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  1. 1301
    “…BACKGROUND: Senescent cells occur in adults with cirrhotic livers independent of the etiology. …”
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    Cellular Senescence in Livers from Children with End Stage Liver Disease
  2. 1302
    “…Hydrogen has been reported to have neuron protective effects due to its antioxidant properties, but the effects of hydrogen on cognitive impairment due to senescence-related brain alterations and the underlying mechanisms have not been characterized. …”
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    Drinking Hydrogen Water Ameliorated Cognitive Impairment in Senescence-Accelerated Mice
  3. 1303
    “…We show that thymic epithelial senescence correlates with significant destruction of epithelial network followed by adipose involution. …”
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    Wnt4 and LAP2alpha as Pacemakers of Thymic Epithelial Senescence
  4. 1304
    por Hydbring, Per, Larsson, Lars-Gunnar
    Publicado 2010
    “…Further, utilizing Cdk2 as a cofactor, Myc directly controlled key genes involved in senescence. We speculate that this new role of Myc/Cdk2 in senescence has relevance for other Myc functions, such as regulation of stemness, self-renewal, immortalization and differentiation, which may have an impact on tissue regeneration. …”
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    Cdk2: a key regulator of the senescence control function of Myc
  5. 1305
    “…Apoptosis was quantified by flow-cytometric analysis of annexin V-binding and senescence was analyzed on the basis of cellular β-galactosidase activity. …”
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    Senescence evasion by MCF-7 human breast tumor-initiating cells
  6. 1306
    “…Expression of RAV1 and its homologues is closely associated with leaf maturation and senescence. RAV1 mRNA increased at a later stage of leaf maturation and reached a maximal level early in senescence, but decreased again during late senescence. …”
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    The RAV1 transcription factor positively regulates leaf senescence in Arabidopsis
  7. 1307
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    The p53 inducing drug dosage may determine quiescence or senescence
  8. 1308
    “…Cellular senescence acts as a barrier to cancer progression, and microRNAs (miRNAs) are thought to be potential senescence regulators. …”
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    miR-22 represses cancer progression by inducing cellular senescence
  9. 1309
    “…Cellular senescence is a program of irreversible cell cycle arrest that normal cells undergo in response to progressive shortening of telomeres, changes in telomeric structure, oncogene activation or oxidative stress. …”
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    Activation of Nuclear Factor-kappa B signalling promotes cellular senescence
  10. 1310
    por Coutu, Daniel L., Galipeau, Jacques
    Publicado 2011
    “…FGFs appear to promote self-renewing proliferation and inhibit cellular senescence in nearly all tissues tested to date. Here we review the role of FGFs and FGFRs in stem cell self-renewal, cellular senescence, and aging.…”
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    Roles of FGF signaling in stem cell self-renewal, senescence and aging
  11. 1311
    “…Tumors are defined by their intense proliferation, but sometimes cancer cells turn senescent and stop replicating. In the stochastic cancer model in which all cells are tumorigenic, senescence is seen as the result of random mutations, suggesting that it could represent a barrier to tumor growth. …”
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    Senescent Cells in Growing Tumors: Population Dynamics and Cancer Stem Cells
  12. 1312
    “…Changes in the shape of the nuclear lamina are exhibited in senescent cells, as well as in cells expressing mutations in lamina genes. …”
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    Robust nuclear lamina-based cell classification of aging and senescent cells
  13. 1313
    por Fabrizio, Paola, Wei, Min
    Publicado 2011
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    Conserved role of medium acidification in chronological senescence of yeast and mammalian cells
  14. 1314
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    Establishment and evaluation of transgenic models of Alzheimer's disease with senescence accelerated background
  15. 1315
    “…Conversely, persistent treatment of wild type cells with low levels of TGFβ slows proliferation and induces senescence, suggesting that TGFβ signaling also contributes to cellular senescence. …”
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    Premature Senescence and Increased TGFβ Signaling in the Absence of Tgif1
  16. 1316
    “…Some of the genes upregulated following CMV-infection are also upregulated during cellular senescence, indicating that CMV causes an immunological phenotype in fibroblasts, which is partially reminiscent of replicative senescent cells. …”
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    The immunoregulatory effects of CMV-infection in human fibroblasts and the impact on cellular senescence
  17. 1317
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    Cancers co-opt cohabitants’ catabolism: Autophagy and senescence in the tumor stroma
  18. 1318
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    Association between functional alterations of senescence and senility and disorders of gait and balance
  19. 1319
    “…Most human somatic cells do not divide indefinitely but enter a terminal growth arrest termed replicative senescence. Replicatively senescent cells are generally believed to arrest in G1 or G0 stage of the cell cycle. …”
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    Replicatively senescent cells are arrested in G1 and G2 phases
  20. 1320
    “…Here, we present an analytic tool to quantify the number of cells in a population that present characteristics of senescence, apoptosis or nuclear irregularities through nuclear morphometric analysis. …”
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    Nuclear Morphometric Analysis (NMA): Screening of Senescence, Apoptosis and Nuclear Irregularities
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