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  1. 3441
    “…Thus, by demonstrating that GSH-depleting agents enhance the antitumor activity of AUR, we highlight new treatment options for RMS by targeting the redox homeostasis.…”
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    Targeting redox homeostasis in rhabdomyosarcoma cells: GSH-depleting agents enhance auranofin-induced cell death
  2. 3442
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    Redox Active Transition Metal ions Make Melanin Susceptible to Chemical Degradation Induced by Organic Peroxide
  3. 3443
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    Redox Regulation of a Gain-of-Function Mutation (N308D) in SHP2 Noonan Syndrome
  4. 3444
    “…RESULTS: Kinetic analysis shows that purified human MTHFD2 exhibits dual redox cofactor specificity, utilizing either NADP(+) or NAD(+) with the more physiologically relevant pentaglutamate folate substrate. …”
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    Human mitochondrial MTHFD2 is a dual redox cofactor-specific methylenetetrahydrofolate dehydrogenase/methenyltetrahydrofolate cyclohydrolase
  5. 3445
    “…Primary and secondary hypertension is associated with kidney redox imbalance resulting in enhanced reactive oxygen species (ROS) and enzymes dependent phospholipid metabolism. …”
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    Redox system and phospholipid metabolism in the kidney of hypertensive rats after FAAH inhibitor URB597 administration
  6. 3446
    “…Lithium-rich layered transition metal oxide positive electrodes offer access to anion redox at high potentials, thereby promising high energy densities for lithium-ion batteries. …”
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    Coupling between oxygen redox and cation migration explains unusual electrochemistry in lithium-rich layered oxides
  7. 3447
    “…This composite nanosystem was prepared by modifying hollow mesoporous silica nanoparticles (HMSNs) with transferrin (Tf) targeting moieties via redox-liable linkage, and was capable of delivering therapeutic cargos (doxorubicin) specifically to the tumor site and subsequently releasing them in an on-demand manner. …”
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    A Transferrin-Conjugated Hollow Nanoplatform for Redox-Controlled and Targeted Chemotherapy of Tumor with Reduced Inflammatory Reactions
  8. 3448
    “…These observed changes in redox state were not due to abnormalities in mitochondrial mass or membrane integrity. …”
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    Elevated Mitochondrial Reactive Oxygen Species and Cellular Redox Imbalance in Human NADPH-Oxidase-Deficient Phagocytes
  9. 3449
    “…The aim of this study was A) to determine redox state and variation in basal (pre-acute stress) immune function during summer, autumn and winter (spring was not assessed due to restrictions in collecting permit) in the fish-eating Myotis (Myotis vivesi; Chiroptera), and B) to determine the effect of acute stress on immunocompetence and redox state during each season. …”
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    Baseline and post-stress seasonal changes in immunocompetence and redox state maintenance in the fishing bat Myotis vivesi
  10. 3450
    por Chan, Yin-Ching, Hwang, Juen-Haur
    Publicado 2018
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    Correction: Effects of Spirulina on the functions and redox status of auditory system in senescence-accelerated prone-8 mice
  11. 3451
    “…These fibroblasts were studied from fundamental viewpoints of growth and morphology, as well as redox, mitochondrial, and autophagic function. It was observed that fibroblasts from PD subjects had higher growth rates, and appeared distinctly different in terms of morphology and spatial organization in culture, compared to control cells. …”
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    Parkinson's Disease Skin Fibroblasts Display Signature Alterations in Growth, Redox Homeostasis, Mitochondrial Function, and Autophagy
  12. 3452
    “…Hence, we propose a universal mechanism that controls protein synthesis by inducing reversible changes in the translation machinery upon modulating the redox status of proteins involved in translation. …”
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    Quantitative proteomics identifies redox switches for global translation modulation by mitochondrially produced reactive oxygen species
  13. 3453
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    Corrigendum: The malaria parasite egress protease SUB1 is a calcium-dependent redox switch subtilisin
  14. 3454
    “…Here, we use rotational-echo double-resonance (REDOR) solid-state NMR to determine the conformations of PKC modulators bound to the PKCδ-C1b domain in the presence of phospholipid vesicles. …”
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    REDOR NMR Reveals Multiple Conformers for a Protein Kinase C Ligand in a Membrane Environment
  15. 3455
    “…Mitochondrial NADP(+)-dependent isocitrate dehydrogenase (idh2) regulates mitochondrial redox balance and reduces oxidative stress-induced cell injury via generation of NADPH. …”
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    Idh2 Deficiency Exacerbates Acrolein-Induced Lung Injury through Mitochondrial Redox Environment Deterioration
  16. 3456
    “…CONCLUSION: Our results point toward a different redox, lipid metabolism, and EV profile in the G6PD(−) FFP units. …”
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    Redox Status, Procoagulant Activity, and Metabolome of Fresh Frozen Plasma in Glucose 6-Phosphate Dehydrogenase Deficiency
  17. 3457
    “…Bru was loaded in micelles (Bru/SS-M) with high loading efficiency, narrow size distribution, and excellent storage stability. The redox-sensitive Bru/SS-M exhibited rapid particle dissociation and drug release in response to a redox environment. …”
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    Redox-sensitive micelles composed of disulfide-linked Pluronic-linoleic acid for enhanced anticancer efficiency of brusatol
  18. 3458
    “…Diurnal oscillation of intracellular redox potential is known to couple metabolism with the circadian clock, yet the responsible mechanisms are not well understood. …”
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    NRF2 regulates core and stabilizing circadian clock loops, coupling redox and timekeeping in Mus musculus
  19. 3459
    “…Overall, they support, complement and extend original findings demonstrating that KRIT1 loss-of-function induces a redox-sensitive and JNK-dependent sustained upregulation of the master Nrf2 antioxidant defense pathway and its downstream target Glyoxalase 1 (Glo1), and a drop in intracellular levels of AP-modified Hsp70 and Hsp27 proteins, leading to a chronic adaptive redox homeostasis that sensitizes cells to oxidative DNA damage and apoptosis. …”
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    Data in support of sustained upregulation of adaptive redox homeostasis mechanisms caused by KRIT1 loss-of-function
  20. 3460
    “…Moreover, by performing protein profiling on isolated astrocytes we showed that an increase in astrocytic DJ-1 expression up-regulated a large group of proteins associated with redox regulation, inflammation and mitochondrial respiration. …”
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    Astroglial DJ-1 over-expression up-regulates proteins involved in redox regulation and is neuroprotective in vivo
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