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  1. 4261
    “…Leaf senescence is an important process of growth and development in plant, and it is a programmed decline controlled by a series of genes. …”
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    Integrative Analyses of Biochemical Properties and Transcriptome Reveal the Dynamic Changes in Leaf Senescence of Tobacco (Nicotiana tabacum L.)
  2. 4262
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    Correction to: Genome-wide and comparative phylogenetic analysis of senescence-associated NAC transcription factors in sunfower (Helianthus annuus)
  3. 4263
    “…The increase in senescent cells in tissues, including the brain, is a general feature of normal aging and age‐related pathologies. …”
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    Loss of lamin‐B1 and defective nuclear morphology are hallmarks of astrocyte senescence in vitro and in the aging human hippocampus
  4. 4264
    “…Indeed, aging, by increasing the senescence burden, is configured as a negative prognostic factor for GBM stage. …”
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    The Longevity-Associated Variant of BPIFB4 Reduces Senescence in Glioma Cells and in Patients’ Lymphocytes Favoring Chemotherapy Efficacy
  5. 4265
    “…Accumulation of senescent bone marrow-derived mesenchymal stem cells (BMMSCs) has led to an age-related bone loss. …”
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    Melatonin Improves the Resistance of Oxidative Stress-Induced Cellular Senescence in Osteoporotic Bone Marrow Mesenchymal Stem Cells
  6. 4266
    “…We show that combined BAP1 deficiency and SF3B1 hotspot mutation lead to senescence and growth arrest in human UM cells. Although p53 protein expression is induced, deletion of TP53 (encoding p53) only modestly rescues the observed senescent phenotype. …”
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    Co‐occurrence of BAP1 and SF3B1 mutations in uveal melanoma induces cellular senescence
  7. 4267
    “…SIMPLE SUMMARY: Cellular senescence may contribute to aging and age-related diseases, and the elimination of senescent cells is considered a promising anti-aging strategy. …”
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    Senolysis-Based Elimination of Chemotherapy-Induced Senescent Breast Cancer Cells by Quercetin Derivative with Blocked Hydroxy Groups
  8. 4268
    “…A plethora of factors have been attributed to underly aging, including oxidative stress, telomere shortening and cellular senescence. Several studies have shown a significant role of the cyclin-dependent kinase inhibitor p16ink4a in senescence and aging. …”
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    Dynamic Spatiotemporal Expression Pattern of the Senescence-Associated Factor p16Ink4a in Development and Aging
  9. 4269
    “…However, its effect on cellular senescence has not been elucidated. Objective: To investigate the mechanism for low concentrations of ICT exerting antitumor activity through induction of cellular senescence. …”
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    Induction of ROS and DNA damage-dependent senescence by icaritin contributes to its antitumor activity in hepatocellular carcinoma cells
  10. 4270
    “…The current study aims to investigate the role of melatonin on senescent bone marrow stromal cells (BMSCs) and the underlying regulating mechanism. …”
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    Melatonin enhances osteoblastogenesis of senescent bone marrow stromal cells through NSD2‐mediated chromatin remodelling
  11. 4271
    “…Cellular senescence is a state of stable growth arrest and a desired outcome of tumor suppressive interventions. …”
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    Pharmacological CDK4/6 inhibition reveals a p53‐dependent senescent state with restricted toxicity
  12. 4272
    “…However, these senescent tumor cells cannot be completely cleared and can induce tumor recurrence. …”
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    Mechanisms of Senescence-Related NKG2D Ligands Release and Immune Escape Induced by Chemotherapy in Neuroblastoma Cells
  13. 4273
    “…Radiation therapy can induce cellular senescence in cancer cells, leading to short‐term tumor growth arrest but increased long‐term recurrence. …”
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    A new function for the serine protease HtrA2 in controlling radiation‐induced senescence in cancer cells
  14. 4274
    “…Pro-inflammatory cytokines (TNF-α, IL-1β, and IL-6) were measured via ELISA. Senescent cells were estimated via SA-β-Gal staining as well as Western blot (senescent markers p21 and p27). …”
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    Blockage of ERCC6 Alleviates Spinal Cord Injury Through Weakening Apoptosis, Inflammation, Senescence, and Oxidative Stress
  15. 4275
    “…NK cell treatment also significantly decreased levels of senescence markers and senescence-associated secretory phenotypes (SASPs) in three senescent adipose tissues when culturing them together. …”
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    Combining adoptive NK cell infusion with a dopamine-releasing peptide reduces senescent cells in aged mice
  16. 4276
    “…Pathologic diabetic wound healing is caused by sequential and progressive deterioration of hemostasis, inflammation, proliferation, and resolution/remodeling. Cellular senescence promotes wound healing; however, diabetic wounds exhibit low levels of senescent factors and accumulate senescent cells, which impair the healing process. …”
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    Altered regulation of mesenchymal cell senescence in adipose tissue promotes pathological changes associated with diabetic wound healing
  17. 4277
    “…Our findings uncover an essential role of Piezo1-mediated mechano-signaling in MuSCs for maintaining quiescence and preventing senescence. Reduced mechano-signaling due to decreased physical activity during aging may contribute to the increase of senescent cells and the decline of MuSC numbers in geriatric mice and humans.…”
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    Mechano-signaling via Piezo1 prevents activation and p53-mediated senescence of muscle stem cells
  18. 4278
    “…Together, the findings expand our understanding of innate immune senescence and reveal the potential of the senolytics as a promising treatment to alleviate the symptom of viral pneumonia, particularly for the older population.…”
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    Dysfunctional telomeres through mitostress‐induced cGAS/STING activation to aggravate immune senescence and viral pneumonia
  19. 4279
    “…Senescent cells (SCs) produce the senescence-associated secretory phenotype to affect the function of neighboring cells and SCs themselves. …”
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    Age-associated callus senescent cells produce TGF-β1 that inhibits fracture healing in aged mice
  20. 4280
    “…Interstitial fibrosis and tubular atrophy, a major cause of kidney allograft dysfunction, has been linked to premature cellular senescence. The mTOR inhibitor Rapamycin protects from senescence in experimental models, but its antiproliferative properties have raised concern early after transplantation particularly at higher doses. …”
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    The mTOR inhibitor Rapamycin protects from premature cellular senescence early after experimental kidney transplantation
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