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  1. 461
    “…We conclude that the ΔDMR1-U2 deletion phenotype should be reconsidered in the light of a functional Igf2as gene.…”
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    Functional Expression Study of Igf2 Antisense Transcript in Mouse
  2. 462
    por Moreno, J.M., Oaknin, D.H., Quiros, M.
    Publicado 1996
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    Squarks and sphalerons
  3. 463
    “…The wound healing assay was performed to assess the migratory ability of U2OS and MG63 cells. Antitumor effects of miR-382-5p were evaluated in nude mice xenografts using U2OS cells. …”
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    MicroRNA-382-5p inhibits osteosarcoma development and progression by negatively regulating VEZF1 expression
  4. 464
    “…In higher eukaryotes, U1 snRNP forms spliceosomes in equal stoichiometry with U2, U4, U5 and U6, however its abundance far exceeds that of the other snRNPs. …”
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    U1 snRNP protects pre-mRNAs from premature cleavage and polyadenylation
  5. 465
    “…The ectopic TTRAP suppressed the growth and colony formation capacity of two osteosarcoma cell lines, U2OS and Saos-2. Cell apoptosis was induced in U2OS cells and the cell cycle was arrested at G2/M phase in Saos-2 cells. …”
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    Overexpression of TTRAP inhibits cell growth and induces apoptosis in osteosarcoma cells
  6. 466
    “…Hyperthermia can induce cell death in various cancer cells, and thus, in this study, we investigated the anticancer method of hyperthermia in human OS (U-2 OS) cells. Treatment at 43 °C for 60 min induced apoptosis in human OS cell lines, but not in primary bone cells. …”
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    Hyperthermia Induces Apoptosis through Endoplasmic Reticulum and Reactive Oxygen Species in Human Osteosarcoma Cells
  7. 467
    “…CTCF knockdown also causes a termination defect on the U2 snRNA genes (U2), by affecting recruitment of negative elongation factor (NELF). …”
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    CTCF regulates NELF, DSIF and P-TEFb recruitment during transcription
  8. 468
    “…We found that PTBP1 binding in the flanking introns allowed normal U2AF and U1 snRNP binding to the target exon splice sites but blocked U2 snRNP assembly in HeLa nuclear extract. …”
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    Large-scale remodeling of a repressed exon ribonucleoprotein to an exon definition complex active for splicing
  9. 469
    “…This study investigates the impact of celastrol on the expression of death receptors 4/5 (DR4/5) on OS cell lines (HOS, U2OS) and cancer cell lysis by γδ T cells. The results showed that celastrol increased transcription of DR4/5 in HOS and U2OS, leading to increased cell surface, and total DR4/5 protein expression. …”
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    Celastrol increases osteosarcoma cell lysis by γδ T cells through up-regulation of death receptors
  10. 470
    “…Western blot analysis data revealed that the expression level of STMN1 was dramatically increased in OS cell lines (HOS, Saos-2, U-2OS and MG-63), when compared to normal osteoblast hFOB1.19 cells. …”
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    Knockdown of STMN1 enhances osteosarcoma cell chemosensitivity through inhibition of autophagy
  11. 471
    por Seo, Jae-Hyun, Kim, Yong-Hyuk
    Publicado 2018
    “…The attack types of KDD CUP 1999 dataset are divided into four categories: user to root (U2R), remote to local (R2L), denial of service (DoS), and Probe. …”
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    Machine-Learning Approach to Optimize SMOTE Ratio in Class Imbalance Dataset for Intrusion Detection
  12. 472
    “…ULMs (UHM ligand motif) mediate protein interactions during spliceosome assembly by binding to UHM (U2AF homology motifs) domains. Using NMR, biophysical methods and crystallography we show that the Rev ULM binds to the UHMs of U2AF65 and SPF45. …”
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    Modulation of HIV-1 gene expression by binding of a ULM motif in the Rev protein to UHM-containing splicing factors
  13. 473
    “…Knockdown of TMEM45B significantly suppressed the proliferation, migration, and invasion of U2OS cells in vitro. Mechanistically, knockdown of TMEM45B sharply downregulated the expression level of β-catenin, cyclin D1, and c-Myc in U2OS cells. …”
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    Silencing Transmembrane Protein 45B (TNEM45B) Inhibits Proliferation, Invasion, and Tumorigenesis in Osteosarcoma Cells
  14. 474
    por Wang, Chao, Wang, Wen-Bo
    Publicado 2018
    “…The proliferation effect of osteosarcoma cell lines U2OS was examined by Cell Counting Kit-8. The invasive and migratory capabilities were determined by transwell invasion and migration assay. …”
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    Telmisartan Induces Osteosarcoma Cells Growth Inhibition and Apoptosis Via Suppressing mTOR Pathway
  15. 475
    “…The calculation result shows that the lattice thermal conductivity of sub-U(3)Si(1.5)Al(0.5) and sub-U(2.5)Si(2)Al(0.5)(I) after alloying exhibits high isotropy as the temperature increases.…”
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    First-principles investigations on the anisotropic elasticity and thermodynamic properties of U(3)Si(2)–Al
  16. 476
    “…In the analysis of the U2 snDNA, we were able to corroborate that U2 snDNA and U5 snDNA were linked in the same tandem array, and this has interest for tracing evolutionary lines. …”
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    Genetic characterization of Plectorhinchus mediterraneus yields important clues about genome organization and evolution of multigene families
  17. 477
    “…After 24h, 1mM NiCl(2) caused a similar and significant reduction of viability in U2OS and HaCat cells, while higher NiCl(2) concentrations and longer exposure times showed a reduced cytotoxic effect in HaCat as compared to U2OS cells. …”
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    Effect of Nickel Chloride on Cell Proliferation
  18. 478
    “…TMEM35 knockdown inhibited cell migration in SaOS2 and U2OS cells in wound-healing assays. In conclusion, TMEM35, a member of the tetraspanin family, serves an important role in the growth of OSA cells.…”
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    Downregulation of coding transmembrane protein 35 gene inhibits cell proliferation, migration and cell cycle arrest in osteosarcoma cells
  19. 479
    “…Consistently, H2A.Z promotes efficient spliceosomal rearrangements involving the U2 snRNP, as H2A.Z loss results in persistent U2 snRNP association and decreased recruitment of downstream snRNPs to nascent RNA. …”
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    The histone variant H2A.Z promotes efficient cotranscriptional splicing in S. cerevisiae
  20. 480
    “…Knockdown of miR-92b significantly inhibited the proliferation and invasion of osteosarcoma U2OS cells (P<0.01). By contrast, overexpression of miR-92b significantly increased U2OS cell proliferation and invasion (P<0.01). …”
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    MicroRNA-92b promotes cell proliferation and invasion in osteosarcoma by directly targeting Dickkopf-related protein 3
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