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Hepatitis B
2
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2
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1
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1
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1
Difteria
1
Enfermedades transmisibles
1
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1
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1
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14861por Farokhizadeh, Zhaleh, Dehbidi, Sahar, Geramizadeh, Bita, Yaghobi, Ramin, Malekhosseini, Seyed Ali, Behmanesh, Mehrdad, Sanati, Mohammad Hossein, Afshari, Afsoon, Moravej, Ali, Karimi, Mohammad Hossein“…We investigated the relationship between miR-499A>G, miR-149C>T, miR-196a2T>C, and miR-146aG>C and HCC susceptibility, examining the interaction of the miRNAs with hepatitis B virus (HBV). METHODS: We evaluated the associations of miR-499A>G (rs3746444), miR-149C>T (rs2292832), miR-196a2T>C (rs11614913), and miR-146aG>C (rs2910164) with HCC susceptibility in 100 HCC patients (70 males and 30 females) and 120 healthy controls (70 males and 50 females), using the PCR-restriction fragment length polymorphism method. …”
Publicado 2019
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14862“…Interestingly, MEG3 was also reduced in chronic hepatitis B (CHB) patients with liver fibrosis when compared with healthy controls. …”
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14863por Shigekawa, Yoshinobu, Hayami, Shinya, Ueno, Masaki, Miyamoto, Atsushi, Suzaki, Norihiko, Kawai, Manabu, Hirono, Seiko, Okada, Ken-ichi, Hamamoto, Ryuji, Yamaue, Hiroki“…KDM5B-positive group had much poorer prognosis than that of the negative group, especially in HCC derived from persistent infection of hepatitis B virus (HBV) or hepatitis C virus (HCV) (54% vs 78%, p=0.015). …”
Publicado 2018
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14864por Huang, Ya-Xin, Zhao, Qi-Yi, Wu, Li-Li, Xie, Dong-Ying, Gao, Zhi-Liang, Deng, Hong“…T follicular helper cells (Tfh cells) affect essential immune pathogenesis in chronic hepatitis B virus (HBV) infection. The CCR7(lo)PD-1(hi) Tfh subset has a partial Tfh effector phenotype and is associated with active Tfh differentiation, whereas the CCR7(hi)PD-1(lo) Tfh subset is a resting phenotype. …”
Publicado 2018
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14865por Pang, Yuanjie, Kartsonaki, Christiana, Turnbull, Iain, Guo, Yu, Clarke, Robert, Chen, Yiping, Bragg, Fiona, Yang, Ling, Bian, Zheng, Millwood, Iona Y., Hao, Juanzhi, Han, Xianyong, Zang, Yajing, Chen, Junshi, Li, Liming, Holmes, Michael V., Chen, Zhengming“…We examined the associations of diabetes with chronic liver diseases and liver cancer and of random plasma glucose (RPG) with these liver diseases among participants without diabetes in Chinese adults and the possible interaction by hepatitis B virus (HBV) infection. The prospective China Kadoorie Biobank recruited 512,891 adults. …”
Publicado 2018
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14866por Sorgho, Pegdwendé Abel, Martinson, Jeremy James, Djigma, Florencia Wendkuuni, Yonli, Albert Théophane, Nagalo, Bolni Marius, Compaore, Tegwinde Rebeca, Obiri-Yeboah, Dorcas, Diarra, Birama, Sombie, Herman Karim, Zongo, Arsène Wendpagnangdé, Ouattara, Abdoul Karim, Soubeiga, Serge Théophile R., Traore, Lassina, Roberts, Lewis R., Simpore, Jacques“…BACKGROUND/OBJECTIVE: Hepatitis B virus (HBV) infection is the leading risk factor for cirrhosis and hepatocellular carcinoma (HCC). …”
Publicado 2018
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14867por Hu, Jianhua, Zhao, Hong, Lou, Danfeng, Gao, Hainv, Yang, Meifang, Zhang, Xuan, Jia, Hongyu, Li, Lanjuan“…METHODS: Patients infected with ACLF-based hepatitis B virus (HBV) from 1 December 2016 to 31 May 2018 were enrolled in our study and were divided into infected and uninfected groups. …”
Publicado 2018
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14868“…Matrine (Mat) has been applied for the treatment of hepatitis B, but its effect on NAFLD is still unknown, and there is no unified view of Mat on the regulation of ER stress in the previous literature. …”
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14869“…None of the incidents reportedly led to transmission of hepatitis B, hepatitis C, or HIV. CONCLUSION: Police officers in this department are at risk for needlestick injuries and other exposures to bloodborne pathogens. …”
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14870“…In the current study, the diagnostic performance of serum AKR1B10 in hepatitis B virus/hepatitis C virus (HBV/HCV)-related liver disorders was evaluated and the unique role of AKR1B10 in diagnosing HCC was assessed. …”
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14871por Yan, Qiang, Wang, Lei, Lai, Liusheng, Liu, Song, Chen, Huaizhou, Zhang, Jiaxing, Dai, Yong, Sui, Weiguo“…A total of six patients with hepatitis B virus (HBV)-related ACLF and six healthy control subjects were prospectively enrolled in the present study. …”
Publicado 2019
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14872“…The most common etiology was alcohol (32/74, 43.2%) in non-HCC group and hepatitis B (30/49, 61.2%) in HCC group (P = 0.009). …”
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14873por Mekonnen, Hailemichael Desalegn, Fisseha, Henok, Getinet, Tewodros, Tekle, Fisseha, Galle, Peter R.“…Older age adjusted Odds Ratio (AOR) (95%CI, p-value) 1.07(1.03-1.09, <0.001), viral hepatitis B (AOR) (95%CI, p-value) 6.19 (1.92-19.93, 0.002), and H. pylori infection (AOR) (95%CI, p-value) 5.22 (2.04–13.31, <0.001) were statistically significantly associated with HCC. …”
Publicado 2018
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14874por Wei, Luopei, Li, Ni, Wang, Gang, Feng, Xiaoshuang, Lyu, Zhangyan, Li, Xin, Wen, Yan, Chen, Yuheng, Chen, Hongda, Chen, Shuohua, Wu, Shouling, Dai, Min, He, Jie“…Especially, the U-shaped association between waist circumference and PLC risk tended to be strengthened among subjects with hepatitis B surface antigen (HBsAg) negativity (HR(Q5vs.Q3) = 2.39, 95%CI: 1.43–3.98; HR(Q1vs.Q3) = 2.27, 95%CI = 1.29–4.01). …”
Publicado 2018
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14875por Farooq, Muhammad, Filliol, Aveline, Simoes Eugénio, Mélanie, Piquet-Pellorce, Claire, Dion, Sarah, Raguenes-Nicol, Céline, Jan, Aurélien, Dimanche-Boitrel, Marie-Thérèse, Le Seyec, Jacques, Samson, Michel“…The present study aimed to define the role of RIPK1 in hepatocytes during fulminant viral hepatitis, a worldwide syndrome mainly observed in hepatitis B virus (HBV) infected patients. Mice deficient for RIPK1, specifically in liver parenchymal cells (Ripk1(LPC-KO)) and their wild-type littermates (Ripk1(fl/fl)), were challenged by either the murine hepatitis virus type 3 (MHV3) or poly I:C, a synthetic analog of double-stranded RNA mimicking viral pathogen-associated molecular pattern. …”
Publicado 2019
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14876por Lin, Chun-Ta, Hsieh, Yue-Ting, Yang, Yeng-Jey, Chen, Shih-Hui, Wu, Cheng-Hsuan, Hwang, Lih-Hwa“…Hepatitis B virus (HBV) infection causes acute and chronic liver inflammation. …”
Publicado 2019
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14877por Xiong, Lei, Wu, Feng, Wu, Qiong, Xu, Liangliang, Cheung, Otto K., Kang, Wei, Mok, Myth T., Szeto, Lemuel L. M., Lun, Cheuk-Yin, Lung, Raymond W., Zhang, Jinglin, Yu, Ken H., Lee, Sau-Dan, Huang, Guangcun, Wang, Chiou-Miin, Liu, Joseph, Yu, Zhuo, Yu, Dae-Yeul, Chou, Jian-Liang, Huang, Wan-Hong, Feng, Bo, Cheung, Yue-Sun, Lai, Paul B., Tan, Patrick, Wong, Nathalie, Chan, Michael W., Huang, Tim H., Yip, Kevin Y., Cheng, Alfred S., To, Ka-Fai“…Conversely, deletion of this enhancer via CRISPR/Cas9 reduces C/EBPβ expression and its genome-wide co-occupancy with BRD4 at H3K27ac-marked enhancers and super-enhancers, leading to drastic suppression of driver oncogenes and HCC tumorigenicity. Hepatitis B X protein transgenic mouse model of HCC recapitulates this paradigm, as C/ebpβ enhancer hypomethylation associates with oncogenic activation in early tumorigenesis. …”
Publicado 2019
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14878por Huang, Chi-Chun, Chen, Hsin-Yi, Chang, Ruei-Hsin, Liao, Pen-An, Lien, Heng-Hui, Hung, Chih-Sheng, Yang, Sien-Sing, Hu, Jui-Ting“…Of the 67 eligible patients, 66 patients (99%) were diagnosed as Barcelona Clinic Liver Cancer stage C and 45 (67%) were Child-Pugh A. Chronic hepatitis B virus infection was the main etiology (67%), followed by hepatitis C virus infection (12%) and alcohol liver disease (8%). …”
Publicado 2019
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14879por Yooda, Arzouma Paul, Sawadogo, Salam, Soubeiga, Serge Théophile, Obiri-Yeboah, Dorcas, Nebie, Koumpingnin, Ouattara, Abdoul Karim, Diarra, Birama, Simpore, Abibou, Yonli, Yetema Dieudonné, Sawadogo, Abdoul-Guaniyi, Drabo, Bia Emile, Zalla, Seimbou, Siritié, Anita Pierrette, Nana, Rodrigue Sosthène, Dahourou, Honorine, Simpore, Jacques“…This study aimed to estimate the residual risk of transmission of HIV, hepatitis B virus (HBV), and hepatitis C virus (HCV) by blood transfusion at the Regional Blood Transfusion Center (RBTC) of Ouagadougou. …”
Publicado 2019
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14880“…Vaccines included: hepatitis B (HBV: birth, 1 month and 6 months); diphtheria, tetanus, acellular pertussis, polio, Haemophilus influenzae b (DTaP-IPV-Hib: 2, 4, 6 and 12 months); pneumococcal conjugate (PCV: 2, 4 and 12 months); measles-mumps-rubella/measles-mumps-rubella-varicella (MMR/MMRV: 12 months) and hepatitis A (HAV: 18 and 24 months). …”
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