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  1. 1681
    “…The complex fungal cell wall of the prevalent pathogen, Candida albicans, is one of the main targets of antifungal drugs and recognized by host immune cells. …”
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    Revisiting the Vital Drivers and Mechanisms of β-Glucan Masking in Human Fungal Pathogen, Candida albicans
  2. 1682
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    The Emerging Uncommon Non-Albicans Candida: Candida Parapsilosis Peritonitis in a Peritoneal Dialysis Patient
  3. 1683
    “…To determine the effect of 3% boric acid solution on cutaneous infections with Candida albicans (CA) in mice and its effect on skin microflora. …”
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    Effects of 3% Boric Acid Solution on Cutaneous Candida albicans Infection and Microecological Flora Mice
  4. 1684
    “…We investigated the relationship between malnutrition and all-cause 28-day mortality among patients with non-albicans candidemia. Between July 2011 and June 2014, all adult patients with non-albicans candidemia, including C. tropicalis, C. glabrata, C. parapsilosis and so on, were enrolled. …”
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    Impact of Nutritional Assessment on the Clinical Outcomes of Patients with Non-albicans Candidemia: A Multicenter Study
  5. 1685
    “…This study evaluates the effects of different programs of complex electromagnetic fields (C.M.F.s) on Candida albicans, in planktonic and sessile phase and on human gingival fibroblasts (HGF cells). …”
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    Complex Electromagnetic Fields Reduce Candida albicans Planktonic Growth and Its Adhesion to Titanium Surfaces
  6. 1686
    “…The opportunistic fungal pathogen Candida albicans undergoes an unusual parasexual cycle wherein diploid cells mate to form tetraploid cells that can generate genetically diverse progeny via a nonmeiotic program of chromosome loss. …”
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    Adaptation to the dietary sugar D-tagatose via genome instability in polyploid Candida albicans cells
  7. 1687
    “…Here, we show that deletion of the Candida albicans gene encoding the δ subunit of the F(1)F(o)-ATP synthase (ATP16) abrogates lethal infection in a mouse model of systemic candidiasis. …”
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    The δ subunit of F(1)F(o)-ATP synthase is required for pathogenicity of Candida albicans
  8. 1688
    “…The C. albicans ent2Δ/Δ null mutant displayed cell wall defects and altered antifungal drug sensitivity. …”
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    Candida albicans ENT2 Contributes to Efficient Endocytosis, Cell Wall Integrity, Filamentation, and Virulence
  9. 1689
    “…PURPOSE: The resistance of C. albicans to traditional antifungal drugs brings a great challenge to clinical treatment. …”
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    Antifungal Effects and Potential Mechanisms of Benserazide Hydrochloride Alone and in Combination with Fluconazole Against Candida albicans
  10. 1690
    “…However, the role played by C. albicans in macrophage foaming and the associated inflammation is poorly understood. …”
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    Candida albicans Induces Foaming and Inflammation in Macrophages through FABP4: Its Implication for Atherosclerosis
  11. 1691
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    Author Correction: Candida albicans biofilm development is governed by cooperative attachment and adhesion maintenance proteins
  12. 1692
    “…We show here that simultaneous activation of the calcineurin and CWI pathways generates a subpopulation of Candida albicans yeast cells that have supra-normal chitin levels interspersed throughout the inner and outer cell wall, and that these cells are non-viable, perhaps due to loss of wall elasticity required for cell expansion and growth. …”
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    Crosstalk between the calcineurin and cell wall integrity pathways prevents chitin overexpression in Candida albicans
  13. 1693
    “…Candida albicans is a commensal of the urogenital tract and the predominant cause of vulvovaginal candidiasis (VVC). …”
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    Estrogen promotes innate immune evasion of Candida albicans through inactivation of the alternative complement system
  14. 1694
    “…In this study, an immunosuppressive mouse model of invasive Candida albicans (C. albicans) infection was constructed to observe the effects of cinnamaldehyde (CA) on the C. albicans cell wall structure and cell wall (1,3)-β-D-glucan contents. …”
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    Effect of Cinnamaldehyde on C. albicans cell wall and (1,3)- β – D-glucans in vivo
  15. 1695
    “…Analysis of the derivatives acting on the most abundant naturally occurring Candida yeast Candida albicans (C. albicans) revealed a strong anti-biofilm activity, especially induced by Pom-1 B, C, and D. …”
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    Increased Activities against Biofilms of the Pathogenic Yeast Candida albicans of Optimized Pom-1 Derivatives
  16. 1696
    “…MATERIALS AND METHODS: C. albicans CA1398, carrying the bioluminescence ACT1p-gLUC59 fusion product, was employed. …”
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    Effects of benzydamine and mouthwashes containing benzydamine on Candida albicans adhesion, biofilm formation, regrowth, and persistence
  17. 1697
    “…In addition, photoinactivation of catalase is shown to facilitate macrophage killing of intracellular Candida albicans. The antifungal efficacy of catalase photoinactivation is further validated using a C. albicans‐induced mouse model of skin abrasion. …”
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    Photoinactivation of Catalase Sensitizes Candida albicans and Candida auris to ROS‐Producing Agents and Immune Cells
  18. 1698
    “…CONCLUSION: The C. glabrata and C. albicans species showed the highest resistance to antifungal drugs. …”
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    High incidence of azole resistance among Candida albicans and C. glabrata isolates in Northeastern Iran
  19. 1699
    “…Smoking triggers environmental changes in the oral cavity and increases the risk of mucosal infections caused by Candida albicans such as oral candidiasis. While cigarette smoke has a significant impact on C. albicans, how e-cigarettes affect this oral pathogen is less clear. …”
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    Effect of Cigarette and E-Cigarette Smoke Condensates on Candida albicans Biofilm Formation and Gene Expression
  20. 1700
    “…The most common fungal strains involved in infections belong to the yeast species Candida albicans. The main virulence factor of C. albicans is the ability to transform from planktonic yeast-form cells into a filamentous form (hyphae or pseudohyphae), with the subsequent formation of biofilm. …”
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    The use of polyhydroxylated carboxylic acids and lactones to diminish biofilm formation of the pathogenic yeast Candida albicans
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