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  1. 2641
    “…HDAC4 overexpression rescued the senescence-induced decrease in DDIT4 and senescence phenotype, which were prevented by DDIT4 knockdown. …”
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    Histone deacetylase 4 reverses cellular senescence via DDIT4 in dermal fibroblasts
  2. 2642
    “…Notably, within the LT-HCC group, a higher percentage of senescence-like T cells was also associated with cancer development. …”
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    Immune Activation, Exhaustion and Senescence Profiles as Possible Predictors of Cancer in Liver Transplanted Patients
  3. 2643
    “…Mouse embryonic fibroblasts establish 40 senescence-associated super-enhancers regardless of how they become senescent, with 50 activated genes located in the vicinity of these enhancers. …”
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    Senescent cells limit p53 activity via multiple mechanisms to remain viable
  4. 2644
    “…OBJECTIVE: To examine whether the senescence-associated secretory phenotype (SASP) index was associated with the likelihood of remission from a depressive episode in older adults. …”
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    Association of Molecular Senescence Markers in Late-Life Depression With Clinical Characteristics and Treatment Outcome
  5. 2645
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    Correction: Increase of circulating IGFBP-4 following genotoxic stress and its implication for senescence
  6. 2646
    “…Collectively, we show that T cell cholesterol efflux pathways suppress T cell apoptosis and senescence, and induce atherosclerosis in middle-aged Ldlr(−/−) mice.…”
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    T cell cholesterol efflux suppresses apoptosis and senescence and increases atherosclerosis in middle aged mice
  7. 2647
    “…Senescence progression is accompanied by a peculiar decrease in the K(+) content in cells from 800–900 to 500–600 µmol/g. …”
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    Monovalent ions and stress-induced senescence in human mesenchymal endometrial stem/stromal cells
  8. 2648
    “…In the present review, we introduce the most common aging and senescence-accelerated animal models and emphasize that sex is a biological variable that should be considered in aging studies. …”
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    Vascular Aging in Rodent Models: Contrasting Mechanisms Driving the Female and Male Vascular Senescence
  9. 2649
    por Shen, Shuang, Ji, Chuanyuan, Wei, Kaifeng
    Publicado 2022
    “…Cellular senescence is frequently evident at etiologic sites of chronic diseases and involves essentially irreversible arrest of cell proliferation, increased protein production, resistance to apoptosis, and altered metabolic activity. …”
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    Cellular Senescence and Regulated Cell Death of Tubular Epithelial Cells in Diabetic Kidney Disease
  10. 2650
    por Hajibarat, Zohreh, Saidi, Abbas
    Publicado 2022
    “…BACKGROUND: Plants use escape strategies including premature senescence and leaf reduction to cope in response to drought stress, which in turn reduces plant leaves and photosynthesis. …”
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    Senescence-associated proteins and nitrogen remobilization in grain filling under drought stress condition
  11. 2651
    “…BACKGROUND: As one of the core aging processes, cellular senescence is associated with tumorigenesis, growth, and immune modulation in cancers. …”
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    Identification of several senescence‐associated genes signature in head and neck squamous cell carcinoma
  12. 2652
    “…Oxidative stress is a primary cause of cellular senescence and contributes to the etiology of numerous human diseases. …”
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    Telomeric 8-oxo-guanine drives rapid premature senescence in the absence of telomere shortening
  13. 2653
    “…This observation supports that genetic and epigenetic incident favors endometrial epithelia cells escape from senescence and fuel EMT process in endometriosis, what could be overcome by downregulation of SIRT1.…”
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    SIRT1 upregulation promotes epithelial-mesenchymal transition by inducing senescence escape in endometriosis
  14. 2654
    “…We speculated that PTEN might promote SKOV3 cell senescence by increasing TRIM39 expression and decreasing P21 degradation. …”
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    Anti‐oncogenic PTEN induces ovarian cancer cell senescence by targeting P21
  15. 2655
    “…Activated protein C pretreatment maintained barrier permeability and prevented glucose-induced expression of senescence and UPR markers in vitro. These data suggest that high glucose-induced maladaptive UPR and associated senescence promote vascular endothelial cell dysfunction, which—however—can be reversed by aPC. …”
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    ER-Stress and Senescence Coordinately Promote Endothelial Barrier Dysfunction in Diabetes-Induced Atherosclerosis
  16. 2656
    “…Correlation analyses revealed that the CDK inhibitor p21 and irradiation induced senescence were differentially regulated between male and female cells. …”
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    Gonadal sex patterns p21-induced cellular senescence in mouse and human glioblastoma
  17. 2657
    “…Further, NitC exhibited its anti-oxidative potential through ameliorating cellular senescence in IL-1β-treated RACs and decreasing NLRP3 inflammasomes activation in LPS-stimulated RAW 264.7 via scavenging ROS. …”
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    Nitidine Chloride Alleviates Inflammation and Cellular Senescence in Murine Osteoarthritis Through Scavenging ROS
  18. 2658
    “…In addition, the molecular mechanisms by which preosteoclasts become senescent as well as how the senescent preosteoclasts impair subchondral bone microenvironment were characterized using in vitro preosteoclast culture system. …”
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    Senescent preosteoclast secretome promotes metabolic syndrome associated osteoarthritis through cyclooxygenase 2
  19. 2659
    “…Our results highlight the m(6)A-independent function of Mettl14 in reprogramming and provide new insight into the interplay between senescence and reprogramming in vitro.…”
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    Mettl14-driven senescence-associated secretory phenotype facilitates somatic cell reprogramming
  20. 2660
    “…Because age-related cellular senescence and type 2 diabetes (T2D) have been recognised as risk factors for CRC development, the recent finding that type 2 diabetic patients present an elevated circulating volume of senescent cells raises the question whether type 2 diabetes facilitates the process of CRC tumorigenesis by inducing premature cell senescence. …”
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    Pro-tumorigenic role of type 2 diabetes-induced cellular senescence in colorectal cancer
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