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  1. 1501
    “…Identification and characterization of newer drugs against C. albicans is, therefore, essential. MMGP1 is a direct cell-penetrating peptide recently identified from marine metagenome, which was found to possess potent antifungal activity against C. albicans. …”
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    Mechanisms of the Antifungal Action of Marine Metagenome-Derived Peptide, MMGP1, against Candida albicans
  2. 1502
    “…The National Institutes of Health Molecular Libraries and Probe Production Centers Network (NIH-MLPCN) screened >300,000 compounds to evaluate their ability to restore fluconazole susceptibility in resistant Candida albicans isolates. Additional counter screens were incorporated to remove substances inherently toxic to either mammalian or fungal cells. …”
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    ML212: A small-molecule probe for investigating fluconazole resistance mechanisms in Candida albicans
  3. 1503
    “…Here, we report that exposure of C. albicans to the mammalian gut triggers a developmental switch, driven by the Wor1 transcription factor, to a commensal cell type. …”
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    Passage through the mammalian gut triggers a phenotypic switch that promotes Candida albicans commensalism
  4. 1504
    “…Candida albicans is responsible for a number of life-threatening infections and causes considerable morbidity and mortality in immunocompromised patients. …”
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    Dynamic Transcript Profiling of Candida albicans Infection in Zebrafish: A Pathogen-Host Interaction Study
  5. 1505
    “…Candida albicans frequently cause oropharyngeal candidiasis in immunocompromised patients. …”
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    Susceptibility tests of oropharyngeal Candida albicans from egyptian patients to fluconazole determined by three methods
  6. 1506
    “…Candida albicans is a human commensal and clinically important fungal pathogen that grows as both yeast and hyphal forms during human, mouse and zebrafish infection. …”
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    NADPH Oxidase-Driven Phagocyte Recruitment Controls Candida albicans Filamentous Growth and Prevents Mortality
  7. 1507
    “…PMNs were more susceptible to C. albicans-mediated killing than macrophages, irrespective of the number of C. albicans cells ingested. …”
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    Altered Dynamics of Candida albicans Phagocytosis by Macrophages and PMNs When Both Phagocyte Subsets Are Present
  8. 1508
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    Fungal Immune Evasion in a Model Host–Pathogen Interaction: Candida albicans Versus Macrophages
  9. 1509
    “…Invasive growth of the fungal pathogen Candida albicans into tissues promotes disseminated infections in humans. …”
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    The MARVEL Domain Protein Nce102 Regulates Actin Organization and Invasive Growth of Candida albicans
  10. 1510
    “…RESULTS: To further elucidate the function of CaCDC4, a C. albicans strain, with one CaCDC4 allele deleted and the other under the repressible C. albicans MET3 promoter (CaMET3p) control, was made before introducing cassettes capable of doxycycline (Dox)-induced expression of various C. albicans Cdc4 (CaCdc4) domains. …”
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    Dissection of the Candida albicans Cdc4 protein reveals the involvement of domains in morphogenesis and cell flocculation
  11. 1511
    “…No statistical difference was found between primary and secondary root canal infections for C.albicans or E.faecalis. Key words:Primary root canal infection, secondary root canal infection, E.faecalis, C.albicans.…”
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    Polymerase chain reaction of enterococcus faecalis and candida albicans in apical periodontitis from Turkish patients
  12. 1512
    “…Here, we report functional characterization of Nap1 in the human-pathogenic fungus Candida albicans. We find that deletion of NAP1 causes constitutive filamentous growth and changes of septin dynamics. …”
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    Phosphoregulation of Nap1 Plays a Role in Septin Ring Dynamics and Morphogenesis in Candida albicans
  13. 1513
    “…We report here that C. albicans actively inhibits macrophage production of nitric oxide (NO). …”
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    Candida albicans Suppresses Nitric Oxide Generation from Macrophages via a Secreted Molecule
  14. 1514
    “…Candida albicans is an opportunistic pathogen that causes candidiasis in humans. …”
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    Inhibitors of the Glyoxylate Cycle Enzyme ICL1 in Candida albicans for Potential Use as Antifungal Agents
  15. 1515
    “…The adhesive phenotype of Candida albicans contributes to its ability to colonize the host and cause disease. …”
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    The Peptide-binding Cavity Is Essential for Als3-mediated Adhesion of Candida albicans to Human Cells
  16. 1516
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    Profile susceptibility to fluconazole and voriconazole antifungals by species of Candida albicans isolated from urine culture
  17. 1517
    “…The commensal yeast Candida albicans is part of the human intestinal microflora and is considered a “pathobiont”, a resident microbe with pathogenic potential yet harmless under normal conditions. …”
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    Intestinal Colonization by Candida albicans Alters Inflammatory Responses in Bruton's Tyrosine Kinase-Deficient Mice
  18. 1518
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    Erratum to: Modulatory role of vitamin A on the Candida albicans-induced immune response in human monocytes
  19. 1519
    “…It was discovered that the OPI1 homolog does not regulate INO1 expression in C. albicans, but it does control SAP2 expression in response to bovine serum albumin containing media. …”
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    Candida albicans OPI1 Regulates Filamentous Growth and Virulence in Vaginal Infections, but Not Inositol Biosynthesis
  20. 1520
    “…Candida albicans is an opportunistic dimorphic fungus that inhabits various host mucosal sites. …”
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    Treatment with some anti-inflammatory drugs reduces germ tube formation in Candida albicans strains
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