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  1. 3081
    “…We also showed how these redox-based signals could be intermingled with other quorum-sensing (QS) signals which are more commonly used in synthetic biology, to control complex behaviors. …”
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    Parsed synthesis of pyocyanin via co-culture enables context-dependent intercellular redox communication
  2. 3082
    “…In this review, we introduce a new concept of the p53 protein by discussing its sensitivity to the cellular redox state. In particular, we focus on the discussion of structural and functional changes following post-translational modifications of redox-sensitive cysteine residues, which are also responsible for interacting with zinc ions for proper structural folding. …”
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    Redox Sensitive Cysteine Residues as Crucial Regulators of Wild-Type and Mutant p53 Isoforms
  3. 3083
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    Lycopene Ameliorates Liver Inflammation and Redox Status in Mice Exposed to Long-Term Cigarette Smoke
  4. 3084
    “…Among these processes are mitochondrial integrity and the associated redox environments, although their direct impact on human neurophysiological function is unknown. …”
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    Neural oscillatory activity serving sensorimotor control is predicted by superoxide-sensitive mitochondrial redox environments
  5. 3085
    “…The electronic effect of tert-butyl groups on intramolecular through-bond interactions between redox units in tetrathiafulvalene-tetraazapyrene (TAP) triads is investigated. …”
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    Effect of tert-butyl groups on electronic communication between redox units in tetrathiafulvalene-tetraazapyrene triads
  6. 3086
    “…BACKGROUND: PKARIα (protein kinase A type I‐α regulatory subunit) is redox‐active independent of its physiologic agonist cAMP. …”
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    Enhanced Heart Failure in Redox‐Dead Cys17Ser PKARIα Knock‐In Mice
  7. 3087
    “…Objectives: We aimed to appraise the current evidence around the impact of pulmonary rehabilitation on redox status, compared with other exercise interventions, to gain an understanding of optimal exercise interventions to modify this pathophysiological mechanism. …”
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    Evidence Around the Impact of Pulmonary Rehabilitation and Exercise on Redox Status in COPD: A Systematic Review
  8. 3088
    “…[Image: see text] Cross-linking poly(glycidyl methacrylate) microparticles with redox-responsive bis(5-amino-l,3,4-thiadiazol-2-yl) disulfide moieties yield redox-active particles (RAPs) capable of electrochemical energy storage via a reversible 2-electron reduction of the disulfide bond. …”
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    Synthesis and Characterization of Redox-Responsive Disulfide Cross-Linked Polymer Particles for Energy Storage Applications
  9. 3089
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    Commentary on NNT Mediates Redox-Dependent Pigmentation via a UVB-And MITF-Independent Mechanism
  10. 3090
    “…Here, we demonstrate that the NP drug delivery vehicle poly(butyl cyanoacrylate) (PBCA) perturbs redox homeostasis in human epithelial cells, and that the degree of redox perturbation dictates divergent effects of PBCA on autophagy. …”
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    Perturbation of Cellular Redox Homeostasis Dictates Divergent Effects of Polybutyl Cyanoacrylate (PBCA) Nanoparticles on Autophagy
  11. 3091
    “…A redox-responsive nanocarrier is a promising strategy for the intracellular drug release because it protects the payload, prevents its undesirable leakage during extracellular transport, and favors site-specific drug delivery. …”
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    Dually Cross-Linked Core-Shell Structure Nanohydrogel with Redox–Responsive Degradability for Intracellular Delivery
  12. 3092
    “…Moreover, cysteine residues in mammalian VDAC paralogs may contribute to the reduction–oxidation (redox) sensor function based on disulfide bond formation and elimination, resulting in redox-sensitive VDAC (rsVDAC). …”
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    Redox-Sensitive VDAC: A Possible Function as an Environmental Stress Sensor Revealed by Bioinformatic Analysis
  13. 3093
    “…Our work highlights an important interplay between the TCA cycle, redox biology, and amino acid homeostasis.…”
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    Disruption of the TCA cycle reveals an ATF4-dependent integration of redox and amino acid metabolism
  14. 3094
    “…Human peroxiredoxin-5 (PRDX5) is a unique redox-sensitive protein that plays a dual role in brain ischemia-reperfusion injury. …”
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    Role of the Redox State of Human Peroxiredoxin-5 on Its TLR4-Activating DAMP Function
  15. 3095
    “…This study investigates the catechin composition and protective effect of green tea extract on senescence-mediated redox imbalance in the livers and kidneys of aged mice. …”
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    Senescence-Mediated Redox Imbalance in Liver and Kidney: Antioxidant Rejuvenating Potential of Green Tea Extract
  16. 3096
    “…Disrupted redox homeostasis was confirmed by dynamic live imaging of the glutathione redox potential using a genetically encoded redox-sensitive fluorescence-based biosensor. …”
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    Peroxide Antimalarial Drugs Target Redox Homeostasis in Plasmodium falciparum Infected Red Blood Cells
  17. 3097
    “…Most existing methodologies focus only on the GSH/GSH disulfide (GSSG) redox couple, but GSH regulation is highly complex and depends on several pathways with multiple redox-active sulfur-containing species. …”
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    Extensive Thiol Profiling for Assessment of Intracellular Redox Status in Cultured Cells by HPLC-MS/MS
  18. 3098
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    Reactions of Recombinant Neuronal Nitric Oxide Synthase with Redox Cycling Xenobiotics: A Mechanistic Study
  19. 3099
    por Ren, Jie, Wang, Aman, Liu, Jiwei, Yuan, Qihang
    Publicado 2021
    “…The aim of our research is to identify a novel redox-related lncRNA prognostic signature (redox-LPS) for better prediction of LUAD prognosis. 535 LUAD samples from The Cancer Genome Atlas (TCGA) database and 226 LUAD samples from the Gene Expression Omnibus (GEO) database were included in our study. 67 redox genes and 313 redox-related lncRNAs were identified. …”
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    Identification and validation of a novel redox-related lncRNA prognostic signature in lung adenocarcinoma
  20. 3100
    “…Inhibition of USP15 and subsequent activation of NRF2 leads to redox perturbations in AML cells, coincident with impaired leukemic cell function. …”
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    The deubiquitinase USP15 modulates cellular redox and is a therapeutic target in acute myeloid leukemia
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