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1581por Sahraei, Mahnaz, Roy, Lopamudra Das, Curry, Jennifer M, Teresa, Tinder L, Nath, Sritama, Besmer, Dahlia, Kidiyoor, Amritha, Dalia, Ritu, Gendler, Sandra J, Mukherjee, Pinku“…This, in turn, influences proliferation and invasion of pancreatic cancer cells leading to higher tumor burden in vivo. …”
Publicado 2012
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1582por Humphris, J. L., Chang, D. K., Johns, A. L., Scarlett, C. J., Pajic, M., Jones, M. D., Colvin, E. K., Nagrial, A., Chin, V. T., Chantrill, L. A., Samra, J. S., Gill, A. J., Kench, J. G., Merrett, N. D., Das, A., Musgrove, E. A., Sutherland, R. L., Biankin, A. V.“…BACKGROUND: Current staging methods for pancreatic cancer (PC) are inadequate, and biomarkers to aid clinical decision making are lacking. …”
Publicado 2012
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1583“…The prevalence of diabetes mellitus in pancreatic cancer patients and control subjects was compared. …”
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1584
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1585por Chiang, Kun-Chun, Yeh, Chun-Nan, Ueng, Shir-Hwa, Hsu, Jun-Te, Yeh, Ta-Sen, Jan, Yi-Yin, Hwang, Tsann-Long, Chen, Miin-FuEnlace del recurso
Publicado 2012
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1586The relationship between patient satisfaction with service quality and survival in pancreatic cancer“…We evaluated the relationship between patient satisfaction with health service quality and survival in patients with pancreatic cancer. PATIENTS AND METHODS: A random sample of 496 pancreatic cancer patients treated at Cancer Treatment Centers of America(®) (CTCA) between July 2007 and December 2010. …”
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1587“…INTRODUCTION: MicroRNAs have been reported to be aberrantly expressed in patients with pancreatic cancer. The aim of the present meta-analysis is to establish the overall diagnostic accuracy of the measurement of microRNA for diagnosing pancreatic cancer. …”
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1588“…Development and progression of pancreatic cancer involves general metabolic disorder, local chronic inflammation, and multistep activation of distinct oncogenic molecular pathways. …”
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1589por Iqbal, J, Ragone, A, Lubinski, J, Lynch, H T, Moller, P, Ghadirian, P, Foulkes, W D, Armel, S, Eisen, A, Neuhausen, S L, Senter, L, Singer, C F, Ainsworth, P, Kim-Sing, C, Tung, N, Friedman, E, Llacuachaqui, M, Ping, S, Narod, S A“…BACKGROUND: Germline mutations in BRCA1 and BRCA2 predispose to pancreatic cancer. We estimated the incidence of pancreatic cancer in a cohort of female carriers of BRCA1 and BRCA2 mutation. …”
Publicado 2012
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1590“…The aim of this study was to evaluate the effects and molecular mechanisms of everolimus on Panc-1 human pancreatic cancer cells. Panc-1 human pancreatic cancer cells were treated with everolimus (10 μg/ml) at selected time points (6, 12 and 24 h). …”
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1591por Papa, Anne-Laure, Basu, Sudipta, Sengupta, Poulomi, Banerjee, Deboshri, Sengupta, Shiladitya, Harfouche, Rania“…BACKGROUND: Pancreatic cancer remains the deadliest of all cancers, with a mortality rate of 91%. …”
Publicado 2012
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1592por Dai, Zhi-Jun, Gao, Jie, Ma, Xiao-Bin, Kang, Hua-Feng, Wang, Bao-Feng, Lu, Wang-Feng, Lin, Shuai, Wang, Xi-Jing, Wu, Wen-YingEnlace del recurso
Publicado 2012
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1593Gastrointestinal Hemorrhage after Concurrent Chemoradiotherapy in Locally Advanced Pancreatic Cancerpor Lee, Kyong Joo, Kim, Hee Man, Jung, Joo Won, Chung, Moon Jae, Park, Jeong Youp, Bang, Seungmin, Park, Seung Woo, Lee, Woo Jung, Seong, Jin Sil, Song, Si Young“…BACKGROUND/AIMS: While chemoradiotherapy (CRT) is considered to be a reasonable treatment for locally advanced pancreatic cancer (LAPC), there is little information about the associated risk of gastrointestinal (GI) hemorrhage. …”
Publicado 2013
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1594por DU, JIAN, TANG, BO, WANG, JINGWEN, SUI, HONGTAO, JIN, XUELI, WANG, LIMING, WANG, ZHONGYU“…However, the antitumor effect of alpinetin on pancreatic cancer cells and the detailed mechanism remain unclear. …”
Publicado 2012
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1595por Liu-Mares, Wen, MacKinnon, Jill A, Sherman, Recinda, Fleming, Lora E, Rocha-Lima, Caio, Hu, Jennifer J, Lee, David J“…BACKGROUND: We sought to identify high-risk areas of pancreatic cancer incidence, and determine if clusters of persons diagnosed with pancreatic cancer were more likely to be located near arsenic-contaminated drinking water wells. …”
Publicado 2013
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1596por Konkalmatt, Prasad R., Deng, Defeng, Thomas, Stephanie, Wu, Michael T., Logsdon, Craig D., French, Brent A., Kelly, Kimberly A.“…Gene therapy using AAV vectors to selectively deliver genes to PDAC cells is an attractive treatment option for pancreatic cancer. However, most AAV serotypes display a broad spectrum of tissue tropism and none of the existing serotypes specifically target PDAC cells. …”
Publicado 2013
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1597por Lee, Woo JinEnlace del recurso
Publicado 2013
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1598“…Of note, inhibition of TLR7 signaling in a mouse p48Cre;Kras(G12D) pancreatic cancer model protected against tumor progression thus paving the road for TLR-blocking strategies to combat tumors.…”
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1599“…However, the putative function of Gal-4 in tumor progression of pancreatic cancer is still incompletely understood. In this study the role of Gal-4 in cancer progression was investigated, using a set of defined pancreatic cancer cell lines, Pa-Tu-8988S (PaTu-S) and Pa-Tu-8988T (PaTu-T), as a model. …”
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1600por Kim, Yeon-Joo, Lee, Woo Jin, Woo, Sang Myung, Kim, Tae Hyun, Han, Sung-Sik, Kim, Bo Hyun, Moon, Sung Ho, Kim, Sang Soo, Koh, Young Hwan, Park, Sang-Jae, Kim, Joo-Young, Kim, Dae Yong, Park, Joong-Won“…BACKGROUND: Although capecitabine has theoretical advantages in the pharmacokinetics, such as higher intratumoral and lower systemic concentration, relative to bolus 5-fluorouracil (5-FU), outcomes of chemoradiotherapy (CRT) with capecitabine or bolus 5-FU have not been directly compared in patients with locally advanced pancreatic cancer. Therefore, we retrospectively compared the outcomes, including toxicity, tumor response, and overall survival, of oral capecitabine plus radiotherapy (RT) with bolus 5-FU plus RT, in patients with locally advanced pancreatic cancer. …”
Publicado 2013
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