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  1. 1021
    “…Overall, homB was associated with the presence of cagA (p = 0.043) and vacA s1 (p < 0.001), whereas homA was found more frequently in cagA-negative (p = 0.062) and vacA s2 (p < 0.001) strains. …”
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    Disease association with two Helicobacter pylori duplicate outer membrane protein genes, homB and homA
  2. 1022
    “…H. pylori virulence markers cagA, cagE, and vacA alleles in local and Afghan nationals with H. pylori-associated gastroduodenal diseases were studied. …”
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    Comparison of the Virulence Markers of Helicobacter Pylori and their Associated Diseases in Patients from Pakistan and Afghanistan
  3. 1023
    “…AIM: To evaluate effect of treatment failure on cagA and vacA genotypes in Helicobacter pylori (H. pylori) isolates from Colombia. …”
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    Effect of treatment failure on the CagA EPIYA motif in Helicobacter pylori strains from Colombian subjects
  4. 1024
    “…We report that H. pylori isolates from Nigeria and South Africa differ significantly in their phylogenetic profiles and in their expression of virulence factors. VacA mosaicism is intensive, resulting in m1-m2 vacA chimeras and frequent s1m1 and s1m2 vacA subtypes in hpAfrica2 strains. …”
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    Helicobacter pylori patient isolates from South Africa and Nigeria differ in virulence factor pathogenicity profile and associated gastric disease outcome
  5. 1025
    “…Increased gastric CD55 and IM were both linked with colonisation by vacA i1‐type strains independently of cagA status, and in vitro studies using isogenic mutants of vacA confirmed the ability of VacA to induce CD55 and sCD55 in gastric epithelial cell lines. siRNA experiments to investigate the function of H. pylori‐induced CD55 showed that CD55 knockdown in gastric epithelial cells partially reduced IL‐8 secretion in response to H. pylori, but this was not due to modulation of bacterial adhesion or cytotoxicity. …”
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    The active form of Helicobacter pylori vacuolating cytotoxin induces decay‐accelerating factor CD55 in association with intestinal metaplasia in the human gastric mucosa
  6. 1026
    “…None of the isolates possessed type Ib, 1c, IIIb, IV and V motifs. The most commonly vacA genotypes were s1am1a and s1m2 in isolates with peptic ulcer and gastritis, respectively. …”
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    Investigation of the association between clinical outcome and the cag pathogenicity-island and other virulence genes of Helicobacter pylori isolates from patients with dyspepsia in Eastern Turkey
  7. 1027
    “…We also tested purified VacA cytotoxin. Survival curves were plotted using the Kaplan-Meier method and LD(50) lethal doses were calculated. …”
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    Use of larvae of the wax moth Galleria mellonella as an in vivo model to study the virulence of Helicobacter pylori
  8. 1028
    “…We previously reported that Helicobacter pylori vacA m1 genotype might be responsible for the difference between the two cities, however, the study only included non-cancer patients. …”
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    Advanced non-cardia gastric cancer and Helicobacter pylori infection in Vietnam
  9. 1029
    “…The toxigenic vacA s1m1 genotype was found to be less common and the plasticity region cluster was found to be disrupted in all the isolates. …”
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    Comparative genomics of Helicobacter pylori isolates recovered from ulcer disease patients in England
  10. 1030
    “…Histopathological diagnosis was performed according to the Sydney System. The cagA and vacA genotypes were established using polymerase chain reaction in paraffin blocks. …”
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    Virulence Genes of Helicobacter pylori Increase the Risk of Premalignant Gastric Lesions in a Colombian Population
  11. 1031
    “…The presence of the cagA and vacA genes was investigated by real-time polymerase chain reaction. …”
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    Relationship between Helicobacter pylori virulence genes and gastroduodenal disease
  12. 1032
    “…Our data support the hypothesis that vacA m1 is closely associated with gastric carcinogenesis.…”
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    Analysis of virulence factors of Helicobacter pylori isolated from a Vietnamese population
  13. 1033
    “…As expected, the deduced sequences of two known virulence determinants (CagA and VacA) are highly divergent, with 77% and 87% mean amino acid sequence identities between East Asian and non-Asian groups, respectively. …”
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    Comparative Genomic Analysis of East Asian and Non-Asian Helicobacter pylori Strains Identifies Rapidly Evolving Genes
  14. 1034
    “…Our purpose was to assess the degree of genomic diversity among H. pylori strains isolated from Iranian-children, on the basis of vacA genotype, cagA status of the strains, sex, age as well as the pathological status of the patients. …”
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    Analysis of Genomic Diversity among Helicobacter pylori Strains Isolated from Iranian Children by Pulsed Field Gel Electrophoresis
  15. 1035
    “…These findings may reveal a coordinated relationship between the vacA c2 and cagPAI genotypes.…”
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    Inverse association of Helicobacter pylori cagPAI genotypes with risk of cardia and non‐cardia gastric adenocarcinoma
  16. 1036
    “…Patients were possessed the vacA s1, m1 (6.1%); s1, m2 (6.1%); s2, m1 (15.1%); and s2, m2 (3.0%) genotypes. 94% of patients with gastritis were positive for H. pylori napA gene. …”
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    Analysis of Correlation between the Seven Important Helicobacter pylori (H. pylori) Virulence Factors and Drug Resistance in Patients with Gastritis
  17. 1037
    “…This may be resulted from the reduction of CagA and VacA expressions as well as the type IV secretion system (T4SS) components and secretion system subunit protein A (SecA) protein which are involved in translocation of CagA and VacA into host cells, respectively. …”
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    Evodiamine Inhibits Helicobacter pylori Growth and Helicobacter pylori-Induced Inflammation
  18. 1038
    “…Hespereti n downregulates major virulence factors such as cytotoxin-associated antigen A (CagA) and vacuolating cytotoxin A (VacA) and decreases the translocation of CagA and VacA proteins into gastric adenocarcinoma (AGS) cells. …”
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    Hesperetin Inhibits Expression of Virulence Factors and Growth of Helicobacter pylori
  19. 1039
    “…The isolates were also genotyped for CagA and VacA genes by PCR. RESULTS: the metronidazole resistance rate was 62.70%. …”
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    Helicobacter pylori resistance to metronidazole and its association with virulence factors in a Moroccan population
  20. 1040
    “…The most prevalent genotypes discovered were VacA s1a (75%), m1a (75%), s2 (70%) and m2 (65%), and cagA (60%). …”
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    Helicobacter pylori strains isolated from raw poultry meat: frequency and molecular characteristics
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