Mostrando 381 - 400 Resultados de 7,900 Para Buscar 'Redfoo~', tiempo de consulta: 2.32s Limitar resultados
  1. 381
    “…Although no single pathway can be identified as the most crucial factor that causes sarcopenia, age-associated dysregulation of redox signaling appears to underlie many deteriorations at physiological, subcellular, and molecular levels. …”
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  2. 382
    “…The unique Mg(2+) storage mechanism involves the dissociation/formation of S–S bonds and changes in the coordination number of Ti, namely, a mixture of conversion and intercalation reaction, accompanied by the joint cationic (Ti) and anionic (S) redox-rich chemistry. Our proposed amorphous and redox-rich design philosophy might provide an innovative direction for developing high-performance cathode materials for multivalent-ion batteries.…”
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  3. 383
    “…Cells metabolize nutrients through a complex metabolic and signaling network that governs redox homeostasis. At the core of this, redox regulatory network is a mutually inhibitory relationship between reduced glutathione and reactive oxygen species (ROS)—two opposing metabolites that are linked to upstream nutrient metabolic pathways (glucose, cysteine, and glutamine) and downstream feedback loops of signaling pathways (calcium and NADPH oxidase). …”
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  4. 384
    “…In this review, we describe the range of chemical and biological tools and techniques currently available for ROS and redox marker measurement in plant cells and tissues. …”
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  5. 385
    “…We review the changes in redox regulation that cancer cells undergo during metastasis. …”
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  6. 386
    “…Therefore, doses below the region of adverse responses are of particular interest and are specified as the hormetic gain zone. The manifestation of redox adaptations aiming to prevent from disturbances of redox homeostasis represent an area of particular interest in hormetic responses, observed after exposure not only to stressors but also to compounds of natural origin, such as phytochemicals. …”
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  7. 387
    “…Non-erythrocytic globins have been implicated in circulatory control through reactions that couple nitric oxide (NO) signaling with cellular oxygen availability and redox status. Small artery endothelial cells (ECs) express free α-globin, which causes vasoconstriction by degrading NO. …”
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  8. 388
    “…[Image: see text] We demonstrate that allosteric effects and redox state changes can be harnessed to create a switch that selectively and reversibly regulates the coordination chemistry of a single site on the surface of a molecular cluster. …”
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  9. 389
    “…The present review focuses on the major causes of central nervous system (CNS) redox homeostasis imbalance comprising mitochondrial dysfunction and endoplasmic reticulum (ER) stress. …”
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  10. 390
    “…Furthermore, the electrochemical and chemical redox behaviors of hypocrellins and hypomycins were investigated under aerobic and anaerobic conditions. …”
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  11. 391
    “…Aqueous and non-aqueous redox flow batteries (RFBs) have limited energy and current densities, respectively, due to the nature of the electrolytes. …”
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  12. 392
  13. 393
    “…Redox biology is at the core of life sciences, accompanied by the close correlation of redox processes with biological activities. …”
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  14. 394
  15. 395
    “…Herein we report a process for intramolecular hydroamination that uses a redox-enabled strategy relying on efficient in situ generation of hydroxylamines by oxidation, followed by Cope-type hydroamination, then reduction of the resulting pyrrolidine N-oxide. …”
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  16. 396
  17. 397
    “…In particular, ROS can either function as signaling molecules that shape physiological cell functions, or act as deleterious end products of unbalanced redox reactions. Indeed, cellular redox status needs to be tightly regulated to ensure proper cellular functioning, and either excessive ROS accumulation or the dysfunction of antioxidant systems can perturb the redox homeostasis, leading to supraphysiological concentrations of ROS and potentially harmful outcomes. …”
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  18. 398
    “…The rhodium complex was tested in the [3+2] cycloaddition of diphenylcyclopropenone and methylphenylacetylene, where it displayed a redox‐switchable behavior. The neutral complex showed moderate activity, which was suppressed when the catalyst was reduced.…”
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  19. 399
    “…The goal of the paper was to discuss the issue of redox equilibrium in the human organism in the course of cardiovascular diseases to systemize updated knowledge in the context of exercise impacts on the organism. …”
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  20. 400
    “…In our work, we discuss redox homeostasis and salivary antioxidant activity during healthy periods and in periods of disease: dental carries, oral cavity cancer, periodontal diseases, cardiovascular diseases, diabetes mellitus, systemic sclerosis, and pancreatitis. …”
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