Mostrando 11,621 - 11,640 Resultados de 12,673 Para Buscar '"intellectual"', tiempo de consulta: 0.22s Limitar resultados
  1. 11621
  2. 11622
  3. 11623
    “…nArgBP2, a candidate gene for intellectual disability, is a postsynaptic protein critical for dendritic spine development and morphogenesis, and its knockdown (KD) in developing neurons severely impairs spine-bearing excitatory synapse formation. …”
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  4. 11624
    “…These data suggest that NLGN4 functions in systems involved in intellectual abilities, social abilities, and sleep and wakefulness, impairments of which are commonly seen in autism. …”
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  5. 11625
  6. 11626
    “…The 16p11.2 duplication is a copy number variant that confers risk for diverse NPDs including autism spectrum disorder, schizophrenia, intellectual disability and epilepsy. We used a mouse model of the 16p11.2 duplication (16p11.2(dup/+)) to uncover molecular and circuit properties associated with this broad phenotypic spectrum, and examined genes within the locus capable of phenotype reversal. …”
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  7. 11627
    “…Imposter syndrome is self-perceived intellectual fraud in areas of success. Sense of belonging and imposter syndrome can influence behavior and well-being and are linked to academic and career outcomes. …”
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  8. 11628
  9. 11629
    “…Multiple regression analysis of a sample limited to patients with intellectual disability (ID) (n = 44; R (2) = 0.366; F = 4.493; P = 0.002) revealed continued psychotherapy at follow‐up (t = 2.610, P = 0.013) and successful reduction in antiseizure medication (t = 2.868; P = 0.007) as positively related with improved QoL. …”
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  10. 11630
    “…SCN2A is also a high confidence risk gene for autism spectrum disorder (ASD) and nonsyndromic intellectual disability (ID). Previous work to determine the functional consequences of SCN2A variants yielded a paradigm in which predominantly gain-of-function (GoF) variants cause epilepsy whereas loss-of-function (LoF) variants are associated with ASD and ID. …”
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  11. 11631
    por Vlase, Ionela, Lähdesmäki, Tuuli
    Publicado 2023
    “…The article aims to understand the role of Web of Science (WOS) as a tool of knowledge management in academia by drawing on the scholarly output volume, the patterns displayed by this volume, and the intellectual structure of cultural heritage research based on WOS-indexed journal articles. …”
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  12. 11632
    “…Mutations in the solute carrier family 6-member 8 (Slc6a8) gene, encoding the protein responsible for cellular creatine (Cr) uptake, cause Creatine Transporter Deficiency (CTD), an X-linked neurometabolic disorder presenting with intellectual disability, autistic-like features, and epilepsy. …”
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  13. 11633
  14. 11634
    por Smith, Kevin L, Dickson, Katherine A
    Publicado 2016
    “…Chapters on the legalities and practicalities of open access in academic libraries address the issues associated with copyright, licensing, and intellectual property and include support for courses that require open access distribution of student work. …”
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  15. 11635
    por RÍOS ORTEGA, JAIME
    Publicado 2007
    “…THE SECOND ONE UNDERLINES THE FUNDAMENTAL ROLE PLAYED BY THE INTELLECTUAL HISTORY OF CONCEPTS AND LIBRARY SCIENCE THEORIES. …”
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  16. 11636
    “…AKAP11 and GRIN2A mutations are also associated with bipolar disorder [2], and epilepsy and developmental delay/intellectual disability [1, 3, 4], respectively. Accessible in both humans and rodents, electroencephalogram (EEG) recordings offer a window into brain activity and display abnormal features in schizophrenia patients. …”
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  17. 11637
  18. 11638
    “…Due to the disruption of bile acid synthesis leading to cholesterol and cholestanol accumulation, CTX manifests as premature cataracts, chronic diarrhea, and intellectual disability in childhood and adolescence. …”
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  19. 11639
    “…Multivariable models predicted intellectual disability increased hours spent caring by primary caregivers (p = 0.01–0.04), and neuropsychiatric comorbidities decreased family functioning (p = 0.02) and caregiver HRQL (p < 0.01). …”
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  20. 11640
    “…Here we describe male triplets, clinically presenting with the phenotype of subtle but distinctive facial features, intellectual disability, increased body weight, neonatal EOEE, and prominently variable abnormal behaviors of autism and sexual arousal. …”
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