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197021“…Studies have shown that peroxisome proliferator-activated receptor- [Formula: see text] (PPAR- [Formula: see text]) agonists could ameliorate renal fibrotic lesions in both diabetic nephropathy and nondiabetic chronic kidney diseases. …”
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197022por Akagiri, Satomi, Naito, Yuji, Ichikawa, Hiroshi, Mizushima, Katsura, Takagi, Tomohisa, Handa, Osamu, Kokura, Satoshi, Yoshikawa, Toshikazu“…In the present study, to evaluate the anti-obesity and anti-diabetic effects of BOF, we investigated the effects of BOF on the white adipose tissue (WAT) weight, the size of adipocytes, adiponectin expression, and oral glucose tolerance test results in high-fat diet-fed male KK/Ta mice. …”
Publicado 2008
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197023por Volvert, Marie-Laure, Seyen, Sandrine, Piette, Marie, Evrard, Brigitte, Gangolf, Marjorie, Plumier, Jean-Christophe, Bettendorff, Lucien“…Benfotiamine (S-benzoylthiamine O-monophosphate), an amphiphilic S-acyl thiamine derivative, prevents the progression of diabetic complications, probably by increasing tissue levels of thiamine diphosphate and so enhancing transketolase activity. …”
Publicado 2008
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197024por Ogah, Okechukwu S, Adegbite, Gail D, Akinyemi, Rufus O, Adesina, Julius O, Alabi, Albert A, Udofia, Oscar I, Ogundipe, Roseline F, Osinfade, Julius KL“…Pericardial diseases, cor-pulmonale, ischaemic heart disease, congenital heart diseases, diabetic heart disease, thyroid heart disease, sickle cell cardiopathy were present in 26(1.8%), 23(1.6%), 9(0.6%), 6(0.4%), 6(0.4%), 6(0.4%), 1(0.1%), and 1(0.1%) respectively. …”
Publicado 2008
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197025por Scholz, Joachim, Mannion, Richard J., Hord, Daniela E., Griffin, Robert S., Rawal, Bhupendra, Zheng, Hui, Scoffings, Daniel, Phillips, Amanda, Guo, Jianli, Laing, Rodney J. C., Abdi, Salahadin, Decosterd, Isabelle, Woolf, Clifford J.“…METHODS AND FINDINGS: Using a structured interview (16 questions) and a standardized bedside examination (23 tests), we prospectively assessed symptoms and signs in 130 patients with peripheral neuropathic pain caused by diabetic polyneuropathy, postherpetic neuralgia, or radicular low back pain (LBP), and in 57 patients with non-neuropathic (axial) LBP. …”
Publicado 2009
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197026“…The selective, high affinity A(2B) adenosine receptor (AdoR) antagonists that were synthesized by several research groups should aid in determining the role of the A(2B) AdoR in inflammatory diseases like asthma or rheumatoid arthritis (RA) and angiogenic diseases like diabetic retinopathy or cancer. CV Therapeutics scientists discovered the selective, high affinity A(2B) AdoR antagonist 10, a 8-(4-pyrazolyl)-xanthine derivative [CVT-6883, K(i)(hA(2B)) = 22 nM; K(i)(hA(1)) = 1,940 nM; K(i)(hA(2A)) = 3,280; and K(i)(hA(3)) = 1,070 nM] that has favorable pharmacokinetic (PK) properties (t(1/2) = 4 h and F > 35% rat). …”
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197027por Baumann, Marcus, Richart, Tom, Sollinger, Daniel, Pelisek, Jaroslav, Roos, Marcel, Kouznetsova, Tatiana, Eckstein, Hans-Henning, Heemann, Uwe, Staessen, Jan A“…BACKGROUND: N(ε)-Carboxymethyllysine (CML) is the major non-cross linking advanced glycation end product (AGE). CML is elevated in diabetic patients and apparent in atherosclerotic lesions. …”
Publicado 2009
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197028por Jimenez-Dalmaroni, Maximiliano J., Xiao, Nengming, Corper, Adam L., Verdino, Petra, Ainge, Gary D., Larsen, Dave S., Painter, Gavin F., Rudd, Pauline M., Dwek, Raymond A., Hoebe, Kasper, Beutler, Bruce, Wilson, Ian A.“…Therefore, an increased level of soluble forms of CD36, which has been reported to be increased in type II diabetic patients, could accelerate atherosclerosis by increasing the pro-inflammatory response to diacylglycerol ligands.…”
Publicado 2009
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197029por Al Ashgar, Hamad, Helmy, Ahmed, Khan, Mohamed Q., Al Kahtani, Khalid, Al Quaiz, Mohammed, Rezeig, Mohammed, Kagevi, Ingvar, Alshehri, Abdullah, Al Kalbani, Abdullah, Al Swat, Khalid, Dahab, Salim, Elkum, Naser, Al Fadda, Mohammed“…Compared to relapsers, sustained responders were significantly younger (P=.005), non-diabetic (P=.005), had higher serum albumin (P=.028), lower alpha-fetoprotein level (P=.026), lower aspartate aminotransferase (AST) (P=.04) levels, and were treatment-naivve (P=.008). …”
Publicado 2009
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197030por Mughal, R. S., Scragg, J. L., Lister, P., Warburton, P., Riches, K., O’Regan, D. J., Ball, S. G., Turner, N. A., Porter, K. E.“…AIMS/HYPOTHESIS: Endothelial cells (ECs) and smooth muscle cells (SMCs) play key roles in the development of intimal hyperplasia in saphenous vein (SV) bypass grafts. In diabetic patients, insulin administration controls hyperglycaemia but cardiovascular complications remain. …”
Publicado 2010
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197031por Coen, Giorgio, Pierantozzi, Andrea, Spizzichino, Daniele, Sardella, Daniela, Mantella, Daniela, Manni, Micaela, Pellegrino, Luigi, Romagnoli, Andrea, Pacifici, Roberta, Zuccaro, Piergiorgio, DiGiulio, Salvatore“…By logistic analysis, the one year increments in Agatston scores were found to be predictors of mortality. Diabetic and hypertensive patients have significantly higher Delta scores. …”
Publicado 2010
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197032por Riquelme, Raquel, Cediel, Rafael, Contreras, Julio, Lourdes, Rodriguez-de la Rosa, Murillo-Cuesta, Silvia, Hernandez-Sanchez, Catalina, Zubeldia, Jose M., Cerdan, Sebastian, Varela-Nieto, Isabel“…However, the Igf1(−/−) null mice in turn developed a prematurely aged stria vascularis reminiscent of the diabetic strial phenotype. Our data indicate that IGF-I is required for the correct development and maintenance of hearing, supporting the idea that IGF-I-based therapies could contribute to prevent or ameliorate age-related hearing loss.…”
Publicado 2010
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197033por Thundyil, John, Tang, Sung-Chun, Okun, Eitan, Shah, Kausik, Karamyan, Vardan T, Li, Yu-I, Woodruff, Trent M, Taylor, Stephen M, Jo, Dong-Gyu, Mattson, Mark P, Arumugam, Thiruma V“…BACKGROUND-: Adiponectin is a hormone produced in and released from adipose cells, which has been shown to have anti-diabetic and anti-inflammatory actions in peripheral cells. …”
Publicado 2010
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197034“…RESULTS: Of the 142 patients, 76 (53.5%) were undertransfused (mean Hb <10 gm%). 96 (67%) of the patients were taking some form of chelation therapy but out of them only 2 (2%) were adequately chelated (S. ferritin <1000 ng/ml). 5 (3.5%) of the patients were known diabetics on insulin therapy. 103 (72%) of the patients were retarded in terms of growth. …”
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197035por Brufau, Gemma, Bahr, Matthias J, Staels, Bart, Claudel, Thierry, Ockenga, Johann, Böker, Klaus HW, Murphy, Elizabeth J, Prado, Kris, Stellaard, Frans, Manns, Michael P, Kuipers, Folkert, Tietge, Uwe JF“…Therefore, we investigated the relationship between plasma BA levels and energy expenditure in humans. Type 2 diabetic (T2DM) patients (n = 12) and gender, age and BMI-matched healthy controls (n = 12) were studied before and after 8 weeks of treatment with a BA sequestrant. …”
Publicado 2010
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197036por Benammar, Chahid, Hichami, Aziz, Yessoufou, Akadiri, Simonin, Anne-Marie, Belarbi, Meriem, Allali , Hocine, Khan, Naim A“…This plant is used in Algerian traditional medicine for its anti-diabetic, sedative, analgesic, anti-inflammatory and hypoglycaemic activities. …”
Publicado 2010
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197037por Zhong, Xiufeng, Huang, Hu, Shen, Jikui, Zacchigna, Serena, Zentilin, Lorena, Giacca, Mauro, Vinores, Stanley A.“…CONCLUSIONS: VEGF-B overexpression promoted pathological retinal and choroidal NV and BRB breakdown without causing inflammation, which is associated with the progression of diabetic retinopathy and age-related macular degeneration, showing that these complications are not dependent on inflammation. …”
Publicado 2011
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197038por Savidge, Tor C.“…Enteric neuropathy is also often associated with abnormal NO production, for example in achalasia and diabetic gastroparesis. Not surprisingly therefore, aberrant nNOS activity is widely implicated in enteric disease, and represents a potential molecular target for therapeutic intervention. …”
Publicado 2011
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197039por Roescher, Nienke, Vosters, Jelle L., Yin, Hongen, Illei, Gabor G., Tak, Paul P., Chiorini, John A.“…To test the utility of ICAM-1 as a therapeutic target, we used local gene therapy in Non Obese Diabetic (NOD) mice to express soluble (s)ICAM-1 to compete with membrane bound ICAM-1 for binding with its receptor. …”
Publicado 2011
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197040“…OBJECTIVE: VEGFR1 and 2 signaling have both been increasingly shown to mediate complications of ischemic retinopathies, including retinopathy of prematurity (ROP), age-related macular degeneration (AMD), and diabetic retinopathy (DR). This study evaluates the effects of blocking VEGFR1 and 2 on pathological angiogenesis and vascular leakage in ischemic retinopathy in a model of ROP and in choroidal neovascularization (CNV) in a model of AMD. …”
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