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  1. 4421
    “…Whether depletion of cellular GSH, associated with neuronal senescence, directly influences Ca(2+ )permeation pathways is not known. …”
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    Loss of glutathione homeostasis associated with neuronal senescence facilitates TRPM2 channel activation in cultured hippocampal pyramidal neurons
  2. 4422
    “…The p53 tumor-suppressor protein has a key role in the induction of cellular senescence, an important barrier to cancer development. …”
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    The CEACAM1 tumor suppressor is an ATM and p53-regulated gene required for the induction of cellular senescence by DNA damage
  3. 4423
    “…METHODS: We examined in different human NSCLC cell lines and detected cellular senescence after cisplatin (DDP) treatment in the presence or absence of DEX. …”
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    Dexamethasone Reduces Sensitivity to Cisplatin by Blunting p53-Dependent Cellular Senescence in Non-Small Cell Lung Cancer
  4. 4424
    “…This makes senescent cells a potential cause of tissue functional decline in aging. …”
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    Postmitotic neurons develop a p21-dependent senescence-like phenotype driven by a DNA damage response
  5. 4425
    “…These results suggest that treatment with a lower concentration of H(2)O(2) in MGFs induces cell cycle arrest, resulting in stress-induced premature senescence, possibly correlated with the development of periodontal diseases.…”
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    Oxidative stress caused by a low concentration of hydrogen peroxide induces senescence-like changes in mouse gingival fibroblasts
  6. 4426
    “…BACKGROUND: Cellular senescence may be a key factor in HIV-related premature biological aging. …”
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    Corneal Endothelial Cells Provide Evidence of Accelerated Cellular Senescence Associated with HIV Infection: A Case-Control Study
  7. 4427
    “…It is believed that the data also link determinate nodulation, plant reproduction, and senescence to CCD7 function for the first time.…”
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    CAROTENOID CLEAVAGE DIOXYGENASE 7 modulates plant growth, reproduction, senescence, and determinate nodulation in the model legume Lotus japonicus
  8. 4428
    “…Senescence is a coordinated process where a plant, or a part of it, engages in programmed cell death to salvage nutrients by remobilizing them to younger tissues or to developing seeds. …”
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    kNACking on heaven’s door: how important are NAC transcription factors for leaf senescence and Fe/Zn remobilization to seeds?
  9. 4429
    “…The knockdown of CPEB1 reduced cell senescence by regulating the expression or distribution of p53 in glioma cells. …”
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    CPEB1, a histone-modified hypomethylated gene, is regulated by miR-101 and involved in cell senescence in glioma
  10. 4430
    “…Aged kidneys 6 weeks post-reperfusion also express higher levels of p53 and p21 compared to the young, correlating with greater increases in senescence associated (SA) β-galactosidase, a known marker of cellular senescence. …”
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    Increased Cellular Senescence and Vascular Rarefaction Exacerbate the Progression of Kidney Fibrosis in Aged Mice Following Transient Ischemic Injury
  11. 4431
    “…Senescence is a stress-responsive form of stable cell cycle exit. …”
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    Redistribution of the Lamin B1 genomic binding profile affects rearrangement of heterochromatic domains and SAHF formation during senescence
  12. 4432
    “…Senescence can compromise tissue repair and regeneration, contributing to tissue and organismal aging via the accumulation of senescent cells, depletion of stem/progenitor cells and secretion of an array of inflammatory cytokines, chemokines, and matrix metalloproteinases. …”
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    Friend or foe: emerging role of nuclear factor kappa-light-chain-enhancer of activated B cells in cell senescence
  13. 4433
    “…Senescent cells are metabolically active and produce a variety of proinflammatory cytokines. …”
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    Oncogene- and Oxidative Stress-Induced Cellular Senescence Shows Distinct Expression Patterns of Proinflammatory Cytokines in Vascular Endothelial Cells
  14. 4434
    “…Harboring the behavioral and histopathological signatures of Alzheimer's disease (AD), senescence accelerated mouse-prone 8 (SAMP8) mice are currently considered a robust model for studying AD. …”
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    Nodes and biological processes identified on the basis of network analysis in the brain of the senescence accelerated mice as an Alzheimer's disease animal model
  15. 4435
    “…METHODS AND RESULTS: Young (5‐month‐old) and senescent (21‐month‐old) FVB wild‐type (WT) or transgenic mice with myocyte‐specific overexpression of catalase were studied. …”
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    Hydrogen Peroxide–Mediated SERCA Cysteine 674 Oxidation Contributes to Impaired Cardiac Myocyte Relaxation in Senescent Mouse Heart
  16. 4436
    “…Proteomic analysis revealed that proteins related to antioxidant activities such as senescence marker protein-30 (SMP30), selenium-binding proteins (SP56) and glutathione S-transferases (GSTs) were up-regulated in Smad3(−/−) mice. …”
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    Smad3 Deficiency Ameliorates Hepatic Fibrogenesis through the Expression of Senescence Marker Protein-30, an Antioxidant-Related Protein
  17. 4437
    “…We recently observed that in senescent animals subject to cyclically applied loading, supplementation with Cyclosporin A (CsA) substantially enhanced the periosteal bone formation rates to levels observed in young animals. …”
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    Distinct Cyclosporin A Doses Are Required to Enhance Bone Formation Induced by Cyclic and Rest-Inserted Loading in the Senescent Skeleton
  18. 4438
    “…Overall, upregulation of cellular senescence gene SENEX, was associated to regulatory T cells accumulation in aged urinary bladder cancer. …”
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    A Novel Cellular Senescence Gene, SENEX, Is Involved in Peripheral Regulatory T Cells Accumulation in Aged Urinary Bladder Cancer
  19. 4439
    “…The importance of p16 in melanoma suggests indolence of the other major senescence pathway through p53. Little or no p53 is expressed in senescent normal human melanocytes, but p16-deficient melanocytes can undergo p53-mediated senescence. …”
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    Senescence evasion in melanoma progression: uncoupling of DNA-damage signaling from p53 activation and p21 expression
  20. 4440
    “…Previous studies have shown that TGF-β induces senescence in several cell types. However, whether TGF-β affects senescence of bmMSCs has not been elucidated. …”
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    TGF-β1 induces senescence of bone marrow mesenchymal stem cells via increase of mitochondrial ROS production
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