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1701por Afifi, Marwa M., Crncec, Adrijana, Cornwell, James A., Cataisson, Christophe, Paul, Debasish, Ghorab, Laila M., Hernandez, Maria O., Wong, Madeline, Kedei, Noemi, Cappell, Steven D.“…Cells can irreversibly exit the cell cycle and become senescent to safeguard against uncontrolled proliferation. …”
Publicado 2023
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1702“…These studies demonstrate that TEMRA cells play a key role in the antiviral response and immune senescence during HIV infection. Finally, we review current therapeutic strategies targeting TEMRA cells that may be clinically beneficial, highlight their potential role in HIV-1 vaccine development, and provide perspectives and predictions for related future applications.…”
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1703por Jalan-Sakrikar, Nidhi, Anwar, Abid, Yaqoob, Usman, Gan, Can, Lagnado, Anthony B., Wixom, Alexander Q., Jurk, Diana, Huebert, Robert C.“…Cellular senescence and biliary fibrosis are prototypical features of obliterative cholangiopathies, such as primary sclerosing cholangitis (PSC). …”
Publicado 2023
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1704por Carver, Chase M., Gomez, Paul T., Rodriguez, Sonia L., Kachergus, Jennifer M., Liu, Yi, Shi, Ji, Tran, Tommy, Wang, Liguo, Melov, Simon, Thompson, E. Aubrey, Schafer, Marissa J.“…In naturally aged mice, we demonstrate that senescent and disease-associated microglia (DAM) phenotypes converge in hippocampus-adjacent white matter. …”
Publicado 2023
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1705“…Plant senescence is the last stage of plant development and a type of programmed cell death, occurring at a predictable time and cell. …”
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1706por Veronesi, Francesca, Contartese, Deyanira, Di Sarno, Laura, Borsari, Veronica, Fini, Milena, Giavaresi, Gianluca“…Ageing is an irreversible and inevitable biological process and a significant risk factor for the development of various diseases, also affecting the musculoskeletal system, resulting from the accumulation of cell senescence. The aim of this systematic review was to collect the in vitro studies conducted over the past decade in which cell senescence was induced through various methods, with the purpose of evaluating the molecular and cellular mechanisms underlying senescence and to identify treatments capable of delaying senescence. …”
Publicado 2023
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1707“…Furthermore, the mechanisms and implications of extracellular matrix senescence-related genes (ECM-SRGs) in pan-cancer have not been investigated. …”
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1708por Horváth, Emőke, Sólyom, Árpád, Székely, János, Nagy, Előd Ernő, Popoviciu, Horațiu“…The inflammatory microenvironment and synthesis of key matrix components are also controlled by SIRT1 and mTORc. Senescent chondrocytes represent the functional end stage of hypertrophic differentiation and characteristically upregulate p16 and p21, but also a variety of inflammatory cytokines, chemokines and metalloproteinases, developing the senescence-associated secretory phenotype. …”
Publicado 2023
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1709por Riviere-Cazaux, Cecile, Carlstrom, Lucas P., Neth, Bryan J., Olson, Ian E., Rajani, Karishma, Rahman, Masum, Ikram, Samar, Mansour, Moustafa A., Mukherjee, Bipasha, Warrington, Arthur E., Short, Susan C., von Zglinicki, Thomas, Brown, Desmond A., Burma, Sandeep, Tchkonia, Tamar, Schafer, Marissa J., Baker, Darren J., Kizilbash, Sani H., Kirkland, James L., Burns, Terry C.“…Moreover, senescent tumor cells could spur the growth of their non-senescent counterparts, thereby accelerating recurrence. …”
Publicado 2023
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1710por Wang, Shuai, Yang, Jiyong, Xiang, Ruian, Li, Congcong, Li, Junyi, Shen, Xingxing, Liu, Wengang, Xu, Xuemeng“…Background: Cellular senescence is associated with age-related pathological changes, senescent cells promote the development of knee osteoarthritis. …”
Publicado 2023
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1712“…We find that apparent X-shaped structures accumulate at telomeres in senescing tlc1 sgs1 mutants in a RAD52- and RAD53-dependent fashion. …”
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1713Publicado 1982“…An in vitro system to investigate the ability of macrophages to recognize and ingest senescent polymorphonuclear neutrophils has been used that uses chromium-labeled neutrophils and staining for myeloperoxidase (MPO). …”
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1714por Xaymardan, Munira, Zheng, Jingang, Duignan, Inga, Chin, Andrew, Holm, Jacquelyne M., Ballard, Victoria L.T., Edelberg, Jay M.“…Studies in a murine cardiac transplantation model demonstrated that synergist interactions of PDGF-AB plus VEGF plus Ang-2 (PVA) provided an immediate restoration of senescent cardiac vascular function. Moreover, PVA injection in young rat hearts, but not PDGF-AB alone or other cytokine combinations, at the time of coronary occlusion suppressed acute myocardial cell death by >50%. …”
Publicado 2004
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1715“…Senescence is a distinct cellular response induced by DNA-damaging agents and other sublethal stressors and may provide novel benefits in cancer therapy. …”
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1716por Nakamura, Asako J, Chiang, Y Jeffrey, Hathcock, Karen S, Horikawa, Izumi, Sedelnikova, Olga A, Hodes, Richard J, Bonner, William M“…The limits on cell division imposed by senescence may play an important role in both organismal aging and in preventing tumorigenesis. …”
Publicado 2008
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1717por Zhou, Qingjun, Wang, Yao, Yang, Lingling, Wang, Ye, Chen, Peng, Wang, Yiqiang, Dong, Xiaoguang, Xie, Lixin“…In this study, the effects of histone deacetylase (HDAC) inhibitors on the activation, proliferation, migration and senescence of corneal stromal cells were evaluated. …”
Publicado 2008
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1718por Wagner, Wolfgang, Bork, Simone, Horn, Patrick, Krunic, Damir, Walenda, Thomas, Diehlmann, Anke, Benes, Vladimir, Blake, Jonathon, Huber, Franz-Xaver, Eckstein, Volker, Boukamp, Petra, Ho, Anthony D.“…Cells in culture undergo senescence after a certain number of cell divisions whereby the cells enlarge and finally stop proliferation. …”
Publicado 2009
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1719por Rodier, Francis, Coppé, Jean-Philippe, Patil, Christopher K., Hoeijmakers, Wieteke A. M., Muñoz, Denise P., Raza, Saba R., Freund, Adam, Campeau, Eric, Davalos, Albert R., Campisi, Judith“…Cellular senescence suppresses cancer by stably arresting the proliferation of damaged cells1. …”
Publicado 2009
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1720“…NaCl induces DNA breaks, thus leading to cellular senescence. Here we showed that Ku86 deficiency accelerated the high NaCl-induced cellular senescence. …”
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