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  1. 1481
    “…Evidence has shown that with age increase, bone marrow stromal cells (BMSCs) differentiate into more adipocytes rather than osteoblasts and undergo senescence, which leads to decreased bone formation and contributes to senile osteoporosis. …”
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    Senile Osteoporosis: The Involvement of Differentiation and Senescence of Bone Marrow Stromal Cells
  2. 1482
    “…However, recent studies have also suggested a dark side of senescence in which a build-up of senescent cells with age leads to increased inflammation due to a senescence-associated secretory phenotype (SASP). …”
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    Senolytics: A Translational Bridge Between Cellular Senescence and Organismal Aging
  3. 1483
    “…The three-band plant senescence reflectance index (PSRI) approximated the visually observed senescence dynamics best, whereas full-spectrum models suffered from a strong year-specificity. …”
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    Spectral Vegetation Indices to Track Senescence Dynamics in Diverse Wheat Germplasm
  4. 1484
    “…The investigation of senescence-associated immune cell subsets from the spleens and mesenteric lymph nodes revealed an increase in PD-1(+)CD44(high) CD4 T cells as well as CD95(+)GL7(+) germinal center B cells, indicating that the long-term circadian misalignment exacerbates immune senescence and consequent chronic inflammation. …”
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    Chronic circadian misalignment accelerates immune senescence and abbreviates lifespan in mice
  5. 1485
    “…Accumulation of senescent cells in tissues has been implicated in aging and a plethora of age-related diseases. …”
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    Implications of Oxidative Stress and Cellular Senescence in Age-Related Thymus Involution
  6. 1486
    “…Cellular senescence is a physiological process reacting to stimuli, in which cells enter a state of irreversible growth arrest in response to adverse consequences associated with metabolic disorders. …”
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    NMR-based metabonomic analysis of HUVEC cells during replicative senescence
  7. 1487
    por Bell, Graham
    Publicado 1993
    “…This paper discusses a novel theory of senescence: the community of pathogens within each host individual evolves during the life-time of the host, and in doing so progressively reduces host vigour. …”
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    Pathogen evolution within host individuals as a primary cause of senescence
  8. 1488
    por J. Heath, John, D. Grant, Michael
    Publicado 2020
    “…This review addresses the potential contribution of cytomegalovirus (CMV)-driven T cell proliferation to cellular senescence and immunosenescence. We first describe molecular processes imposing cell cycle arrest, the foundation of cellular senescence, then focus on the unique distribution, phenotype and function of CMV-specific CD8(+) T cells in the context of cellular senescence and “inflammaging”. …”
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    The Immune Response Against Human Cytomegalovirus Links Cellular to Systemic Senescence
  9. 1489
    “…In this study, we sought to characterize the EV production of senescent endothelial cells. To achieve this, Human Umbilical Vascular Endothelial Cells (HUVECs) were replicated until they reached senescence, as determined by measurement of Senescence‐Associated β‐Galactosidase activity via microscopy and flow cytometry. …”
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    Increased production of functional small extracellular vesicles in senescent endothelial cells
  10. 1490
    “…Premature senescence of neonatal cardiac myocytes was induced by prolonged culture time in an oxygen-rich postnatal environment. …”
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    Cardiomyocyte Contractility and Autophagy in a Premature Senescence Model of Cardiac Aging
  11. 1491
    “…Introduction Aging is associated with the pathogenesis of many endocrine disorders such as cardiovascular diseases and diabetes. Cell senescence has been reported as one of their mechanisms. …”
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    OR03-04 The Study of Cell Senescence in Cortisol-Producing Adrenocortical Adenomas
  12. 1492
    “…To understand the role of the extensive senescence-associated 3D genome reorganization, we generated genome-wide chromatin interaction maps, epigenome, replication-timing, whole-genome bisulfite sequencing, and gene expression profiles from cells entering replicative senescence (RS) or upon oncogene-induced senescence (OIS). …”
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    4D Genome Rewiring during Oncogene-Induced and Replicative Senescence
  13. 1493
    “…Co-staining of the senescence marker p21, the osteoblastic marker osteopontin, detection of senescence-associated heterochromatin foci, and senescence-associated β-galactosidase is possible within one test approach requiring fewer cells. …”
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    A Novel Protocol for Detection of Senescence and Calcification Markers by Fluorescence Microscopy
  14. 1494
    “…RESULTS: We develop CellAge (http://genomics.senescence.info/cells), a manually curated database of 279 human genes driving cellular senescence, and perform various integrative analyses. …”
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    A multidimensional systems biology analysis of cellular senescence in aging and disease
  15. 1495
    “…Telomere shortening in human cells leads to a DNA damage response that signals replicative senescence. While insufficient loading of TRF2 at shortened telomeres contributes to the DNA damage response in senescence, the contribution of TRF1 to senescence induction has not been determined. …”
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    Persistent telomere cohesion protects aged cells from premature senescence
  16. 1496
    “…Senescence is a state of proliferative arrest which has been described as a protective mechanism against the malignant transformation of cells. …”
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    The bright and dark side of extracellular vesicles in the senescence-associated secretory phenotype
  17. 1497
    “…In addition, senescent cells produce and secrete a complex mixture of molecules, commonly known as the senescence-associated secretory phenotype (SASP), that regulate most of their non-cell-autonomous effects. …”
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    Targeting Cardiac Stem Cell Senescence to Treat Cardiac Aging and Disease
  18. 1498
    “…Cellular senescence was first described as a physiological tumor cell suppressor mechanism that leads to cell growth arrest with production of the senescence-associated secretory phenotype known as SASP. …”
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    Senescence in the Development and Response to Cancer with Immunotherapy: A Double-Edged Sword
  19. 1499
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    In Vitro Toxicological Screening of Stable and Senescing Cultures of Aphanizomenon, Planktothrix, and Raphidiopsis
  20. 1500
    “…We suggest that, as in leaves, molecular mechanisms involved in nitrogen (N) resorption are also activated in senescent roots. An analysis of N concentration indicated that N levels decreased during the senescence of both organs. …”
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    Seasonal senescence of leaves and roots of Populus trichocarpa—is the scenario the same or different?
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