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“…The topics covered include, the Dirichlet problem (after Bedford–Taylor), Monge–Ampère foliations and laminated currents, polynomial hulls and Perron envelopes with no analytic structure, a self-contained presentation of Krylov regularity results, a modernized proof of the Calabi–Yau theorem (after Yau and Kolodziej), an introduction to infinite dimensional riemannian geometry, geometric structures on spaces of Kähler metrics (after Mabuchi, Semmes and Donaldson), generalizations of the regularity theory of Caffarelli–Kohn–Nirenberg–Spruck (after Guan, Chen and Blocki) and Bergman approximation of geodesics (after Phong–Sturm and Berndtsson). …”
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“…Antipov, A. S. Trubnikov, AYu. Rylov, AV. Khutoretsky) and astrophysics theoreticians from the Astronomical Council of the USSR Academy of Sciences (AM. …”
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“…Contributions by Shavkat Ayupov, Amine Ben Amor, Karim Boulabiar, Qingying Bu, Gerard Buskes, Martijn Caspers, Jurie Conradie, Garth Dales, Marcel de Jeu, Peter Dodds, Theresa Dodds, Julio Flores, Jochen Glück, Jacobus Grobler, Wolter Groenevelt, Markus Haase, Klaas Pieter Hart, Francisco Hernández, Jamel Jaber, Rien Kaashoek, Turabay Kalandarov, Anke Kalauch, Arkady Kitover, Erik Koelink, Karimbergen Kudaybergenov, Louis Labuschagne, Yongjin Li, Nick Lindemulder, Emiel Lorist, Qi Lü, Miek Messerschmidt, Susumu Okada, Mehmet Orhon, Denis Potapov, Werner Ricker, Stephan Roberts, Pablo Román, Anton Schep, Claud Steyn, Fedor Sukochev, James Sweeney, Guido Sweers, Pedro Tradacete, Jan Harm van der Walt, Onno van Gaans, Jan van Neerven, Arnoud van Rooij, Freek van Schagen, Dominic Vella, Mark Veraar, Anthony Wickstead, Marten Wortel, Ivan Yaroslavtsev, and Dmitriy Zanin.…”
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“…On one hand, the breakdown of immune homeostasis directly induces systemic inflammation; on the other hand, these cells initiate and perpetuate synovitis and tissue damages through the interaction with fibroblast-like synoviocytes (FLS). …”
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“…However, it has been documented to play a role in other conditions, including SAPHO syndrome (synovitis, acne, pustulosis, hyperostosis, osteitis), a rare autoinflammatory disorder. …”
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“…Long-term complications of hemophilia such as arthropathy, synovitis, and arthritis can lead to the development of recurrent chronic pain. …”
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“…It was also observed in adjuvant-induced arthritis (AIA) mice that FAP-targeted ZF-NPs (FAP-ZF-NPs) could significantly suppress synovitis in vivo, inhibit synovial tissue angiogenesis, protect articular cartilage, and reduce M1 macrophage infiltration in synovium in AIA mice. …”
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“…The highest predictive value of treatment failure was obtained with the combination of increased circulating SEMA4A and/or Disease Activity Score (DAS) 28-CRP > 3.2 and/or active synovitis on doppler ultrasound (HR 10.42, 95% CI 1.41–76.94). …”
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“…We present a case of a 60-year-old male with ACTS secondary to acute synovitis due to rheumatoid arthritis. The patient complained of strong numbness from the thumb to the ring finger and pain in the palm, and he was unable to actively flex or extend his fingers. …”
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“…Osteoarthritis (OA) is characterized by progressive cartilage destruction and synovitis; however, there are no approved disease-modifying OA drugs. …”
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“…In view of the significance of CD44 in DJD pathogenesis and the potential biological role for PRG4, this review aims to summarize the involvement of PRG4-CD44 signaling in controlling synovitis, synovial hypertrophy, and tissue fibrosis in DJDs.…”
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“…Mycoplasma synoviae is a significant cause of respiratory disease and synovitis among chickens, and has an adverse economic impact on broiler breeding efforts. …”
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“…More importantly, the amelioration of the disease was associated with reversal of existing structural damage, including synovitis and periosteal bone erosions evident on histology. …”
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“…Intradermal injection of five out of eight assayed cosmetic products without further additives resulted in arthritis with synovitis. One of the products induced a very aggressive arthritis, which had declined after 5-9 weeks. …”
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“…BACKGROUND: Familial Mediterranean fever (FMF) is an autosomal recessive disease characterized by inflammatory serositis (fever, peritonitis, synovitis and pleuritis). The gene locus responsible for FMF was identified in 1992 and localized to the short arm of chromosome 16. …”
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