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  1. 1801
    “…Compared to field conditions, remobilization from leaves was similar (N–S–Cu) or increased by nutrient deficiency (K–P–Mg) while nutrient deficiency had no effect on Mo–Zn–B–Ca–Mn, which seemed to be non-mobile during leaf senescence under field conditions. However, Ca and Mn were largely mobilized from roots (-97 and -86% of their initial root contents, respectively) to shoots. …”
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    Leaf mineral nutrient remobilization during leaf senescence and modulation by nutrient deficiency
  2. 1802
    “…Much like replicative senescence, the irreversible cell-cycle arrest induced by eroded telomeres, accelerated senescence occurs when replicative cells suffer irreparable DNA double-strand breaks (DSBs). …”
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    Ionizing radiation-induced foci persistence screen to discover enhancers of accelerated senescence
  3. 1803
    “…This results in p53/Rb-dependent, premature senescence of human fibroblast, and in the depletion of hematopoietic cells in telomerase-deficient mice. …”
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    Telomerase abrogates aneuploidy-induced telomere replication stress, senescence and cell depletion
  4. 1804
    “…Poor first winter survival in Miscanthus × giganteus has been anecdotally attributed to incomplete first autumn senescence, but these assessments never paired first-year with older M. × giganteus in side-by-side trials to separate the effect of weather from stand age. …”
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    Autumnal leaf senescence in Miscanthus × giganteus and leaf [N] differ by stand age
  5. 1805
    “…Therefore, we sought to investigate to determine if PINK1-PARK2-mediated mitophagy is involved in the regulation of CS extract (CSE)-induced cell senescence and in COPD pathogenesis. Mitochondrial damage, ROS production, and cell senescence were evaluated in primary human bronchial epithelial cells (HBEC). …”
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    PARK2-mediated mitophagy is involved in regulation of HBEC senescence in COPD pathogenesis
  6. 1806
    “…We also identified and quantified senescence and telomerase activity. Finally, we determined the expression of proteins related to replicative senescence (p53-p21-pRB) and stress-induced senescence (p16-pRB) pathways. …”
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    Rat Notochordal Cells Undergo Premature Stress-Induced Senescence by High Glucose
  7. 1807
    “…One of the traits of oncogene-induced senescence (OIS) is the so-called senescence-associated secretory phenotype or senescence secretome. …”
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    Role of ADAM17 in the non-cell autonomous effects of oncogene-induced senescence
  8. 1808
    “…Drought stress triggers mature leaf senescence, which supports plant survival and remobilization of nutrients; yet leaf senescence also critically decreases post-drought crop yield. …”
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    Carbon/Nitrogen Imbalance Associated with Drought-Induced Leaf Senescence in Sorghum bicolor
  9. 1809
    “…Here, we perform methylome analysis of actively dividing and deeply senescent normal human epithelial cells. RESULTS: We identify senescence-associated differentially methylated positions (senDMPs) from multiple experiments using cells from one donor. …”
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    The senescent methylome and its relationship with cancer, ageing and germline genetic variation in humans
  10. 1810
    “…CONCLUSIONS: We found that females at their reproductive senescence are more susceptible to hyperoxia, compared to males. …”
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    Diminished Resistance to Hyperoxia in Brains of Reproductively Senescent Female CBA/H Mice
  11. 1811
    “…Cultivation of rodent LSECs associates with a rapid onset of stress-induced senescence a few days post isolation, which limits genetic and biochemical studies ex vivo. …”
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    Liver Sinusoidal Endothelial Cells Escape Senescence by Loss of p19ARF
  12. 1812
    Publicado 2015
    “…Scott is now a leading scientist in the cancer field and his work has helped to shed light on mechanisms of cell apoptosis and senescence to better understand and treat cancer. In this interview, he talks about this incredible scientific journey.…”
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    Exploring cell apoptosis and senescence to understand and treat cancer: an interview with Scott Lowe
  13. 1813
    por Johnson, Sara A, Cambier, John C
    Publicado 2004
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    Ageing, autoimmunity and arthritis: Senescence of the B cell compartment – implications for humoral immunity
  14. 1814
    “…However, the long-sustained conviction that senescence is a truly irreversible process has recently been challenged. …”
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    In vitro evidence for senescent multinucleated melanocytes as a source for tumor-initiating cells
  15. 1815
    “…Congruently, 5-aza-2′-deoxycytydine, a general inhibitor of DNA methylation, reverses the Kras(G12D)-induced PanIN development by promoting senescence. This requirement of Nupr1 expression, however, is not restricted to the pancreas since in lung of Nupr1(–/–) mice the expression of Kras(G12D) induces senescence instead of transformation. …”
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    Pivotal Role of the Chromatin Protein Nupr1 in Kras-Induced Senescence and Transformation
  16. 1816
    “…This study shows that all immune traits do not senesce at the same rate. In fact, the patterns observed depend upon the immune traits measured and the breeding status of the individual.…”
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    Aging in personal and social immunity: do immune traits senesce at the same rate?
  17. 1817
    “…Furthermore, significant correlations were detected within the group of genes involved in drought stress (r = 0.84) and those acting in leaf senescence (r = 0.64), as well as between leaf senescence genes and the leaf colour (r = 0.34). …”
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    Expression profiling of genes involved in drought stress and leaf senescence in juvenile barley
  18. 1818
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    Open a senescent's heart and look into his brain to find the Takotsubo trigger!
  19. 1819
    “…A novel class of vacuoles, “senescence-associated vacuoles” (SAVs), characterized by intense proteolytic activity appear during senescence in chloroplast-containing cells of leaves. …”
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    Senescence-Associated Vacuoles, a Specific Lytic Compartment for Degradation of Chloroplast Proteins?
  20. 1820
    “…Specific increases of ROS level have been demonstrated as potentially critical for induction and maintenance of cell senescence process. Causal connection between ROS, aging, age-related pathologies, and cell senescence is studied intensely. …”
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    ROS, Cell Senescence, and Novel Molecular Mechanisms in Aging and Age-Related Diseases
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