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  1. 3661
    “…Background: Pancreatic cancer represents the most lethal malignancy among all digestive cancers. …”
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    Herbals and Plants in the Treatment of Pancreatic Cancer: A Systematic Review of Experimental and Clinical Studies
  2. 3662
    “…The present cross-sectional prospective study (from January 2017 until December 2020) aimed to evaluate the interrelation between nutritional parameters at the initial evaluation with the changes in QOL indicators and the Progression-Free Survival and Overall Survival in 97 patients with pancreatic cancer. Dietary and nutritional risk assessments, and an evaluation of Frailty and Functional Status were performed. …”
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    Pancreatic Cancer Prognosis, Malnutrition Risk, and Quality of Life: A Cross-Sectional Study
  3. 3663
    “…Pancreatic cancer (PC) is one of the most lethal malignancies, the mortality and morbidity of which have been increasing over the past decade. …”
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    Ferroptosis-Related IncRNAs Are Prognostic Biomarker of Overall Survival in Pancreatic Cancer Patients
  4. 3664
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    Insufficiency of compound immune checkpoint blockade to overcome engineered T cell exhaustion in pancreatic cancer
  5. 3665
    “…BACKGROUND: Pancreatic cancer is a malignancy with one of the poorest prognoses amongst all cancers. …”
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    Stereotactic radiotherapy and the potential role of magnetic resonance-guided adaptive techniques for pancreatic cancer
  6. 3666
    “…To further understand the role of gastrokine proteins in PDAC development, GKN-deficient KC mice were developed by intercrossing gastrokine-deficient mice with KC mice. Panc-02 (pancreatic cancer cells of mouse origin) were genetically modified to express GKN1 for further in vitro and in vivo analysis. …”
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    De novo expression of gastrokines in pancreatic precursor lesions impede the development of pancreatic cancer
  7. 3667
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    p53-driven lipidome influences non-cell-autonomous lysophospholipids in pancreatic cancer
  8. 3668
    “…Here, we describe how ATP13A3, a P-type ATPase, functions as a polyamine transporter in response to different polyamine stimuli and polyamine-targeted therapies in highly proliferating pancreatic cancer cells. We assessed the expression, cellular localization and the response of the human ATP13A3 protein to polyamine treatments in different pancreatic cancer cell lines using Western blot and immunofluorescence microscopy. …”
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    ATP13A3 facilitates polyamine transport in human pancreatic cancer cells
  9. 3669
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    Altered Plasma Fatty Acid Abundance Is Associated with Cachexia in Treatment-Naïve Pancreatic Cancer
  10. 3670
    “…OBJECTIVE: Pancreatic cancer is portrayed to become the second leading cause of cancer-related death within the next years. …”
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    Additional MRI for initial M-staging in pancreatic cancer: a cost-effectiveness analysis
  11. 3671
    “…MST1r deregulation substantially impinged on drug response in the experimental model, recapitulating the p53(R270H)‐dependent phenotype, and strongly correlated with p53 mutant and aggressive phenotype in pancreatic cancer patients. As cellular plasticity in the final stages of the evolution of pancreatic cancer seems to predominantly originate from epigenetic mechanisms, we propose that mutant p53 participates in the acquisition of a lethal phenotype by fine‐tuning the chromatin landscape.…”
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    p53 mutations define the chromatin landscape to confer drug tolerance in pancreatic cancer
  12. 3672
    por Li, Maoquan
    Publicado 2021
    “…Pancreatic cancer has become a major disease affecting people's health because of its insidiousness, rapid progression and poor prognosis. …”
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    Clinical practice guidelines for the interventional treatment of advanced pancreatic cancer (5(th) edition)
  13. 3673
    “…Nonetheless, the effect of bile acids on the occurrence and development of pancreatic cancer remains a matter of debate. In the present study, to explore the direct effects of high serum concentrations of bile acids on pancreatic cancer and the possible related mechanisms, human pancreatic cancer (PANC-1) cells were subject to different concentrations of bile acid mixtures to assess cell viability and the migration and invasion ability. …”
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    A high bile acid environment promotes apoptosis and inhibits migration in pancreatic cancer
  14. 3674
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    Patient‐derived organoids, creating a new window of opportunities for pancreatic cancer patients
  15. 3675
    “…By adding a mechanistic layer to precision oncology, adaptive processes of tumors under therapy can be exploited, particularly in highly plastic tumors, such as pancreatic cancer.…”
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    Identification of treatment‐induced vulnerabilities in pancreatic cancer patients using functional model systems
  16. 3676
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    Isoforms of the orphan nuclear receptor COUP-TFII differentially modulate pancreatic cancer progression
  17. 3677
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    Sympathetic axonal sprouting induces changes in macrophage populations and protects against pancreatic cancer
  18. 3678
    “…BACKGROUND: The relationship between pancreatic cancer (PC) and type 2 diabetes mellitus (T2DM) has long been widely recognized, but the interaction mechanisms are still unknown. …”
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    Identification of the Shared Gene Signatures and Biological Mechanism in Type 2 Diabetes and Pancreatic Cancer
  19. 3679
    por Luo, Wenhao, Wang, Yawen, Zhang, Taiping
    Publicado 2022
    “…Pancreatic cancer (PC) is a growing global burden, remaining one of the most lethal cancers of the gastrointestinal tract. …”
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    Win or loss? Combination therapy does improve the oncolytic virus therapy to pancreatic cancer
  20. 3680
    “…Furthermore, a drug screening pipeline based on CIGER is proposed to identify potential treatments of drug-resistant pancreatic cancer. Our predictions have been validated by experiments, thereby showing the effectiveness of CIGER for phenotypic compound screening of precision medicine.…”
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    Chemical-induced gene expression ranking and its application to pancreatic cancer drug repurposing
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