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  1. 1141
    “…Pancreatic ductal adenocarcinoma (PDAC), the most common type of pancreatic cancer, is the 4th most frequent cause of cancer-related death worldwide, primarily due to the inherent chemoresistant nature and metastatic capacity of this tumor. …”
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    The Ever-Evolving Concept of the Cancer Stem Cell in Pancreatic Cancer
  2. 1142
    “…Pancreatic cancer, most commonly referring to pancreatic ductal adenocarcinoma (PDAC), remains one of the most deadly diseases, with very few effective therapies available. …”
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    Precision Immuno-Oncology: Prospects of Individualized Immunotherapy for Pancreatic Cancer
  3. 1143
    por Kandel, Pujan, Wallace, Michael B.
    Publicado 2018
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    Advanced EUS Guided Tissue Acquisition Methods for Pancreatic Cancer
  4. 1144
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    Precision medicine meets the DNA damage response in pancreatic cancer
  5. 1145
    “…Pancreatic cancer is known as one of the most lethal cancers in the world. …”
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    Engineered T lymphocytes eliminate lung metastases in models of pancreatic cancer
  6. 1146
    “…OBJECTIVES: The aim of this research was to study whether plasma microRNAs (miRNA) can be used for early detection of pancreatic cancer (PC) by analyzing prediagnostic plasma samples collected before a PC diagnosis. …”
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    Plasma Micro-RNA Alterations Appear Late in Pancreatic Cancer
  7. 1147
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    Distinct epigenetic landscapes underlie the pathobiology of pancreatic cancer subtypes
  8. 1148
    “…Pancreatic cancer remains one of the most malignant tumors with a poor prognosis. …”
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    Microarray expression profile analysis of circular RNAs in pancreatic cancer
  9. 1149
    “…With dismal survival rate pancreatic cancer remains one of the most aggressive and devastating malignancy. …”
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    Liquid biopsy in pancreatic cancer: the beginning of a new era
  10. 1150
    “…The aim of this study was to evaluate the efficacy of Abraxane(®) (nab-paclitaxel) in Italian patients with metastatic pancreatic cancer (MPC). PATIENTS AND METHODS: We conducted a retrospective analysis of 80 patients. …”
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    Clinical efficacy of nab-paclitaxel in patients with metastatic pancreatic cancer
  11. 1151
    por Matters, Gail L., Harms, John F.
    Publicado 2018
    “…G protein–coupled receptors (GPCRs), which are key target proteins for drug discovery and comprise a large proportion of currently marketed therapeutics, hold significant promise for tumor imaging and targeted treatment, particularly for pancreatic cancer.…”
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    Utilizing Peptide Ligand GPCRs to Image and Treat Pancreatic Cancer
  12. 1152
    “…BACKGROUND: The morbidity and mortality of pancreatic cancer vary considerably around the world. The aim of this study was to characterize and evaluate recent changes in incidence and incidence-based mortality in the USA. …”
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    Rising trends in pancreatic cancer incidence and mortality in 2000–2014
  13. 1153
    por Mueller, Sebastian, Engleitner, Thomas, Maresch, Roman, Zukowska, Magdalena, Lange, Sebastian, Kaltenbacher, Thorsten, Konukiewitz, Björn, Öllinger, Rupert, Zwiebel, Maximilian, Strong, Alex, Yen, Hsi-Yu, Banerjee, Ruby, Louzada, Sandra, Fu, Beiyuan, Seidler, Barbara, Götzfried, Juliana, Schuck, Kathleen, Hassan, Zonera, Arbeiter, Andreas, Schönhuber, Nina, Klein, Sabine, Veltkamp, Christian, Friedrich, Mathias, Rad, Lena, Barenboim, Maxim, Ziegenhain, Christoph, Hess, Julia, Dovey, Oliver M., Eser, Stefan, Parekh, Swati, Constantino-Casas, Fernando, de la Rosa, Jorge, Sierra, Marta I., Fraga, Mario, Mayerle, Julia, Klöppel, Günter, Cadiñanos, Juan, Liu, Pentao, Vassiliou, George, Weichert, Wilko, Steiger, Katja, Enard, Wolfgang, Schmid, Roland M., Yang, Fengtang, Unger, Kristian, Schneider, Günter, Varela, Ignacio, Bradley, Allan, Saur, Dieter, Rad, Roland
    Publicado 2018
    “…Our study uncovers universal principles in Ras-driven oncogenesis with potential relevance beyond pancreatic cancer.…”
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    Evolutionary routes and KRAS dosage define pancreatic cancer phenotypes
  14. 1154
    por Kanat, Ozkan, Ertas, Hulya
    Publicado 2018
    “…Despite the availability of potent chemotherapy regimens, such as 5-fluorouracil, folinic acid, irinotecan, and oxaliplatin (FOLFIRINOX) and nab-paclitaxel plus gemcitabine, treatment outcomes in metastatic pancreatic cancer (PC) remain unsatisfactory. The presence of an abundant fibrous stroma in PC is considered a crucial factor for its unfavorable condition. …”
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    Shattering the castle walls: Anti-stromal therapy for pancreatic cancer
  15. 1155
    “…Our data indicate that GATA4 gene might play a role in cell proliferation and differentiation during the progression of pancreatic cancer.…”
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    GATA4 inhibits cell differentiation and proliferation in pancreatic cancer
  16. 1156
    “…Genome-wide association studies (GWASs) have identified many single nucleotide polymorphisms (SNPs) that are significantly associated with pancreatic cancer susceptibility. We sought to replicate the associations of 61 GWAS-identified SNPs at 42 loci with pancreatic cancer in Japanese and to develop a risk model for the identification of individuals at high risk for pancreatic cancer development in the general Japanese population. …”
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    Prediction model for pancreatic cancer risk in the general Japanese population
  17. 1157
    “…B cells have been described to infiltrate pancreatic cancer and may be associated with tertiary lymphoid structures associated with good prognosis, or, in contrast, promote tumor growth. …”
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    KRAS RENAISSANCE(S) in Tumor Infiltrating B Cells in Pancreatic Cancer
  18. 1158
    “…Our results acknowledge a decrease of drug side effects in normal cells and an enhancement of drug efficacy against to the pancreatic cancer cells reported.…”
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    Gold Nanoparticles for Targeting Varlitinib to Human Pancreatic Cancer Cells
  19. 1159
    “…There is accumulating evidence that deregulated Notch signaling affects cancer development, and specifically pancreatic cancer (PC) progression. Notch canonical and non-canonical signaling has diverse impact on PC. …”
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    The Role of Notch Signaling and Leptin-Notch Crosstalk in Pancreatic Cancer
  20. 1160
    “…Moreover, cerulein treatment increased PRDM14 expression in PK‐1 and AsPC‐1 pancreatic cancer cell lines. Our results suggest that inflammation increases the expression of PRDM14, which regulates cancer stem‐like phenotypes, and this occurs prior to PDAC initiation and progression.…”
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    PRDM14 is overexpressed in chronic pancreatitis prior to pancreatic cancer
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