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  1. 1521
    “…OBJECTIVE: Accumulating evidence from other countries indicates potential associations between gout and cardiovascular diseases; however, the associations of gout with cardiovascular diseases, particularly stroke, ischemic heart disease, and heart failure, remain ambiguous in the Korean population. …”
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    An elevated likelihood of stroke, ischemic heart disease, or heart failure in individuals with gout: a longitudinal follow-up study utilizing the National Health Information database in Korea
  2. 1522
    “…BACKGROUND: Gout is a common arthritic disease resulting from elevated serum uric acid (SUA) level. …”
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    Polymorphisms in GCKR, SLC17A1 and SLC22A12 were associated with phenotype gout in Han Chinese males: a case–control study
  3. 1523
    “…The aim of this study was to determine the association between P2X7R rs3751142 and CARD8 rs2043211 polymorphisms and gout susceptibility in male Korean subjects. This study enrolled a total of 242 male patients with gout and 280 healthy controls. …”
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    Genetic Association for P2X7R rs3751142 and CARD8 rs2043211 Polymorphisms for Susceptibility of Gout in Korean Men: Multi-Center Study
  4. 1524
    por Moir, D. M.
    Publicado 1895
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    Medicine—Waters and Baths for Chronic Gout and Its Complications—The Salts of Uranium in Diabetes Melitus—Intravenous Mercurial Injections in Syphilis—The Liver as an Organ of Elemination of Corpuscular Elements—Anarcotine
  5. 1525
    por Bampfield, R. W.
    Publicado 1822
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    Observations on Dr. Scudamore's Reply to Mr. Bampfield's Paper on the Use of Colchicum in Gout; with a Vindication of Himself, and a Farther Defenoe of the Character and Utility of That Medicine in the Treatment of That Disease
  6. 1526
    Publicado 1827
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    A Treatise on a Modified Application of Moxa in the Treatment of Stiff and Contracted Joints, and Also in Chronic Rheumatism, Rheumatic Gout, Lumbago, Sciatica, Indolent Tumours, &c. &c.
  7. 1527
    “…(1) Background: To determine which factors are associated with the volume of monosodium urate (MSU) crystal deposition quantified by dual-energy computed tomography (DECT) in urate-lowering therapy (ULT)-naive gout patients. (2) Methods: In this multicenter cross-sectional study, DECT scans of knees and feet/ankles were prospectively obtained from ULT-naive gout patients. …”
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    Association of Specific Comorbidities with Monosodium Urate Crystal Deposition in Urate-Lowering Therapy-Naive Gout Patients: A Cross-Sectional Dual-Energy Computed Tomography Study
  8. 1528
    “…In conclusion, the findings show that EtOH80 extract has a substantial anti-gout effect by lowering uric acid levels and suppressing pro-inflammatory mediator production due to the andrographolide content, that might be beneficial in the treatment of gouty-inflammation.…”
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    Extracts of Andrographis paniculata (Burm.f.) Nees Leaves Exert Anti-Gout Effects by Lowering Uric Acid Levels and Reducing Monosodium Urate Crystal-Induced Inflammation
  9. 1529
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    Corrigendum to Health-related quality of life in gout in primary care: Baseline findings from a cohort study Seminars in Arthritis & Rheumatism, 48 (2018) 61-69
  10. 1530
    por Zahir, Farah R.
    Publicado 2022
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    Editorial on the Paper “The Epidemiology and Genetics of Hyperuricemia and Gout across Major Racial Groups: A Literature Review and Population Genetics Secondary Database Analysis” by Butler, Alghoubayshi and Roman
  11. 1531
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    Risk of a major adverse cardiovascular event (MACE) following first-ever hospitalisation for acute gout: a Western Australian population-level linked data study.
  12. 1532
    “…OBJECTIVE: The aim was to test the feasibility of a randomized controlled trial exploring whether omega-3 fatty acid supplementation limits gout flares during treat-to-target urate-lowering treatment (T2T-ULT). …”
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    Feasibility of conducting a randomized, placebo-controlled study assessing whether omega-3 fatty acids prevent gout flares when starting urate-lowering treatment
  13. 1533
    “…INTRODUCTION: Hyperuricemia and gout are receiving an increasing scientific and medical attention because of their relatively high prevalence and their association with relevant co-morbidities. …”
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    A randomized pilot trial assessing the reduction of gout episodes in hyperuricemic patients by oral administration of Ligilactobacillus salivarius CECT 30632, a strain with the ability to degrade purines
  14. 1534
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    Afflux massif de victimes pédiatriques
  15. 1535
    “…OBJECTIVE: To assess 12‐month safety and efficacy of pegloticase + methotrexate (MTX) versus pegloticase + placebo (PBO) cotherapy in a PBO‐controlled, double‐blind trial (A randomized, double‐blind, placebo‐controlled, multicenter, efficacy and safety study of methotrexate to increase response rates in patients with uncontrolled gout receiving pegloticase [MIRROR RCT]). METHODS: Patients with uncontrolled gout (serum urate level [SU] ≥7 mg/dl, oral urate‐lowering therapy failure or intolerance, and presence of one or more gout symptoms [one or more tophi, two or more flares in 12 months, gouty arthropathy]) were randomized 2:1 to receive pegloticase (8‐mg infusion every 2 weeks) with blinded MTX (oral 15 mg/week) or PBO for 52 weeks. …”
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    A Randomized, Double‐Blind, Placebo‐Controlled Multicenter Efficacy and Safety Study of Methotrexate to Increase Response Rates in Patients With Uncontrolled Gout Receiving Pegloticase: 12‐Month Findings
  16. 1536
    “…INTRODUCTION: Gout is the most prevalent arthritis and significantly impacts on function and quality of life. …”
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    Gout impacts on function and health-related quality of life beyond associated risk factors and medical conditions: results from the KING observational study of the Italian Society for Rheumatology (SIR)
  17. 1537
    “…INTRODUCTION: In gout, monosodium urate (MSU) crystals deposit intra-articularly and cause painful arthritis. …”
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    Urate Crystal Induced Inflammation and Joint Pain Are Reduced in Transient Receptor Potential Ankyrin 1 Deficient Mice – Potential Role for Transient Receptor Potential Ankyrin 1 in Gout
  18. 1538
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    Erratum to: Polymorphisms in GCKR, SLC17A1 and SLC22A12 were associated with phenotype gout in Han Chinese males: a case–control study
  19. 1539
    “…Here, 1,993 primary gout patients and 4,902 health examination participants (3,305 males and 1,597 females) were genotyped with R90H and W258X. …”
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    The effects of URAT1/SLC22A12 nonfunctional variants,R90H and W258X, on serum uric acid levels and gout/hyperuricemia progression
  20. 1540
    Publicado 1855
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    Observations on the Life, Disease, and Death of John Hunter, in Elucidation of the Nature and Treatment of Gout and Angina Pectoris, Being the Oration Delivered before the Hunterian Society, at Its Thirty-Sixth Anniversary
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