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  1. 941
    “…A hallmark trait of cellular senescence is the acquisition of a senescence-associated secretory phenotype (SASP). …”
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    NF90 coordinately represses the senescence-associated secretory phenotype
  2. 942
    “…The induction of senescence in tumor cells impairs transformation and promotes an anticancer immune response resulting from the production by senescent cells of cytokines and chemokines, an aspect known as “senescence-associated secretory phenotype” (SASP). …”
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    Senescence and NFκB: A trojan horse in tumors?
  3. 943
    “…Although ionizing radiation is known to induce cellular senescence in vitro and in vivo, its long-term in vivo effects are not well defined. …”
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    Prolonged expression of senescence markers in mice exposed to gamma-irradiation
  4. 944
    “…BACKGROUND: Senescent endothelial cells acquire functional properties that make the vascular wall more prone to atherosclerotic changes. …”
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    Sulodexide reduces senescence-related changes in human endothelial cells
  5. 945
    “…When the cell cycle is arrested, even though growth-promoting pathways such as mTOR are still active, then cells senesce. For example, induction of either p21 or p16 arrests the cell cycle without inhibiting mTOR, which, in turn, converts p21/p16-induced arrest into senescence (geroconversion). …”
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    Hyper-mitogenic drive coexists with mitotic incompetence in senescent cells
  6. 946
    “…In the roots of 71-day-old senescent plants, nitroproteome analysis enabled the identification a total of 16 nitrotyrosine-immunopositive proteins. …”
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    Protein tyrosine nitration in pea roots during development and senescence
  7. 947
    “…In cold extracts of senescent leaves of the Lime tree (Tilia cordata), two colorless nonfluorescent chlorophyll catabolites (NCCs) were identified, named Tc-NCC-1 and Tc-NCC-2, as well as a polar yellow chlorophyll catabolite (YCC), named Tc-YCC. …”
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    Chlorophyll Catabolites in Senescent Leaves of the Lime Tree (Tilia cordata)
  8. 948
    “…CONCLUSIONS: Our data provide a strong quantitative example of how extrinsic mortality can shape evolution of senescence in a vertebrate clade. Nothobranchius is emerging as a genomic model species. …”
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    Parallel evolution of senescence in annual fishes in response to extrinsic mortality
  9. 949
    por Pantziarka, Pan
    Publicado 2013
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    Li Fraumeni syndrome, cancer and senescence: a new hypothesis
  10. 950
    “…1. Senescence, the physiological decline that results in decreasing survival and/or reproduction with age, remains one of the most perplexing topics in biology. …”
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    Plants do not count… or do they? New perspectives on the universality of senescence
  11. 951
    “…Yet the heterogeneity of the senescence phenotype has hindered the understanding of its onset. …”
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    The Length of the Shortest Telomere as the Major Determinant of the Onset of Replicative Senescence
  12. 952
    por Darzynkiewicz, Zbigniew
    Publicado 2013
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    Perturbation of nucleotide metabolism - the driving force of oncogene-induced senescence
  13. 953
    “…Aging and age-related diseases are associated with cellular senescence that results in variable apoptosis susceptibility to oxidative stress. …”
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    Histone Modifications in Senescence-Associated Resistance to Apoptosis by Oxidative Stress()()
  14. 954
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    Rapamycin suppresses brain aging in senescence-accelerated OXYS rats
  15. 955
    “…CONCLUSIONS: The findings support the hypothesis that compounds in wsCSE induces amnion cell senescence via a mechanism involving ROS and DNA damage. …”
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    Senescence of Primary Amniotic Cells via Oxidative DNA Damage
  16. 956
    “…The purpose of the present work was to verify whether TO-RGN may also be effective in counteracting the expression of the senescence marker beta-galactosidase and of senescence-associated gene expression patterning, engaged during prolonged culture of human adipose-derived stem cells (hADSCs). …”
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    Anti-senescence efficacy of radio-electric asymmetric conveyer technology
  17. 957
    “…In this review we describe our current understanding of cellular senescence, examine in detail the intricate pathways linking the DDR, ROS and SASP, and evaluate their impact on the stability of the senescent phenotype.…”
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    Telomeres, oxidative stress and inflammatory factors: partners in cellular senescence?
  18. 958
    “…A growing body of evidence demonstrates that the accumulation of senescent cells is a plausible ageing mechanism. It has been proposed that the senescence of vascular cells plays a causal role in the development of cardiovascular pathologies. …”
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    A relationship exists between replicative senescence and cardiovascular health
  19. 959
    “…Oncogenic stress-induced senescence (OIS) prevents the ability of oncogenic signals to induce tumorigenesis. …”
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    Glucose metabolism and hexosamine pathway regulate oncogene-induced senescence
  20. 960
    “…Our results further revealed that repression of Shc expression by let-7a delays senescence of human diploid fibroblasts (HDFs). In sum, our findings link let-7a abundance to the expression of p66Shc, which in turn controls the replicative lifespan of HDFs.…”
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    let-7-repressesed Shc translation delays replicative senescence
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