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  1. 1241
    “…Helicobacter pylori has been classified as a gastric carcinogen, because it causes gastritis and gastric cancer by injecting the virulent toxin CagA and translocating VacA. The present study investigated the inhibitory action of piperine on H. pylori growth and adhesion. …”
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    Inhibitory effect of piperine on Helicobacter pylori growth and adhesion to gastric adenocarcinoma cells
  2. 1242
    “…Both YN4-84 and YN1-91 harbor intact cag pathogenicity island (cagPAI) and have EPIYA-A/B/D type at the carboxyl terminal of cagA. The vacA gene type is s1m2i1. Another major finding was a 32.5-kb prophage integrated in the YN4-84 genome. …”
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    Comparative Genomics of a Helicobacter pylori Isolate from a Chinese Yunnan Naxi Ethnic Aborigine Suggests High Genetic Divergence and Phage Insertion
  3. 1243
    “…For the in vitro experiments, the bacteria were cultured in BHI broth and RT-qPCR and western blot analysis were carried out to determine the mRNA and protein levels of virulence factors (CagA and VacA) in the cultures. Our results revealed that rhLf not only inhibited the growth of H. pylori, but also suppressed the expression of two major virulence factors. …”
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    Recombinant human lactoferrin enhances the efficacy of triple therapy in mice infected with Helicobacter pylori
  4. 1244
    “…RESULTS: Increased AGO2 was detected in autophagy-deficient ATG5−/− and ATG16−/− mouse embryonic fibroblast cells (MEFs) in comparison with wild-type MEFs. Chemical agents and VacA toxin, which disrupt autophagy, increased AGO2 expression in MEFs, epithelial cells lines, and human monocytes, respectively. …”
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    Microbial Disruption of Autophagy Alters Expression of the RISC Component AGO2, a Critical Regulator of the miRNA Silencing Pathway
  5. 1245
    “…H. pylori EVs are spherical and contain CagA and VacA. They can induce the production of tumor necrosis factor-α, interleukin (IL)-6 and IL-1β by macrophages, and IL-8 by gastric epithelial cells. …”
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    Helicobacter pylori-derived extracellular vesicles increased in the gastric juices of gastric adenocarcinoma patients and induced inflammation mainly via specific targeting of gastric epithelial cells
  6. 1246
    “…The resulting data demonstrate that overexpression of HtrA in H. pylori gives rise to elevated rates of HtrA secretion, cleavage of E-cadherin, bacterial transmigration and delivery of the type IV secretion system (T4SS) effector protein CagA into polarized epithelial cells, but did not affect IL-8 chemokine production or the secretion of vacuolating cytotoxin VacA and γ-glutamyl-transpeptidase GGT. CONCLUSIONS: These data provide for the first time genetic evidence in H. pylori that HtrA is a novel major virulence factor controlling multiple pathogenic activities of this important microbe.…”
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    Overexpression of serine protease HtrA enhances disruption of adherens junctions, paracellular transmigration and type IV secretion of CagA by Helicobacter pylori
  7. 1247
    “…Additionally, the nucleotide diversity of horB and vacA genes from Colombian isolates was lower than in the reference strains and showed a significant genetic differentiation supporting the hypothesis of independent clades with recent evolution. …”
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    Phylogenomics of Colombian Helicobacter pylori isolates
  8. 1248
    “…In the present study, gastric biopsies were used for genotyping cagA and vacA genes, determining the autophagic activity, and the severity of gastric inflammation response. …”
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    Helicobacter pylori CagA Protein Negatively Regulates Autophagy and Promotes Inflammatory Response via c-Met-PI3K/Akt-mTOR Signaling Pathway
  9. 1249
    “…Using two different cell lines, we observed that H. pylori infection decreased Afadin protein levels, independently of CagA, T4SS, and VacA virulence factors. H. pylori infection of cell lines recapitulated several EMT features, displacing and downregulating multiple proteins from cell–cell junctions, and increasing the expression of ZEB1, Vimentin, Slug, N-cadherin, and Snail. …”
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    Afadin Downregulation by Helicobacter pylori Induces Epithelial to Mesenchymal Transition in Gastric Cells
  10. 1250
    “…There was a significant association between vacA genotype and rifaximin resistance (P = 0.042). …”
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    Five alternative Helicobacter pylori antibiotics to counter high levofloxacin and metronidazole resistance in the Dominican Republic
  11. 1251
    “…We chose three loci within the Cytotoxin-associated gene-A Pathogenicity Island (CagPAI) (cagA,cagE,virB11), vacA, homA and homB as markers of virulence with reported association to GC. …”
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    Differential Helicobacter pylori Plasticity in the Gastric Niche of Subjects at Increased Gastric Cancer Risk
  12. 1252
    “…The cagA virulence gene was detected in all 10 patients, and 2 patients had H. pylori strains with different EPIYA motifs. Differences in vacA genotypes were detected in 4 patients. CONCLUSIONS: Co-infection by two H. pylori strains was confirmed by RAPD fingerprinting. …”
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    The analysis of virulence factors and antibiotic resistance between Helicobacter pylori strains isolated from gastric antrum and body
  13. 1253
    “…CTB fused to 11 epitopes predicted of pathogenic (UreB(170–189), VacA(459–478), CagA(1103–1122), GGT(106–126), NapA(30–44), and OipA(211–230)) and colonization (HpaA(33–52), FlaA(487–506), FecA(437–456), BabA(129–149), and SabA(540–559)) proteins from H. pylori. …”
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    Immunoinformatics Approach to Design a Novel Epitope-Based Oral Vaccine Against Helicobacter pylori
  14. 1254
    “…Constrained principal coordinate analyses revealed that the presence of Helicobacter pylori and the cagA and vacA virulence genotypes influenced the structure of the gastric microbiome. …”
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    Gastric Mucosa-Associated Microbial Signatures of Early Gastric Cancer
  15. 1255
    “…An In vitro coculture study demonstrated that H. pylori-specific antigen CagA/VacA-primed dendritic cells (DCs) induced proliferation and IFN-γ, TNF as well as IL-17 production by CD103(+)CD4(+)T cells from patients with H. pylori-positive gastritis, while blocking CD103 with a neutralizing antibody reduced proliferation and IFN-γ, TNF, and IL-17 production by CD103(+)CD4(+)T cells cocultured with DCs. …”
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    CD103 Promotes the Pro-inflammatory Response of Gastric Resident CD4(+) T Cell in Helicobacter pylori-Positive Gastritis
  16. 1256
    “…Helicobacter pylori causes chronic gastritis through cag pathogenicity island (cagPAI), vacuolating cytotoxin A (VacA), lipopolysaccharides (LPS), and flagellin as pathogen-related molecular patterns (PAMPs), which, in combination with the pattern recognition receptors (PRRs) of host cells promotes the expression and secretion of inflammation-causing cytokines and activates innate immune responses such as inflammasomes. …”
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    Interruption of Helicobacter pylori-Induced NLRP3 Inflammasome Activation by Chalcone Derivatives
  17. 1257
    “…No difference was found in iceA or vacA alleles genotypes. Primary resistance to clarithromycin was lower than that reported in Southeast Brazil. …”
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    Clarithromycin-resistant H. pylori primary strains and virulence genotypes in the Northeastern region of Brazil
  18. 1258
    Publicado 2016
    Tabla de Contenidos: “…: México frente a Nicaragua, 1978-1982 / Mónica Toussaint -- Las relaciones méxico-nicaragüenses : entre la cordialidad y el distanciamiento, 1983-1985 / Mireya Tinoco -- Quién me quita lo bailado? : Gerardo Camacho Vaca, agregado cultural mexicano en Nicaragua, 1974-1984 / Fabián Campos Hernández -- Fotografía de prensa y movimientos armados : Pedro Valtierra en Nicaragua / Mónica Morales Flores -- La Dirección Federal de Seguridad y los revolucionarios guatemaltecos, 1947-1985 / Fabián Campos Hernández -- La región de fronteras, de zona de conflicto, selvas y alzamientos a retaguardia estratégica de la URNG : entre la defensa territorial y el apoyo a la guerrilla / Mario Eduardo Valdez Gordillo -- México, un país de refugio? …”
    Libro
    México ante el conflicto centroamericano : testimonio de una época /
  19. 1259
    “…The isolated OMVs were negative for both CagA and VacA virulence factors. Treatment of HSCs with H. pylori-derived OMVs significantly increased the expression of fibrosis markers. …”
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    The Effects of Helicobacter pylori-Derived Outer Membrane Vesicles on Hepatic Stellate Cell Activation and Liver Fibrosis In Vitro
  20. 1260
    “…CONCLUSIONS: Among Zambian patients seen at a single center, antibodies to H. pylori (CagA, VacA, Omp, HcpC, HP0305 and HpaA) were associated with active gastritis.…”
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    Serum antibodies to selected Helicobacter pylori antigens are associated with active gastritis in patients seen at the University Teaching Hospital in Lusaka, Zambia
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