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  1. 2001
    por Cheung, Yuk-Yam, Hui, Mamie
    Publicado 2017
    “…We evaluated the in vitro and in vivo effects of nikkomycin Z combined with an echinocandin (anidulafungin or micafungin) against two Candida albicans isolates and their lab-derived echinocandin-resistant fks mutants with FKS1 S645Y and FKS1 S645P. …”
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    Effects of Echinocandins in Combination with Nikkomycin Z against Invasive Candida albicans Bloodstream Isolates and the fks Mutants
  2. 2002
    “…RESULTS: In total, 35 non-albicans Candida isolates were identified from VVC patients. …”
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    Molecular Identification and Antifungal Susceptibility Pattern of Non-albicans Candida Species Isolated from Vulvovaginal Candidiasis
  3. 2003
    “…Candida albicans is an important opportunistic fungal pathogen capable of causing both mucosal and disseminated disease. …”
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    A chromosome 4 trisomy contributes to increased fluconazole resistance in a clinical isolate of Candida albicans
  4. 2004
    “…Mitochondrial respiration and steady state cellular phospholipid homeostasis are not impacted by inactivation of GEM1 in C. albicans. There are two major virulence-related consequences of disrupting mitochondrial morphology by GEM1 inactivation: C. albicans becomes hypersusceptible to cell wall stress, and is unable to grow invasively. …”
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    The Mitochondrial GTPase Gem1 Contributes to the Cell Wall Stress Response and Invasive Growth of Candida albicans
  5. 2005
    por Alizadeh, F, Khodavandi, A, Zalakian, S
    Publicado 2017
    “…Ergosterol content and gene expression profiling of sterol 14α-demethylase (ERG11) gene in fluconazole-susceptible and –resistant C. albicans were investigated. RESULTS: The specimens consisted of C. albicans (46.67%), Candida krusei (41.67%), and Candida tropicalis (11.67%). …”
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    Quantitation of ergosterol content and gene expression profile of ERG11 gene in fluconazole-resistant Candida albicans
  6. 2006
    “…The transition from commensalism to pathogenicity of Candida albicans reflects both the host inability to mount specific immune responses and the microorganism’s dimorphic switch efficiency. …”
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    Genomic and Phenotypic Variation in Morphogenetic Networks of Two Candida albicans Isolates Subtends Their Different Pathogenic Potential
  7. 2007
    “…BACKGROUND: Cinnamon (Cinnamomum zeylanicum) bark extract exhibits potent inhibitory activity against Candida albicans but the antifungal mechanisms of this essential oil remain largely unexplored. …”
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    Cinnamomum zeylanicum bark essential oil induces cell wall remodelling and spindle defects in Candida albicans
  8. 2008
    “…Candida albicans is the leading cause of fungal infections; yet, complex genetic interaction analysis remains cumbersome in this diploid pathogen. …”
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    A CRISPR Cas9-based gene drive platform for genetic interaction analysis in Candida albicans
  9. 2009
    “…Here, we used the intracellular zinc reporter, zinbo-5, which allowed visualization of zinc in the endoplasmic reticulum and other components of the internal membrane system in Candida albicans. We provide evidence for a link between cyclic AMP/PKA- and zinc-signaling in this major human fungal pathogen. …”
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    Cyclic AMP Pathway Activation and Extracellular Zinc Induce Rapid Intracellular Zinc Mobilization in Candida albicans
  10. 2010
    “…Materials and Methods: E. faecalis (ATCC 29212) and C. albicans (ATCC 90028) standard strains were used for this study. …”
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    Determination of the Minimum Inhibitory Concentrations of Alexidine and Chlorhexidine Against Enterococcus faecalis and Candida albicans: An In Vitro Study
  11. 2011
    “…The hyphal development of Candida albicans (C. albicans) has been considered as an essential virulent factor for host cell damage. …”
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    ERG3 and ERG11 genes are critical for the pathogenesis of Candida albicans during the oral mucosal infection
  12. 2012
    “…In contrast, luteal progesterone increases epithelial expression of CD44 and CD47 to promote neutrophil migration into the vaginal lumen and Candida albicans destruction. Distinctive to vaginal mucosa, neutrophil infiltration is contingent to sex hormones to prevent sperm from neutrophil attack; although it may compromise immunity during ovulation. …”
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    Estrogen Receptor-Alpha (ESR1) Governs the Lower Female Reproductive Tract Vulnerability to Candida albicans
  13. 2013
    por Jiang, Linghuo, Pan, Hongbo
    Publicado 2018
    “…RESULTS: We show here that CaPhm7 is localized to the plasma membrane in both the yeast and hyphal cells of C. albicans. C. albicans cells lacking CaPHM7 are sensitive to SDS and ketoconazole but tolerant to rapamycin and zinc. …”
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    Functions of CaPhm7 in the regulation of ion homeostasis, drug tolerance, filamentation and virulence in Candida albicans
  14. 2014
    “…The data presented here are related to the research article entitled “Knockout of autophagy gene, ATG5 in mice vaginal cells abrogates cytokine response and pathogen clearance during vaginal infection of Candida albicans” (Shroff et al., 2018) [1]. The cited research article describes the role of autophagy in host immune response against C. albicans infection of mice vagina. …”
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    Data on autophagy markers and anti-candida cytokines expression in mice in response to vaginal infection of Candida albicans
  15. 2015
    “…Considering the significance in survival and virulence, we have made an attempt to understand modulations in the membrane and cell wall properties of Candida albicans hyphae induced by temperature (37 °C) and neutral pH and yeast form cells grown under low hydrostatic pressure (LHP). …”
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    Microenvironment Responsive Modulations in the Fatty Acid Content, Cell Surface Hydrophobicity, and Adhesion of Candida albicans Cells
  16. 2016
    “…Key words:Terminalia catappa Linn, Biofilm, Candida albicans.…”
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    Effects of Terminalia catappa Linn. Extract on Candida albicans biofilms developed on denture acrylic resin discs
  17. 2017
    “…Candida albicans is one of the most common causes of hospital-acquired urinary tract infections (UTIs). …”
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    Cranberry-derived proanthocyanidins induce a differential transcriptomic response within Candida albicans urinary biofilms
  18. 2018
    “…Candida albicans TIMM1768 is a highly virulent strain utilized as a model organism for the study of gastrointestinal and oral candidiasis. …”
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    Utilization of Hybrid Assembly Approach to Determine the Genome of an Opportunistic Pathogenic Fungus, Candida albicans TIMM 1768
  19. 2019
    “…The expansion of Azoles resistance among C. albicans strains is considered an important medical problem. …”
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    Antifungal effects of ethanolic and aqueous extracts of Vitex agnus-castus against vaginal isolates of Candida albicans
  20. 2020
    “…Candida albicans is an opportunistic pathogen and responsible for candidiasis. …”
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    Antibiofilm and Antivirulence Activities of 6-Gingerol and 6-Shogaol Against Candida albicans Due to Hyphal Inhibition
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