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  1. 1541
    “…BACKGROUND: The fungal pathogen Candida albicans colonizes the gastrointestinal (GI) tract of mammalian hosts as a benign commensal. …”
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    The game theory of Candida albicans colonization dynamics reveals host status-responsive gene expression
  2. 1542
    “…Most surface-accessible glucan on C. albicans yeast and hyphae is limited to isolated Dectin-1–binding sites. …”
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    Nanoscopic cell-wall architecture of an immunogenic ligand in Candida albicans during antifungal drug treatment
  3. 1543
    “…We also show that C. albicans Sec15 interacts directly with the polarity determinant Bem1 and the type V myosin, Myo2. …”
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    Sec15 links bud site selection to polarised cell growth and exocytosis in Candida albicans
  4. 1544
    por Lee, Seung-Bae
    Publicado 2016
    “…OBJECTIVES: The purpose of this study was to investigate the antifungal effect of bee venom (BV) and sweet bee venom (SBV) against Candida albicans (C. albicans) clinical isolates. METHODS: In this study, BV and SBV were examined for antifungal activities against the Korean Collection for Type Cultures (KCTC) strain and 10 clinical isolates of C. albicans. …”
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    Antifungal Activity of Bee Venom and Sweet Bee Venom against Clinically Isolated Candida albicans
  5. 1545
    “…The multidrug transporter proteins Cdr1p and Cdr2p of the ABCG subfamily are major players in the development of MDR in Candida albicans. Because several genes coding for ABC proteins exist in the genome of C. albicans, but only Cdr1p and Cdr2p have established roles in MDR, it is implicit that the other members of the ABC family also have alternative physiological roles. …”
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    Pleiotropic effects of the vacuolar ABC transporter MLT1 of Candida albicans on cell function and virulence
  6. 1546
    “…Candida albicans and Fusobacterium nucleatum are well-studied oral commensal microbes with pathogenic potential that are involved in various oral polymicrobial infectious diseases. …”
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    Morphological and physiological changes induced by contact-dependent interaction between Candida albicans and Fusobacterium nucleatum
  7. 1547
    “…UV exposure reduced Candida albicans antigen-induced delayed-type hypersensitivity. …”
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    Prevention of ultraviolet radiation-induced immunosuppression by sunscreen in Candida albicans-induced delayed-type hypersensitivity
  8. 1548
    “…The purpose of this study was to investigate whether immersion of a denture surface in lemongrass extract (LGE) has effects on C. albicans biofilms, human cell viability and denture surface. …”
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    In vitro Effects of Lemongrass Extract on Candida albicans Biofilms, Human Cells Viability, and Denture Surface
  9. 1549
    “…Chorioamnionitis is caused by intrauterine infection with microorganisms including Candida albicans (C.albicans). Chorioamnionitis is associated with postnatal intestinal pathologies including necrotizing enterocolitis. …”
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    Intra-amniotic Candida albicans infection induces mucosal injury and inflammation in the ovine fetal intestine
  10. 1550
    “…Together, these studies not only reveal why PPZ1 does not negatively impact GLC7 activity in vivo but also demonstrate that the family of fungus-specific phosphatases—especially PPZ1 from C. albicans—are highly suitable targets for the development of novel drugs that specifically target C. albicans without cross-reacting with human phosphatases.…”
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    Molecular Insights into the Fungus-Specific Serine/Threonine Protein Phosphatase Z1 in Candida albicans
  11. 1551
    “…The group treated with adhesive showed high fungal growth compared to the control group, whereas chlorhexidine showed high potency to prevent C. albicans, whereas adhesive increased the adhesion of C. albicans to acrylic surface. …”
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    Comparative assessment of the effectiveness of different cleaning methods on the growth of Candida albicans over acrylic surface
  12. 1552
    “…During interactions with its mammalian host, the pathogenic yeast Candida albicans is exposed to a range of stresses such as superoxide radicals and cationic fluxes. …”
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    Pho4 mediates phosphate acquisition in Candida albicans and is vital for stress resistance and metal homeostasis
  13. 1553
    “…In this work, we examined the antifungal activity of the eosinophil RNase 3 and the skin‐derived RNase 7, two proteins expressed by innate cell types that are directly involved in the host defense against fungal infection. Candida albicans has been selected as a suitable working model for testing RNase activities toward a eukaryotic pathogen. …”
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    Exploring the mechanisms of action of human secretory RNase 3 and RNase 7 against Candida albicans
  14. 1554
    “…Our data highlight how mechanisms regulating genome stability are rewired in C. albicans.…”
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    Sir2 regulates stability of repetitive domains differentially in the human fungal pathogen Candida albicans
  15. 1555
    “…The principal aim of this study was to investigate the in vitro co-infection of Caco-2 cells with Candida albicans and enterohemorrhage Escherichia coli (EHEC). …”
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    Enterohemorrhagic Escherichia coli promotes the invasion and tissue damage of enterocytes infected with Candida albicans in vitro
  16. 1556
    “…Planktonic cells of C. albicans, C. tropicalis and their 1:1 co-culture showed maximal growth in SDB. …”
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    Culture media profoundly affect Candida albicans and Candida tropicalis growth, adhesion and biofilm development
  17. 1557
    “…CONCLUSIONS: Collectively, these results indicate that honokiol acts as a pro-oxidant in C. albicans. Furthermore, the SOD inhibitor DDC can be used to potentiate the activity of honokiol against C. albicans.…”
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    Honokiol induces reactive oxygen species-mediated apoptosis in Candida albicans through mitochondrial dysfunction
  18. 1558
    “…Our results clarify that membrane disruption appears to be the mechanism by which the C. albicans cells are killed and this occurs after FITC-PuroA translocation and binding to intracellular targets.…”
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    Revealing the sequence of interactions of PuroA peptide with Candida albicans cells by live-cell imaging
  19. 1559
    “…Using the non-vertebrate model host Galleria mellonella, we evaluated whether clinical isolates of Lactobacillus spp. are able to provide protection against Candida albicans infection. Among different strains of Lactobacillus paracasei, Lactobacillus rhamnosus and Lactobacillus fermentum, we verified that L. paracasei 28.4 strain had the greatest ability to prolong the survival of larvae infected with a lethal dose of C. albicans. …”
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    Lactobacillus paracasei modulates the immune system of Galleria mellonella and protects against Candida albicans infection
  20. 1560
    “…C. albicans-induced pIEC damage was reduced to a similar extent by multiple different C. parapsilosis strains, but strains differed in their ability to inhibit C. albicans invasion of pIECs, with the inhibitory activity correlating with their adhesiveness for C. albicans and epithelial cells. …”
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    Candida parapsilosis Protects Premature Intestinal Epithelial Cells from Invasion and Damage by Candida albicans
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