“…GAD antibodies, Islet cell antibodies and Zinc transporter 8 antibodies were negative. Renal function was normal. She responded well to basal insulin treatment, with an achieved Hb A1c of 7%. …”
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“…Genital examination, newborn screen, ECG, renal US, thyroid function, 17-OH progesterone, and ACTH stimulation tests were normal. …”
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“…Introduction Fibroblast Growth Factor 23 (FGF23) is a hormone which controls phosphate homeostasis by increasing renal excretion of phosphate and preventing hydroxylation of 25-OH vitamin D. …”
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“…The risk of a subsequent new diagnosis of cancer or hospital admission with incident cardiovascular, renal or liver disease was not related to glucose category. …”
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“…In parallel, the patient had a diagnosis of primary hyperparathyroidism with serum calcium of 10.7mg/dL(8.6-10.2), vitamin D 29.8ng/dL(>20), PTH 115pg/ml(15-65) and serum phosphorus of 2.2mg/dL(2.8-4.1), and normal renal function. 99mTc-sestamibi scan showed an increased parathyroid gland posterior to the superior pole of the right lobe of the thyroid. …”
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“…METHODS: In 66 adults with ≥50 years type 1 diabetes (T1D) duration and 73 nondiabetic controls from age/sex-matched subgroups (65 ± 8 years old and 77[55%] were women) we evaluated the performance of estimated GFR (eGFR) by creatinine (Modification of Diet and Renal Disease [MDRD], Chronic Kidney Disease–Epidemiology [CKD-EPI](cr)), cystatin C (CKD-EPI(cys), CKD-EPI(cr-cys)), and β(2)-microglobulin (β2M) compared with measured GFR by inulin clearance (mGFR). …”
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“…Laboratory analysis showed a serum calcium of 12.8 mg/dl (8.5-10.5), albumin of 3.8 g/dl (3.5-5), mild transaminitis and normal renal function. A contrasted CT of the abdomen and pelvis revealed a 26 cm right ovarian mass, hypodense lesions throughout the liver, moderate ascites and omental fat nodularity worrisome for ovarian malignancy and peritoneal carcinomatosis. …”
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“…Cardiac catheterization was deferred due to declining renal function. Subsequent myocardial perfusion imaging displayed only an apical cap artifact and coronary CT angiogram was free from atherosclerosis. …”
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“…Conclusion: Based on pathophysiology, hypokalemic paralysis is divided into 2 groups: hypokalemic periodic paralysis due to acute shift of potassium into cells, and hypokalemic non-periodic paralysis resulting from a large total body deficit of potassium. 25% of cases of hypokalemic paralysis are secondary to hypokalemic non-periodic paralysis associated with chronic alcoholism, diuretic use, anorexia/bulimia nervosa, renal tubular acidosis, primary hyperaldosteronism, and Gitelman syndrome. …”
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“…Pseudohypoparathyroidism 1B (PHP1B) or inactivating PTH/PTHrp Signaling Disorder type 3 (iPPSD3), according to the new classification, presents mainly with hypocalcemia and hyperphosphatemia due to renal resistance towards parathyroid hormone. In addition, patients display early onset obesity and impaired pubertal growth. …”
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“…Conclusion: Patients with osteoblastic metastases and renal impairment are at particularly increased risk of hypocalcemia after denosumab, which can be potentially life-threatening. …”
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“…SDHD mutation also predisposes to renal cell carcinoma and pituitary adenoma. Head and neck PGL are mostly non-secretory (95%), however, 3-methoxytyramine is a new marker that has been found to be elevated in one-third of head and neck PGL. …”
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“…Continuous veno-venous hemofiltration (CVVH) was initiated for anuric renal failure. She required ventilator and pressor support on day 7 (D7) of hospitalization as urgent preparations were being made for LVAD exchange (given LVAD malfunction). …”
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“…FH is associated with autosomal dominant hereditary leiomyomatosis and renal cell cancer (HLRCC), and has early evidence of correlation with hereditary PCC/PGL. …”
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“…Background: Empagliflozin is a sodium glucose co-transporter 2 (SGLT-2) inhibitor that works in the proximal renal tubules reducing resorption of glucose and increasing urinary glucose excretion. …”
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“…This may be compounded by altered renal glucose threshold in sick infants. References: 1. …”
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“…Secondary hyperparathyroidism is mostly seen with renal failure and hypovitaminosis D. Skeletal complications of hyperparathyroidism are rare with better screening, and include osteitis fibrosa cystica, bone cysts and brown tumors; the later are composed of numerous multinucleated giant cells admixed with stromal cells and matrix, often seen in the jaw, long bones and ribs. …”
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“…Clinical Case: An 82 year-old woman with a history of renal cell carcinoma and recent post-operative pulmonary embolism treated with apixiban, presented with acute-onset headache, diplopia, and vomiting. …”
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“…Urine culture from left renal pelvis also grew C. glabrata sensitive to fluconazole. …”
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“…DISCUSSION: PS has been associated with malignancies, liver disease, infections, malabsorption, pulmonary or renal glucagonoma and pancreatitis. The pathogenesis of NME is not clear, but is believed to be multifactorial, involving excess of glucagon, deficiency of amino acid, zinc and/or free fatty acids. …”
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