3) oxidase alone, but unaffected by ≤200 μM sulfide when either bd enzyme acted as the only terminal oxidase. Accordingly, wild-type E. coli showed sulfide-insensitive respiration and growth under conditions favouring the expression of bd oxidases. …”
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    The Terminal Oxidase Cytochrome bd Promotes Sulfide-resistant Bacterial Respiration and Growth
  19. 1739
    “…CONCLUSIONS: NLR appears to be a useful and simple parameter to predict the clinical outcomes of patients with terminal cancer.…”
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    Neutrophil/lymphocyte ratio has a prognostic value for patients with terminal cancer
  20. 1740
    “…We identified key differences in the encoding of reward and choice in dopamine terminals in dorsal versus ventral striatum: DA terminals in ventral striatum responded more strongly to reward consumption and reward-predicting cues, whereas DA terminals in dorsomedial striatum responded more strongly to contralateral choices. …”
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    Reward and choice encoding in terminals of midbrain dopamine neurons depends on striatal target
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