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1481por Song, CherynEnlace del recurso
Publicado 2023
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1482
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1483
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1484por Kurttio, Päivi, Auvinen, Anssi, Salonen, Laina, Saha, Heikki, Pekkanen, Juha, Mäkeläinen, Ilona, Väisänen, Sari B, Penttilä, Ilkka M, Komulainen, Hannu“…However, more information about its renal effects in humans following chronic exposure through drinking water is required. …”
Publicado 2002
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1485“…In particular, there is concern that high protein intake may promote renal damage by chronically increasing glomerular pressure and hyperfiltration. …”
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1486
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1487por Oken, D E“…Renal micropuncture studies have greatly changed our views on the pathophysiology of acute renal failure caused by nephrotoxins. …”
Publicado 1976
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1488“…Immunotherapy results in a small overall survival advantage in metastatic renal cell carcinoma (RCC), but there is a need to develop more effective systemic therapies. …”
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1489
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1490por Linnebjerg, Helle, Kothare, Prajakti A, Park, Soomin, Mace, Kenneth, Reddy, Shobha, Mitchell, Malcolm, Lins, Robert“…What is already known about this subject Nonclinical studies have shown that exenatide is primarily cleared by the renal system. It was not known to what degree the clinical pharmacokinetics and tolerability would be affected by increasing renal impairment (RI). …”
Publicado 2007
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1491“…A series of 33 patients with metastatic renal cancer and evidence of progression of the disease—apart from pulmonary metastases—was treated with hormones (progestogens in 31 cases, androgens in 2 cases) at the Rotterdamsch Radio-Therapeutisch Instituut. …”
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1492“…A primary renal tumour of childhood with histological appearances different from the nephroblastoma is described. …”
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1493“…Analysis of data from 1434 children with primary renal tumours revealed 57 who developed bone metastases. …”
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1494“…Forty-six separate renal tumours developed in 36/80 Wistar male rats given a single i.v. dose of streptozotocin (25 mg/kg body wt) to induce diabetes mellitus. …”
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1495“…It is well known that ouabain, a specific inhibitor of Na-K ATPase-dependent transport, interferes with renal tubular salt reabsorption. In this study, we employed radiochemical methods to measure the kinetics of [3H]ouabain binding to slices of rabbit renal medulla and high resolution quantitative autoradiography to determine the location and number of cellular binding sites. …”
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1496
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1497“…Our results lead us to believe that renin in the form in which it is extracted from the kidney cannot be the agent causing chronic renal hypertension. The reasons against accepting renin as the pressor substance responsible for the hypertension of renal ischemia may be summed up as follows:— 1. …”
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1498“…The chemical mediator of renal hypertension is rapidly destroyed by the metabolic activity of the normal kidney. …”
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1499“…A drop has been observed in the renal threshold for hemoglobin in dogs, of over 60 per cent, following repeated injections daily. …”
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1500“…Acute renal hypertension is produced by the injection of the amino acid dopa (l-dihydroxyphenylalanine) into the partially or completely ischemic kidney of the cat. …”
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