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  1. 1921
    “…To understand differences in host-Candida albicans interactions that occur during colonization of healthy or compromised hosts, production of phenotypic variants and colonization of healthy or immunodeficient mice by C. albicans were studied. …”
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    Variation in Candida albicans EFG1 Expression Enables Host-Dependent Changes in Colonizing Fungal Populations
  2. 1922
    “…However, we show that there is significant dislocation between the proteome and transcriptome in C. albicans. Glucose triggers the degradation of the ICL1 and PCK1 transcripts in C. albicans, yet isocitrate lyase (Icl1) and phosphoenolpyruvate carboxykinase (Pck1) are stable and are retained. …”
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    The Evolutionary Rewiring of Ubiquitination Targets Has Reprogrammed the Regulation of Carbon Assimilation in the Pathogenic Yeast Candida albicans
  3. 1923
    “…Candida albicans, a common fungal pathogen which diverged from the baker’s yeast Saccharomyces cerevisiae has the unique ability to utilise N-acetylglucosamine, an amino sugar and exhibits phenotypic differences. …”
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    N-Acetylglucosamine Kinase, HXK1 Is Involved in Morphogenetic Transition and Metabolic Gene Expression in Candida albicans
  4. 1924
    “…Three of these opportunistic fungal pathogens: Aspergillus fumigatus, Candida albicans and Cryptococcus neoformans pose the biggest concern for the immune-compromised host. …”
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    The role of dendritic cells in the innate recognition of pathogenic fungi (A. fumigatus, C. neoformans and C. albicans)
  5. 1925
    por Su, Chang, Lu, Yang, Liu, Haoping
    Publicado 2013
    “…Candida albicans is able to undergo reversible morphological changes between yeast and hyphal forms in response to environmental cues. …”
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    Reduced TOR signaling sustains hyphal development in Candida albicans by lowering Hog1 basal activity
  6. 1926
    “…Combination drug therapies are employed to treat patients with HIV, cancer, or tuberculosis, and has considerable promise in the treatment of fungal infections like cryptococcal meningitis and C. albicans infections. Our studies reported here demonstrate that posaconazole exhibits in vitro synergy with caspofungin or FK506 against drug susceptible or resistant C. albicans strains. …”
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    Posaconazole Exhibits In Vitro and In Vivo Synergistic Antifungal Activity with Caspofungin or FK506 against Candida albicans
  7. 1927
    “…The network is enriched with genes upregulated in C. albicans cells growing in the host. Our findings indicate that many aspects of commensalism and pathogenicity are intertwined and that the ability of this microorganism to colonize multiple niches relies on a large, integrated circuit.…”
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    Candida albicans Commensalism and Pathogenicity Are Intertwined Traits Directed by a Tightly Knit Transcriptional Regulatory Circuit
  8. 1928
    “…Oropharyngeal candidiasis (OPC) is an opportunistic infection caused by Candida albicans. Despite its prevalence, little is known about C. albicans-specific immunity in the oral mucosa. …”
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    Th17 cells confer long term adaptive immunity to oral mucosal Candida albicans infections
  9. 1929
    “…Candida albicans is the most common human fungal pathogen causing mucosal and systemic infections. …”
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    Functional genomics identifies type I interferon pathway as central for host defense against Candida albicans
  10. 1930
    “…Therefore, α1,2-mannose of N-mannan is important for both immune recognition and virulence of C. albicans.…”
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    The Mnn2 Mannosyltransferase Family Modulates Mannoprotein Fibril Length, Immune Recognition and Virulence of Candida albicans
  11. 1931
    “…The high affinity iron uptake system of the opportunistic pathogenic yeast Candida albicans has been shown to be essential for virulence. …”
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    Involvement of the mitogen activated protein kinase Hog1p in the response of Candida albicans to iron availability
  12. 1932
    “…Chitosan was shown to effectively augment the effect of PDI mediated by toluidine blue O (TBO) against C. albicans that were incubated with chitosan for 30 min following PDI. …”
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    The Use of Chitosan to Enhance Photodynamic Inactivation against Candida albicans and Its Drug-Resistant Clinical Isolates
  13. 1933
    “…We report a case of Candida albicans endocarditis in a child with acute lymphoblastic leukemia on chemotherapy. …”
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    Candida albicans endocarditis in a child with acute lymphoblastic leukemia: A dreaded complication of intensive chemotherapy
  14. 1934
    “…The purpose of this study was to evaluate the time to positivity (TTP) of blood cultures in patients with Candida albicans BSIs and to assess its impact on clinical outcome. …”
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    Time to blood culture positivity as a predictor of clinical outcome in patients with Candida albicans bloodstream infection
  15. 1935
    “…INTRODUCTION: An important consideration in endodontic treatment is the elimination of microorganisms, including fungi, from the complex three- dimensional root canal system. Candida Albicans (CA) has a major role in endodontic treatment failure as the most important fungus isolated from the root canal system. …”
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    Presence of Candida Albicans in Root Canal System of Teeth Requiring Endodontic Retreatment with and without Periapical Lesions
  16. 1936
    por Buu, Leh-Miauh, Chen, Yee-Chun
    Publicado 2013
    “…BACKGROUND: The polymorphic species Candida albicans is the major cause of candidiasis in humans. …”
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    Sap6, a secreted aspartyl proteinase, participates in maintenance the cell surface integrity of Candida albicans
  17. 1937
    “…In this study, we demonstrated that a subset of murine J774A.1 macrophage cell line (8% to 17%) and peritoneal macrophages (8.5% to 15%) form METs-like structures (METs-LS) in response to Escherichia coli and Candida albicans challenge. We found only a portion of murine METs-LS, which are released by dying macrophages, showed detectable killing effects on trapped E. coli but not C. albicans. …”
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    Escherichia coli and Candida albicans Induced Macrophage Extracellular Trap-Like Structures with Limited Microbicidal Activity
  18. 1938
    “…Here we show that the hyphae of the human fungal pathogen Candida albicans continue to extend throughout the whole of mitosis. …”
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    In Candida albicans, phosphorylation of Exo84 by Cdk1-Hgc1 is necessary for efficient hyphal extension
  19. 1939
    por Bai, Feng-Yan
    Publicado 2014
    “…Candida albicans is a commensal microorganism in the mucosa of healthy individuals, but is also the most common opportunistic fungal pathogen of humans. …”
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    Association of genotypes with infection types and antifungal susceptibilities in Candida albicans as revealed by recent molecular typing strategies
  20. 1940
    “…The ability of C. albicans to form biofilms is a major virulence factor and a challenge for management. …”
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    A Novel Antifungal Is Active against Candida albicans Biofilms and Inhibits Mutagenic Acetaldehyde Production In Vitro
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