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  1. 401
    “…Strikingly, MSCs-based therapy significantly ameliorated both clinical and histopathological severity of PE symptoms including decreasing the blood pressure and proteinuria, suppressing glomerulonephritis, protecting the feto-placental development. The therapy also reversed abnormal TNF-α expression in uterine and splenic lymphocytes. …”
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    Mesenchymal Stem Cells Ameliorate Th1-Induced Pre-Eclampsia-Like Symptoms in Mice via the Suppression of TNF-α Expression
  2. 402
    “…The syndrome is also characterized by impaired blood flow through the utero-placental circulation and relative placental ischemia, which in turn, may generate feto-placental endothelial dysfunction. Endothelial dysfunction in offspring born from preeclamptic pregnancies has been associated with an increased risk of cardiovascular disease, including hypertension, later in life. …”
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    Impaired adenosine-mediated angiogenesis in preeclampsia: potential implications for fetal programming
  3. 403
    “…All 8 patients were young males and showed a mature teratoma or a mature teratoma with a focal immature teratoma in the resected specimens. Serum alpha-feto protein was variably elevated. Seven patients experienced an increase in tumor size after the chemotherapy. …”
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    Exuberant Vasculoconnective Component in Mediastinal Mixed Germ Cell Tumors
  4. 404
    “…Our data suggest an interplay between Tregs and uMCs that is relevant for the changes required at the feto-maternal interface for the normal development of pregnancy.…”
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    Transfer of regulatory T cells into abortion-prone mice promotes the expansion of uterine mast cells and normalizes early pregnancy angiogenesis
  5. 405
    “…The villous tree of the human placenta is a complex three-dimensional (3D) structure with branches and nodes at the feto-maternal border in the key area of gas and nutrient exchange. …”
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    Novel 3D light microscopic analysis of IUGR placentas points to a morphological correlate of compensated ischemic placental disease in humans
  6. 406
    “…Double immune-fluorescence with trophoblast-specific markers was used to identify galectin-expressing cells at the feto-maternal interface in the decidua. Gal-3 was significantly downregulated only in the extravillous trophoblast of IUGR placentas. …”
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    Placental Expression Patterns of Galectin-1, Galectin-2, Galectin-3 and Galectin-13 in Cases of Intrauterine Growth Restriction (IUGR)
  7. 407
    “…Maternal bilirubin levels and INR have a role in predicting adverse feto-maternal outcome.…”
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    Predictors of Fetal and Maternal Outcome in the Crucible of Hepatic Dysfunction During Pregnancy
  8. 408
    “…Our results suggest that uNKs and uMCs can counterbalance their effects at the feto–maternal interface and jointly promote SA remodeling and placentation.…”
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    Safeguarding of Fetal Growth by Mast Cells and Natural Killer Cells: Deficiency of One Is Counterbalanced by the Other
  9. 409
    “…DISCUSSION: This case demonstrates the potential for severe pulmonary coccidioidomycosis and vascular strain of pregnancy-associated vascular expansion in the first trimester of pregnancy and the possibility of a favorable pregnancy outcome in subsequent pregnancies after appropriate treatment. The route of feto-maternal transmission and placental lesions in coccidioidomycosis are discussed.…”
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    Coccidioidomycosis in pregnancy: Case report and literature review of associated placental lesions
  10. 410
    “…Pregnancies affected by gestational diabetes mellitus are at risk for the development of preeclampsia, a severe life threatening condition, associated with significant feto-maternal morbidity and mortality. It is a risk factor for long-term health in women and their offspring. …”
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    Excessive Neutrophil Activity in Gestational Diabetes Mellitus: Could It Contribute to the Development of Preeclampsia?
  11. 411
    “…During disease induction, Ezh2 loss derepresses a subset of bivalent promoters that resolve toward gene activation, inducing a feto-oncogenic program that includes genes such as Plag1, whose overexpression phenocopies Ezh2 loss to accelerate AML induction in mouse models. …”
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    Contrasting requirements during disease evolution identify EZH2 as a therapeutic target in AML
  12. 412
    “…Trophoblast-derived extracellular vesicles (EVs), either at the feto–maternal interface or in circulation, target CD4(+) T cells. …”
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    Unravelling the Role of Trophoblastic-Derived Extracellular Vesicles in Regulatory T Cell Differentiation
  13. 413
    “…Macrophage migration inhibitory factor (MIF) is a multifunctional cytokine abundantly present at the feto-maternal interface proposed to play a role in establishment of pregnancy. …”
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    Macrophage migration inhibitory factor is involved in endovascular trophoblast cell function in vitro
  14. 414
    “…The placenta is important in feto-maternal tolerance. We have used placenta-derived decidua stromal cells (DSCs) to treat graft-versus-host disease and found an immunomodulatory and anti-inflammatory effect. …”
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    Treatment of radiculomyelopathy in two patients with placenta-derived decidua stromal cells
  15. 415
    “…Recently, it has been described that fractalkine (CX3CL1, FKN) might have an important role in the feto–maternal interaction during gestation since the trophoblast cells express fractalkine receptor (CX3CR1) and the endometrium cells secrete fractalkine. …”
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    Fractalkine Regulates HEC-1A/JEG-3 Interaction by Influencing the Expression of Implantation-Related Genes in an In Vitro Co-Culture Model
  16. 416
    “…AIM: To study the association between early pregnancy BMI and feto-maternal outcome among nulliparous women METHODOLOGY: This is a prospective observational study, with a sample size of 294. …”
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    Maternal early pregnancy body mass index and pregnancy outcomes among nulliparous women registered in tertiary care hospital and urban slum hospital of a metropolitan city
  17. 417
    “…Calcium-selective transient receptor potential Vanilloid 6 (TRPV6) channels are expressed in fetal labyrinth trophoblasts as part of the feto–maternal barrier, necessary for sufficient calcium supply, embryo growth, and bone development during pregnancy. …”
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    Transient Receptor Potential Vanilloid 6 (TRPV6) Proteins Control the Extracellular Matrix Structure of the Placental Labyrinth
  18. 418
    “…This implies that along the feto-maternal placenta interface an interaction occurs between two retrovirus-derived genes, Peg10 and retrotransposon Gag like 1 (Rtl1, also called Peg11), that is essential for the maintenance of fetal capillary endothelial cells.…”
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    PEG10 viral aspartic protease domain is essential for the maintenance of fetal capillary structure in the mouse placenta
  19. 419
    “…A sufficient vascular network within the feto-maternal interface is necessary for placental function. …”
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    Estrogens Regulate Placental Angiogenesis in Horses
  20. 420
    “…CD8+ T cells are the most frequent T cell population in the immune cell compartment at the feto-maternal interface. Due to their cytotoxic potential, the presence of CD8+ T cells in the immune privileged pregnant uterus has raised considerable interest. …”
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    The New Old CD8+ T Cells in the Immune Paradox of Pregnancy
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