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  1. 32881
    por Wu, Zeng-Hong, Cai, Fucheng, Zhong, Yi
    Publicado 2020
    “…Guanylate binding proteins (GBPs) belongs to the interferons (IFNs) induced guanylate-binding protein family (Guanosine triphosphatases, GTPases) consisting of seven homologous members, termed GBP1 to GBP7. …”
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    Comprehensive Analysis of the Expression and Prognosis for GBPs in Head and neck squamous cell carcinoma
  2. 32882
    por Cann, Alan J.
    Publicado 2012
    “…The chapter also discusses the virus infections of plants, immune responses to virus infections in animals, apoptosis (programmed cell death), interferons (the blocking of virus infection), and evasion of immune responses. …”
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    Infection
  3. 32883
    “…ZCCHC3-deficiency markedly inhibited TLR3- but not TLR4-mediated induction of type I interferons (IFNs) and proinflammatory cytokines. Zcchc3(−/−) mice were more resistant to poly(I:C)- but not lipopolysaccharide-induced inflammatory death. …”
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    ZCCHC3 modulates TLR3-mediated signaling by promoting recruitment of TRIF to TLR3
  4. 32884
    “…The excessive release of cytokines and chemokines such as CCL2, CCL22, CXCL9 and CXCL12 and certain interferons and interleukins related genes like IFIH1, IFI44, IFIT1 and IL10 were significantly higher in patients with severe clinical presentation compared to mild and moderate presentations. …”
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    Host transcriptomic profiling of COVID-19 patients with mild, moderate, and severe clinical outcomes
  5. 32885
    “…In the course of immune responses to viruses, an initial activation of innate immunity typically occurs and the production of type I and III interferons is essential in the control of viral spread. …”
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    Immunological Aspects Related to Viral Infections in Severe Asthma and the Role of Omalizumab
  6. 32886
    por Ding, Xuewei, Ren, Yi, He, Xiaojie
    Publicado 2021
    “…Cell-free DNA/RNA is the most effective inducer of type I interferons (IFN-I). Resident renal cells (rather than infiltrating immune cells) are the main source of IFN-I in the kidney. …”
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    IFN-I Mediates Lupus Nephritis From the Beginning to Renal Fibrosis
  7. 32887
    “…Immediately after viral infection, elements of innate immunity, such as physical barriers, various phagocytic cells, group of cytokines, interferons (IFNs), and IFN-stimulated genes, provide the first line of defense for viral clearance. …”
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    Acute Infection of Viral Pathogens and Their Innate Immune Escape
  8. 32888
    “…CRS is associated with high circulating concentrations of several pro-inflammatory cytokines, including interleukins, interferons, tumor necrosis factors, colony-stimulating factors, and transforming growth factors. …”
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    Cytokine Release Syndrome Associated with T-Cell-Based Therapies for Hematological Malignancies: Pathophysiology, Clinical Presentation, and Treatment
  9. 32889
    “…Several cytokines and chemokines have been identified as biomarkers or therapeutic targets of NPSLE; in particular, the levels of type 1 interferons, TNFs, and IL-6 are elevated in SLE patient’s cerebrospinal fluid (CSF), and these factors contribute to the pathology of depression. …”
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    Accumulation of Senescent Neural Cells in Murine Lupus With Depression-Like Behavior
  10. 32890
    “…Using Ixodes ovatus Ehrlichia (IOE), a tick-borne pathogen that causes severe shock-like illness and bone marrow (BM) aplasia, type I and II interferons (IFNs) promoted loss of HSPCs via increased cell death and enforced quiescence. …”
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    IL-18R-mediated HSC quiescence and MLKL-dependent cell death limit hematopoiesis during infection-induced shock
  11. 32891
    “…In the present study, we analyzed human monocyte-derived macrophages (MDMs) using RM, revealing that macrophage activation by lipopolysaccharides (LPS), interferons (IFN), or cytokines can be identified by lipid composition, which significantly differs in M0 (resting), M1 (IFN-γ/LPS), M2a (IL-4/IL-13), and M2c (IL-10) MDMs. …”
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    Lipidome profiling with Raman microspectroscopy identifies macrophage response to surface topographies of implant materials
  12. 32892
    “…Finally, analysis of pyroptosis markers in plasma from COVID-19 patients with characterized severe pneumonia due to autoantibodies against, or inborn errors of, type I interferons (IFNs) highlights GSDME/caspase-3 as potential markers of disease severity. …”
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    Human NLRP1 is a sensor of pathogenic coronavirus 3CL proteases in lung epithelial cells
  13. 32893
    “…The engagement of TLRs with viral PAMPs triggers downstream signaling pathways leading to the production of inflammatory cytokines, interferons (IFNs) and other molecules essential for the prevention of viral replication. …”
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    Innate Immune Response to Dengue Virus: Toll-like Receptors and Antiviral Response
  14. 32894
    “…We observed that two groups of nucleic acid recognizing molecules, Toll-like receptors (TLRs) and the cytoplasmic retinoic acid-inducible gene (RIG)-I-like receptors (RLRs) form the backbone of host responses to pathogen nucleic acids, with additional support provided by absent in melanoma 2 (AIM2) and DNA-dependent activator of Interferons (IFNs)-regulatory factors (DAI) like cytosolic activity. …”
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    Host-pathogen protein-nucleic acid interactions: A comprehensive review
  15. 32895
    por Musella, Martina, Guarracino, Andrea, Manduca, Nicoletta, Galassi, Claudia, Ruggiero, Eliana, Potenza, Alessia, Maccafeo, Ester, Manic, Gwenola, Mattiello, Luca, Soliman Abdel Rehim, Sara, Signore, Michele, Pietrosanto, Marco, Helmer-Citterich, Manuela, Pallocca, Matteo, Fanciulli, Maurizio, Bruno, Tiziana, De Nicola, Francesca, Corleone, Giacomo, Di Benedetto, Anna, Ercolani, Cristiana, Pescarmona, Edoardo, Pizzuti, Laura, Guidi, Francesco, Sperati, Francesca, Vitale, Sara, Macchia, Daniele, Spada, Massimo, Schiavoni, Giovanna, Mattei, Fabrizio, De Ninno, Adele, Businaro, Luca, Lucarini, Valeria, Bracci, Laura, Aricò, Eleonora, Ziccheddu, Giovanna, Facchiano, Francesco, Rossi, Stefania, Sanchez, Massimo, Boe, Alessandra, Biffoni, Mauro, De Maria, Ruggero, Vitale, Ilio, Sistigu, Antonella
    Publicado 2022
    “…Here, we show that type I interferons (IFNs-I) function as molecular hubs of resistance during immunogenic chemotherapy, triggering the epigenetic regulator demethylase 1B (KDM1B) to promote an adaptive, yet reversible, transcriptional rewiring of cancer cells towards stemness and immune escape. …”
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    Type I IFNs promote cancer cell stemness by triggering the epigenetic regulator KDM1B
  16. 32896
    “…In the moderate efficacy group, we discussed interferons, glatiramer acetate, teriflunomide and dimethyl fumarate, while in the high efficacy group we discussed fingolimod, cladribine, natalizumab, ocrelizumab, alemtuzumab and ofatumumab. …”
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    How to choose initial treatment in multiple sclerosis patients: a case-based approach
  17. 32897
    “…Large-scale genetic association studies have indicated that genetic variations affecting SARS-CoV-2 receptors (angiotensin-converting enzymes, transmembrane serine protease-2) and immune components (Interferons, Interleukins, Toll-like receptors and Human leukocyte antigen) are critical host determinants related to the severity of COVID-19. …”
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    Human genetic basis of severe or critical illness in COVID-19
  18. 32898
    “…Type I and III interferons (IFN-I/λ) are important antiviral mediators against SARS-CoV-2 infection. …”
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    Severe COVID-19 patients have impaired plasmacytoid dendritic cell-mediated control of SARS-CoV-2
  19. 32899
    por Desch, Laura, Kunstfeld, Rainer
    Publicado 2013
    “…Up to now, emerging new therapy options as molecular-targeted agents, for example, pazopanib, imatinib, or somatostatin analogues as well as immunologicals, for example, imiquimod and interferons, also showed less success concerning the disease-free response rates. …”
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    Merkel Cell Carcinoma: Chemotherapy and Emerging New Therapeutic Options
  20. 32900
    “…Multiple PRR families, including Toll-like receptors (TLRs), RIG-I-like receptors (RLRs) and NOD-like receptors (NLRs), contribute significantly to viral detection, leading to induction of cytokines, chemokines and type I interferons (IFNs), which subsequently facilitate the eradication of the virus. …”
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    Host-Viral Interactions: Role of Pattern Recognition Receptors (PRRs) in Human Pneumovirus Infections
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