Mostrando 1 - 20 Resultados de 552 Para Buscar '"zar"', tiempo de consulta: 0.25s Limitar resultados
  1. 1
    por Williams, Andrew 1970-
    Publicado 2011
    Libro
  2. 2
    por Kross, Jaan
    Publicado 1992
    Libro
  3. 3
    Publicado 1980
    Libro
  4. 4
    “…In this review, we survey the history of functional studies on ZAR1, with an emphasis on how the ZAR1-RLCK network functions to trap diverse effectors. …”
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    “…Demethylation treatment of various lung cancer cell lines reversed ZAR1 promoter hypermethylation and subsequently re-established ZAR1 expression. …”
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  8. 8
    por Zar, Jerrold H., 1941-
    Publicado 1996
    Libro
  9. 9
    por Zar, Jerrold H., 1941-
    Publicado 2010
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    Libro
  10. 10
    por Zar, Jerrold H., 1941-
    Publicado 1999
    Libro
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  12. 12
    “…RESULTS: We are the first to report that ZAR1 is epigenetically inactivated not only in lung cancer but also across cancer types, and ZAR1 methylation occurs across its complete CpG island. …”
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  13. 13
    “…The Arabidopsis nucleotide-binding leucine-rich repeat protein ZAR1 can recognize at least six distinct families of pathogenic effector proteins to mount an effector-triggered immune response. …”
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    “…Here, we analyzed the methylation status of ZAR1, which has been reported to be aberrantly methylated in a few human cancers. …”
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    “…The zar1-3 zar2-1 double mutant exhibited stronger resistance to B. cinerea compared with zar1-3 single mutant, zar2-1 single mutant, and wild-type plants. …”
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  18. 18
    “…While studying the activity of BRCA2 gene promoter in breast cancer cells, we discovered that this promoter has bi-directional activity and the product of the reverse activity (a ZAR1-like protein, we named ZAR2) silences the forward promoter at the G0/G1 phase of the cell. …”
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  19. 19
    por Shirazi, Abolfazl, Motaghi, Ehsan
    Publicado 2013
    “…Moreover, no relationship was observed between the relative abundance of Zar1 transcript in zygotes produced in different conditions and the corresponding subsequent embryonic development.…”
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  20. 20
    “…In the patient-derived glioma cell lines NULU and ZAR, which exhibit drug-resistant phenotypes, we investigated the effect of combined AE (Aloe-emodin) and TMZ (temozolomide) and found a significant additive inhibitory effect on cell growth and a promising cytotoxic effect on both cell lines compared to treatment with single agents. …”
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