“…Despite a growing number of genome data available, little is known about C. albicans, a major fungal pathogen. Most of the human population carries C. albicans as commensal, but it can cause systemic infection that may lead to the death of the host if the immune system has deteriorated. …”
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“…Candida albicans is an opportunistic pathogen and is recognised and phagocytosed by macrophages. …”
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“…The pathogenicity of the opportunistic human fungal pathogen Candida albicans depends on its ability to inhibit effective destruction by host phagocytes. …”
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“…RESULTS: When evaluated together, the adherence of S. mutans and C. albicans was increased in the ceramic bracket group. …”
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“…The transcription factors downstream of these pathways and their DNA binding sites within the promoters of target genes are well characterised in Saccharomyces cerevisiae, but not in C. albicans. The promoters of the C. albicans class I CHS genes (CaCHS2 and CaCHS8) were functionally dissected with the aim of identifying and characterising the transcription factors and promoter elements that mediate the transcriptional up-regulation of CaCHS2 and CaCHS8 in response to cell wall stress. …”
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“…A striking and clinically relevant virulence trait of the human fungal pathogen Candida albicans is its ability to grow and switch reversibly among different morphological forms. …”
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“…Although there is considerable infor-mation about the adherence of Candida albicans to buccal epithelial cells and prosthetic materials, there is very little infor-mation available about the adherence of Candida albicans to glass ionomer materials. …”
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“…In the present study, we found that the Pph3–Psy2 phosphatase complex is responsible for Rfa2 dephosphorylation both during normal G(1)-phase and under DNA replication stress in Candida albicans. Phosphorylated Rfa2 extracted from pph3Δ or psy2Δ G(1) cells exhibited diminished binding affinity to dsDNA (double-stranded DNA) but not to ssDNA. …”
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“…Systemic infections of humans with the fungal pathogen Candida albicans are associated with a high mortality rate. …”
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“…Indeed, the yeast proteins are rapidly degraded when expressed in C. albicans, indicating a conservation of the machinery needed for CCI. …”
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“…This study investigated the efficacy of calcium hydroxide and chlorhexidine gel for the elimination of intratubular Candida albicans (C. albicans). Human single-rooted teeth contaminated with C. albicans were treated with calcium hydroxide, 2% chlorhexidine gel, calcium hydroxide plus 2% chlorhexidine gel, or saline (0.9% sodium chloride) as a positive control. …”
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“…Candida albicans is an opportunistic human fungal pathogen that causes candidiasis. …”
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“…The effects of low molecular weight (96.5 KDa) chitosan on the pathogenic yeast Candida albicans were studied. Low concentrations of chitosan, around 2.5 to 10 μg·mL(−1) produced (a) an efflux of K(+) and stimulation of extracellular acidification, (b) an inhibition of Rb(+) uptake, (c) an increased transmembrane potential difference of the cells, and (d) an increased uptake of Ca(2+). …”
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“…These findings suggested that C. albicans reproduces mainly in a clonal manner, with certain degree of DNA microevolution. …”
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“…Candida albicans is an opportunistic and polymorphic fungal pathogen that causes mucosal, disseminated and invasive infections in humans. …”
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“…RESULTS: The growth rate of Candida albicans was determined in the cultures. The results indicated that the honey prevented the growth of C. albicans greatly only at an 80% concentration, whereas, miconazole inhibited it completely. …”
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“…This study presents the first global transcriptional profiling and phenotypic characterization of the major human opportunistic fungal pathogen, Candida albicans, grown in spaceflight conditions. Microarray analysis revealed that C. albicans subjected to short-term spaceflight culture differentially regulated 452 genes compared to synchronous ground controls, which represented 8.3% of the analyzed ORFs. …”
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“…Candida albicans invades endothelial cells by binding to N-cadherin and other cell surface receptors. …”
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