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  1. 2401
    “…Additionally, PQ was found to induce astrocytic senescence and an SASP in vitro and in vivo, and senescent cell depletion in the latter protects against PQ-induced neuropathology. …”
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    Cellular Senescence Is Induced by the Environmental Neurotoxin Paraquat and Contributes to Neuropathology Linked to Parkinson’s Disease
  2. 2402
    “…Senescent cells secrete a variety of inflammatory factors, collectively referred to as the senescence-associated secretory phenotype (SASP). …”
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    Innate immune sensing of cytosolic chromatin fragments through cGAS promotes senescence
  3. 2403
    “…In PLWHIV with expansion of CMV-specific T-cells or increased T-cell senescence, CMV-specific polyfunctionality was maintained. …”
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    Cytomegalovirus-specific T-cells are associated with immune senescence, but not with systemic inflammation, in people living with HIV
  4. 2404
    “…Timely regulated senescence is thought to be beneficial, whereas chronic senescence such as during normal or premature aging is deleterious as it favors most, if not all, age-related diseases. …”
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    Transcriptional repression of DNA repair genes is a hallmark and a cause of cellular senescence
  5. 2405
    “…In this article, we discuss these issues and work through the process of building an integrated model for cellular senescence as a single cell and in a simple tissue context.…”
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    Systems modelling ageing: from single senescent cells to simple multi-cellular models
  6. 2406
    “…Autophagy, cellular senescence, programmed cell death and necrosis are key responses of a cell facing a stress. …”
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    An Interplay between Senescence, Apoptosis and Autophagy in Glioblastoma Multiforme—Role in Pathogenesis and Therapeutic Perspective
  7. 2407
    “…Aging involves tissue accumulation of senescent cells (SC) whose elimination through senolytic approaches may evoke organismal rejuvenation. …”
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    Induction, regulation and roles of neural adhesion molecule L1CAM in cellular senescence
  8. 2408
    “…Ophiocordyceps sinensis (O. sinensis) seems to be able to alleviate airway epithelial cell senescence in chronic obstructive pulmonary disease (COPD). …”
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    The Function of Ophiocordyceps sinensis in Airway Epithelial Cell Senescence in a Rat COPD Model
  9. 2409
    “…Herein, we will focus on current repository of genomic “senescence” stage of aging, which includes health decline, structural changes of the genome, faulty DNA damage response and DNA damage, telomere shortening, and epigenetic alterations. …”
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    Genomic Instabilities, Cellular Senescence, and Aging: In Vitro, In Vivo and Aging-Like Human Syndromes
  10. 2410
    “…Furthermore, we revealed that baicalin-induced senescence in tumor cells is due to its inhibition of telomerase reverse transcriptase expression in tumor cells, and that MAPK ERK and p38 signaling pathways are causatively involved in the regulation of colon cancer cell apoptosis and senescence mediated by baicalein and baicalin. …”
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    Baicalein and baicalin inhibit colon cancer using two distinct fashions of apoptosis and senescence
  11. 2411
    “…Telomere length and the rate of telomere shortening have been suggested as particularly useful physiological biomarkers of the processes involved in senescent decline of somatic and reproductive function. …”
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    Telomere dynamics in wild banded mongooses: Evaluating longitudinal and quasi-longitudinal markers of senescence
  12. 2412
    “…In the H358 and H441 cells, which expressed TTF-1 and E-cadherin, survivin knockdown alone induced senescence, not apoptosis. However, in monolayer culture, the H358 cells and H441 cells in which survivin was silenced, underwent significant apoptosis following combined treatment with ABT-263, a Bcl-2 inhibitor, and trametinib, a MEK inhibitor. …”
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    Survivin knockdown induces senescence in TTF-1-expressing, KRAS-mutant lung adenocarcinomas
  13. 2413
    “…Results showed that senescing leaves can accumulate large amounts of luteoloside, extremely higher than that in young and semi-lignified leaves and other tissues. …”
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    Transcriptome Analysis Reveals Molecular Signatures of Luteoloside Accumulation in Senescing Leaves of Lonicera macranthoides
  14. 2414
    “…Genetic ablation of the tumor suppressor PTEN in prostatic epithelial cells (PECs) induces cell senescence. However, unlike oncogene-induced senescence, no hyperproliferation phase and no signs of DNA damage response (DDR) were observed in PTEN-deficient PECs; PTEN loss-induced senescence (PICS) was reported to be a novel type of cellular senescence. …”
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    PTEN deletion in luminal cells of mature prostate induces replication stress and senescence in vivo
  15. 2415
    por Wang, Yan, Zhang, Yuanyuan, Wang, Lei
    Publicado 2018
    “…Circadian clock and senescence have been shown to tightly intertwined with each other in numerous eukaryotes, but the regulation of circadian oscillator on triggering leaf senescence, and vice versa, remains largely unknown in higher plants. …”
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    Cross Regulatory Network Between Circadian Clock and Leaf Senescence Is Emerging in Higher Plants
  16. 2416
    “…Various molecular pathways can induce senescence and these different pathways dictate the phenotypic and metabolic changes that accompany the transition to, and maintenance of, the senescence state. …”
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    Loss of the ubiquitin conjugating enzyme UBE2E3 induces cellular senescence
  17. 2417
    “…Autumn senescence of deciduous trees is characterized by chlorophyll degradation and flavonoid synthesis. …”
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    Degradation of chlorophyll and synthesis of flavonols during autumn senescence—the story told by individual leaves
  18. 2418
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    Embryonic senescent cells re-enter cell cycle and contribute to tissues after birth
  19. 2419
    “…Besides photoprotecting, we propose that epidermal anthocyanins may be principally synthetized to maintain an efficient carbon-sink strength in young and senescent leaves, thus extending the RLP leaf lifespan.…”
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    Multiple Consequences Induced by Epidermally-Located Anthocyanins in Young, Mature and Senescent Leaves of Prunus
  20. 2420
    “…After 4 weeks, nuclear γH2AX deposition and senescence‐associated β‐galactosidase staining indicative of DNA damage and premature senescence, respectively, were increased in the aortic endothelium of L5‐treated but not L1‐treated mice. …”
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    Human electronegative LDL induces mitochondrial dysfunction and premature senescence of vascular cells in vivo
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