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  1. 3961
    “…We hypothesised that miR-18a in the plasma is a potential biomarker in patients with pancreatic cancer. METHODS: miR-18a is located in the miR-17–92 cluster and reported to be highly expressed in pancreatic cancer tissues. …”
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    Novel diagnostic value of circulating miR-18a in plasma of patients with pancreatic cancer
  2. 3962
    “…BACKGROUND: The retroperitoneal margin is a common site of positive surgical margins in patients with resectable pancreatic cancer. Preoperative margin-intensive therapy (MIT) involves delivery of a single high dose of ablative radiotherapy (30 Gy) focused on this surgically inaccessible margin, utilizing stereotactic techniques in an effort to reduce local failure following surgery. …”
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    Dosimetric and motion analysis of margin-intensive therapy by stereotactic ablative radiotherapy for resectable pancreatic cancer
  3. 3963
    “…BACKGROUND: Pancreatic cancer is a deadly disease characterised by high incidence of TP53 mutations. …”
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    The proline TP53 variant stimulates likely lymphangiogenesis in an orthotopic mouse model of pancreatic cancer
  4. 3964
    “…The aim of this paper is to present the early data on a protocol in progress in patients with resected pancreatic cancer.…”
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    Intraperitoneal Gemcitabine Chemotherapy Treatment for Patients with Resected Pancreatic Cancer: Rationale and Report of Early Data
  5. 3965
    “…We compared prospectively the utility of two platforms for CTC enumeration and characterisation in pancreatic cancer patients in a pilot exploratory study. …”
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    A pilot study to explore circulating tumour cells in pancreatic cancer as a novel biomarker
  6. 3966
    “…MUC4 is a large transmembrane type I glycoprotein that is overexpressed in pancreatic cancer (PC) and has been shown to be associated with its progression and metastasis. …”
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    Role of MUC4-NIDO domain in the MUC4-mediated metastasis of pancreatic cancer cells
  7. 3967
    “…O-linked glycans of secreted and membrane bound proteins play an important role in the pathogenesis of pancreatic cancer by modulating immune responses, inflammation, and tumorigenesis. …”
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    Overexpression of GalNAc-transferase GalNAc-T3 Promotes Pancreatic Cancer Cell Growth
  8. 3968
    “…However, its effect is limited as some cancers including pancreatic cancer show de novo resistance to TRAIL induced apoptosis. …”
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    Glycogen Synthase Kinase-3 Inhibition Sensitizes Pancreatic Cancer Cells to TRAIL-Induced Apoptosis
  9. 3969
    “…Cancer cell invasion and metastasis are the most important adverse prognostic factors for pancreatic cancer. Identification of biomarkers associated with outcome of pancreatic cancer may provide new approaches and targets for anticancer therapy. …”
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    RhoT1 and Smad4 Are Correlated with Lymph Node Metastasis and Overall Survival in Pancreatic Cancer
  10. 3970
    por Lightsey, D
    Publicado 2012
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    Comment on ‘Red and processed meat consumption and risk of pancreatic cancer: meta-analysis of prospective studies’
  11. 3971
    por Wei, Peng, Tang, Hongwei, Li, Donghui
    Publicado 2012
    “…To identify genetic susceptibility factors for pancreatic cancer, we conducted pathway analysis of genome-wide association study (GWAS) data in 3,141 pancreatic cancer patients and 3,367 controls with European ancestry. …”
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    Insights into Pancreatic Cancer Etiology from Pathway Analysis of Genome-Wide Association Study Data
  12. 3972
    “…CONCLUSIONS: We have constructed a robust quantitative MS profiling system and used it to validate alterations of modified apolipoproteins in multiple cohorts of patients with pancreatic cancer.…”
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    Altered Plasma Apolipoprotein Modifications in Patients with Pancreatic Cancer: Protein Characterization and Multi-Institutional Validation
  13. 3973
    “…Chiefly, our discoveries establish a role for APLP2 in the growth of pancreatic cancer cells and show that inhibitors preventing APLP2 cleavage reduce the viability of pancreatic cancer cells.…”
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    Relevance of amyloid precursor-like protein 2 C-terminal fragments in pancreatic cancer cells
  14. 3974
    “…These findings demonstrate that chemically targeting developmental regulators of exocrine pancreas can be translated into an approach with potential impact on therapeutic response in pancreatic cancer, and suggest that counteracting the pro-malignant side effect of HDAC inhibitors can enhance their anti-tumor activity.…”
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    Targeting developmental regulators of zebrafish exocrine pancreas as a therapeutic approach in human pancreatic cancer
  15. 3975
    “…BACKGROUND: Pancreatic cancer is a highly malignant disease with an extremely poor prognosis. …”
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    Class I and Class II Histone Deacetylases Are Potential Therapeutic Targets for Treating Pancreatic Cancer
  16. 3976
    por JIANG, LIXIN, HU, BING, GUO, QIAN, CHEN, LI
    Publicado 2013
    “…The purpose of this study was to determine whether high-intensity focused ultrasound (HIFU) is an effective treatment for pancreatic cancer in an athymic nude mouse model. Female athymic nude mice (n=44) were inoculated subcutaneously with 5–7×10(6) SW1990 human pancreatic cancer cells. …”
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    Treatment of pancreatic cancer in a nude mouse model using high-intensity focused ultrasound
  17. 3977
    “…Clusterin inhibition could augment the chemosensitivity of human pancreatic cancer cells by inhibition of clusterin-dependent pERK1/2 activation. …”
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    Knockdown of clusterin sensitizes pancreatic cancer cells to gemcitabine chemotherapy by ERK1/2 inactivation
  18. 3978
    “…A human pancreatic cancer cell line (Capan-1) was injected into the spleen of mice after hepatic ischemia-reperfusion (I/R group). …”
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    Increased E-selectin in hepatic ischemia-reperfusion injury mediates liver metastasis of pancreatic cancer
  19. 3979
    “…Gemcitabine resistance is a common problem of pancreatic cancer chemotherapy, and how to reverse it plays an important role in the treatment of pancreatic cancer. …”
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    Emodin reverses gemcitabine resistance in pancreatic cancer cells via the mitochondrial apoptosis pathway in vitro
  20. 3980
    “…Treatment of MiaPaCa-2 and Panc-1 pancreatic cancer cell lines with CDDO-Me induced the production of hydrogen peroxide and superoxide anions and inhibited the telomerase activity. …”
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    Inhibition of Telomerase Activity by Oleanane Triterpenoid CDDO-Me in Pancreatic Cancer Cells is ROS-Dependent
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