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  1. 4641
    “…Expression of senescence-associated β-galactosidase is induced in a subpopulation of OAC cells following FLOT and CROSS chemotherapy treatment, along with enhanced expression of TIM-3 and A2aR ICs. …”
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    Chemotherapy regimens induce inhibitory immune checkpoint protein expression on stem-like and senescent-like oesophageal adenocarcinoma cells
  2. 4642
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    Targeting cellular senescence prevents glucocorticoid-induced bone loss through modulation of the DPP4-GLP-1 axis
  3. 4643
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    Acupoint catgut embedding improves senescence in a rat model of ageing by regulating mitophagy via the PINK1 pathway
  4. 4644
    “…Autophagy plays a critical role in stem cell maintenance and is related to cell growth and cellular senescence. It is important to find a quality-control marker for predicting senescent cells. …”
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    Positively Correlated CD47 Activation and Autophagy in Umbilical Cord Blood-Derived Mesenchymal Stem Cells during Senescence
  5. 4645
    “…Senescence of bone marrow-derived mesenchymal stem cells (BMSCs) has been widely reported to be closely correlated with aging-related diseases, including osteoporosis (OP). …”
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    LRRc17 controls BMSC senescence via mitophagy and inhibits the therapeutic effect of BMSCs on ovariectomy-induced bone loss
  6. 4646
    “…Downregulation of the classical NCT pathway in senescent cells has been reported. However, whether this is a hallmark that exists across all types of cellular senescence remains unknown, and whether the mRNA export machinery is altered during senescence has not been demonstrated. …”
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    Global transcriptional downregulation of TREX and nuclear trafficking machinery as pan-senescence phenomena: evidence from human cells and tissues
  7. 4647
    “…SIMPLE SUMMARY: Chemotherapy can lead to cellular senescence in tumor cells. Here we demonstrate that oxaliplatin induces senescence in p53-proficient colorectal cancer (CRC) cells and leads to the G2-phase arrest in all lines studied (HCT116(p53+/+,) HCT116(p53−/−), LoVo, SW48, and SW480). …”
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    Oxaliplatin-Induced Senescence in Colorectal Cancer Cells Depends on p14(ARF)-Mediated Sustained p53 Activation
  8. 4648
    “…Long-term IL-32θ accumulation triggered permanent senescence and chromosomal instability in MDA-MB-231 cells. …”
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    Interleukin-32θ Triggers Cellular Senescence and Reduces Sensitivity to Doxorubicin-Mediated Cytotoxicity in MDA-MB-231 Cells
  9. 4649
    “…Cellular senescence and lung aging are associated with the pathogenesis of chronic obstructive pulmonary disease (COPD). …”
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    p16-3MR: A Novel Model to Study Cellular Senescence in Cigarette Smoke-Induced Lung Injuries
  10. 4650
    “…We found that dia-hiPSC-ECs had disrupted glycine homeostasis, increased senescence, and impaired mitochondrial function and angiogenic potential as compared with healthy hiPSC-ECs. …”
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    Diabetic Endothelial Cells Differentiated From Patient iPSCs Show Dysregulated Glycine Homeostasis and Senescence Associated Phenotypes
  11. 4651
    “…Here, we identified the anti-senescence effect of Arid1a deficiency in the progression of pancreatic intraepithelial neoplasia (PanIN) by profiling the transcriptome of individual PanINs in a mouse model. …”
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    Single-PanIN-seq unveils that ARID1A deficiency promotes pancreatic tumorigenesis by attenuating KRAS-induced senescence
  12. 4652
    “…During leaf senescence, the final stage of leaf development, nutrients are recycled from leaves to other organs, and therefore proper control of senescence is thus critical for plant fitness. …”
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    An alternative splicing variant of PtRD26 delays leaf senescence by regulating multiple NAC transcription factors in Populus
  13. 4653
    “…In this study, we investigated the protective effect of luteolin against oxidative stress-induced cellular senescence and its molecular mechanism using hydrogen peroxide (H(2)O(2))-induced cellular senescence model in House Ear Institute-Organ of Corti 1 cells (HEI-OC1). …”
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    Luteolin inhibits H(2)O(2)-induced cellular senescence via modulation of SIRT1 and p53
  14. 4654
    “…Differential Survivin localization also resulted in distinctive TMZ-triggered transcriptional pathways, associated with senescence and chromosome instability as shown by global transcriptome analysis. …”
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    Localization matters: nuclear-trapped Survivin sensitizes glioblastoma cells to temozolomide by elevating cellular senescence and impairing homologous recombination
  15. 4655
    “…This alleviated the cellular senescence caused by UVA irradiation. These results indicated that SPCA1 might exert a protective effect on UVA-induced senescence in HDFs via forming a negative feedback loop. …”
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    A Negative Feedback Loop in Ultraviolet A-Induced Senescence in Human Dermal Fibroblasts Formed by SPCA1 and MAPK
  16. 4656
    “…The function of CCCH in regulating leaf senescence, especially its roles in abscisic acid (ABA)-mediated leaf senescence is largely unknown. …”
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    CCCH protein-PvCCCH69 acted as a repressor for leaf senescence through suppressing ABA-signaling pathway
  17. 4657
    “…Cellular senescence may be a side effect of chemotherapy and other anti-cancer treatments that may promote inflammation and paracrine secondary senescence in healthy tissues. …”
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    The lack of functional DNMT2/TRDMT1 gene modulates cancer cell responses during drug-induced senescence
  18. 4658
    “…Alteration of RNA splicing is a hallmark of cellular senescence, which is associated with age-related disease and cancer development. …”
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    PRPF19 regulates p53-dependent cellular senescence by modulating alternative splicing of MDM4 mRNA
  19. 4659
    “…Immunohistochemistry for senescence markers, that is, high mobility group box-1 (HMGB1), p16, p15, and decoy receptor 2 (DCR2), was performed in tissue microarrays of 1140 IBC samples. …”
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    Immunohistochemical Expressions of Senescence-Associated Secretory Phenotype and Its Association With Immune Microenvironments and Clinicopathological Factors in Invasive Breast Cancer
  20. 4660
    “…Gene set enrichment analysis revealed a senescence-like signature with strong induction of p53 and p21 but in the absence of canonical hallmarks of senescence such as β-galactosidase positivity, suggesting incomplete cell fate commitment. …”
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    MYCN-induced nucleolar stress drives an early senescence-like transcriptional program in hTERT-immortalized RPE cells
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