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  1. 1641
    “…Emerging laboratory and animal studies indicate that green tea inhibits development and progression of pancreatic cancer, but evidence from epidemiologic studies appears inconsistent and inconclusive. …”
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    Green Tea Consumption and Risk of Pancreatic Cancer: A Meta-analysis
  2. 1642
    “…OBJECTIVES: The aim of this study was to investigate whether 6 candidate serum miRNAs and their interactions with serum folate level were associated with the risk for pancreatic cancer (PC). METHOD: A hospital-based case-control study including 74 incident PC cases and 74 controls was conducted. …”
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    Interaction of Serum microRNAs and Serum Folate With the Susceptibility to Pancreatic Cancer
  3. 1643
    por Furukawa, Toru
    Publicado 2015
    “…Pancreatic cancer is characterized by constitutive activation of the mitogen-activated protein kinase (MAPK) pathway. …”
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    Impacts of Activation of the Mitogen-Activated Protein Kinase Pathway in Pancreatic Cancer
  4. 1644
    “…BACKGROUND: Previous studies regarding the association between cruciferous vegetable intake and pancreatic cancer risk have reported inconsistent results. …”
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    Cruciferous vegetable consumption and the risk of pancreatic cancer: a meta-analysis
  5. 1645
    “…The role of DEPTOR in the growth and survival of pancreatic cancer cells has not previously been determined. …”
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    DEPTOR has growth suppression activity against pancreatic cancer cells
  6. 1646
    Publicado 2015
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    Correction: Targeting and Cytotoxicity of SapC-DOPS Nanovesicles in Pancreatic Cancer
  7. 1647
    “…It has long been regarded that pancreatic cancer (PC) is a life-threatening malignant tumor. …”
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    Expression of c-fos Was Associated with Clinicopathologic Characteristics and Prognosis in Pancreatic Cancer
  8. 1648
    por Liou, Geou-Yarh, Storz, Peter
    Publicado 2015
    “…This oncogene-caused chronic microinflammation can accelerate the pathogenesis of pancreatic cancers.…”
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    Inflammatory macrophages in pancreatic acinar cell metaplasia and initiation of pancreatic cancer
  9. 1649
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    Exploiting Base Excision Repair to Improve Therapeutic Approaches for Pancreatic Cancer
  10. 1650
    “…BACKGROUND: Pancreatic cancer accompanied by a moderate-sized pseudocyst with extrapancreatic growth is extremely rare. …”
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    Pancreatic cancer accompanied by a moderate-sized pseudocyst with extrapancreatic growth
  11. 1651
    “…The aim of the present study was the direct covalent coupling of the epidermal growth factor receptor (EGFR)-specific monoclonal antibody (mAb) to the surface of poly(lactide)-co-glycolide (PLGA)-polyethylene glycol (PEG) nanoparticles in order to achieve a cell type-specific drug carrier system against pancreatic cancer. The PLGA-PEG-NH(2) diblock copolymer was synthesized by coupling reaction via amide linkage between PEG-diamine and activated PLGA. …”
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    Gemcitabine-loaded PLGA-PEG immunonanoparticles for targeted chemotherapy of pancreatic cancer
  12. 1652
    “…In conclusion, COMT suppressed pancreatic cancer and its high expression predicted longer survival time. …”
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    Catechol-O-methyltransferase, a new target for pancreatic cancer therapy
  13. 1653
    “…Therefore, BRCA1/2 mutation carriers with pancreatic cancer may have distinct biologic outcomes. …”
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    Overall survival and clinical characteristics of pancreatic cancer in BRCA mutation carriers
  14. 1654
    por Arem, Hannah, Yu, Kai, Xiong, Xiaoqin, Moy, Kristin, Freedman, Neal D., Mayne, Susan T., Albanes, Demetrius, Amundadottir, Laufey T., Arslan, Alan A., Austin, Melissa, Bamlet, William R., Beane-Freeman, Laura, Bracci, Paige, Canzian, Federico, Chanock, Stephen J, Cotterchio, Michelle, Duell, Eric J., Gallinger, Steve, Giles, Graham G., Goggins, Michael, Goodman, Phyllis J., Hartge, Patricia, Hassan, Manal, Helzlsouer, Kathy, Henderson, Brian, Holly, Elizabeth A., Hoover, Robert, Jacobs, Eric J., Kamineni, Aruna, Klein, Alison, Klein, Eric, Kolonel, Laurence N., Li, Donghui, Malats, Núria, Männistö, Satu, McCullough, Marjorie L., Olson, Sara H., Orlow, Irene, Peters, Ulrike, Petersen, Gloria M., Porta, Miquel, Severi, Gianluca, Shu, Xiao-Ou, Van Den Eeden, Stephen, Visvanathan, Kala, White, Emily, Yu, Herbert, Zeleniuch-Jacquotte, Anne, Zheng, Wei, Tobias, Geoffrey S., Maeder, Dennis, Brotzman, Michelle, Risch, Harvey, Sampson, Joshua N., Stolzenberg-Solomon, Rachael Z.
    Publicado 2015
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    Correction: Vitamin D Metabolic Pathway Genes and Pancreatic Cancer Risk
  15. 1655
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    Use of Radiographic Criteria to Predict Outcomes Following Surgery for Pancreatic Cancer
  16. 1656
    “…In conclusion, Fusobacterium species were detected in pancreatic cancer tissue. Tumor Fusobacterium species status is independently associated with a worse prognosis of pancreatic cancer, suggesting that Fusobacterium species may be a prognostic biomarker of pancreatic cancer.…”
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    Association of Fusobacterium species in pancreatic cancer tissues with molecular features and prognosis
  17. 1657
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    Identification of artesunate as a specific activator of ferroptosis in pancreatic cancer cells
  18. 1658
    “…Gemcitabine (GEM) is currently the standard option for the treatment of pancreatic cancer but its short half-life and rapid metabolism has caused for new modality for delivery of GEM. …”
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    Cytotoxicity of gemcitabine-loaded thermosensitive liposomes in pancreatic cancer cell lines
  19. 1659
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    Targeting of surface alpha-enolase inhibits the invasiveness of pancreatic cancer cells
  20. 1660
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    Modeling Combined Chemotherapy and Particle Therapy for Locally Advanced Pancreatic Cancer
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