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  1. 4321
  2. 4322
    “…This resulted in the identification of up to 1248 proximal cysteines, which could sense the redox state of the SNO site, opening new perspectives on the biological effects of redox switches. …”
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  3. 4323
    “…The effects of obesity management via sleeve gastrectomy (SG) and lifestyle intervention (LS) on inflammatory cytokines, redox status, and CVD risk were studied in this work. …”
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  4. 4324
    “…Here, the reversible six-electron redox electrochemistry is constructed by activating the sulfur oxidation reaction (SOR) process of the elaborate mesocrystal NiS(2) (M-NiS(2)). …”
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  5. 4325
    “…Immuno spin-trapping showed increased radicals formed on the filamentous actin of PrP(-/-) and PrP(GPI-/-) slices, but not WT and PrP(+/-) slices, post-recovery suggesting ongoing dysregulation of redox balance in the slices lacking GPI-anchored PrP(C). …”
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  6. 4326
  7. 4327
  8. 4328
    “…The aim of this study is to explore the putative influence of “golden tomato” (GT) on Metabolic Syndrome (MetS), especially focusing on the effects on redox homeostasis. Firstly, the differential chemical properties of the GT food matrix were characterized in terms of phytonutrient composition and antioxidant capacities with respect to red tomato (RT). …”
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  9. 4329
    “…Disturbances in the intracellular redox environment resulting from various diseases and aging interfere with proper calcium signaling, however, the details are unclear. …”
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  10. 4330
  11. 4331
    “…Purpose: To investigate the neuroprotective role of ZLHXTY capsules for ICH-induced oxidative injury through the regulation of redox imbalance with the Nrf2 signaling pathway. …”
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  12. 4332
    “…CONCLUSIONS: Oral exposure to DM in mice induces behavioural deficits, mPFC and hippocampal neuronal degenerations via redox imbalance in the brain of mice. These observations confirm the neurotoxicity of DM extracts and raises concerns on the safety and potential adverse effects of DM in humans. …”
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  13. 4333
    “…Notably, cells lacking TRR1 and HSP42 exhibited severe synthetic slow growth exacerbated by oxidative stress, signifying a critical role for Hsp42 under redox-challenged conditions. Finally, we demonstrated that Hsp42 localization patterns in trr1∆ cells mimic those observed in chronically aging and glucose-starved cells, linking nutrient depletion and redox imbalance with management of misfolded proteins via a mechanism of long-term sequestration.…”
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  14. 4334
  15. 4335
    “…The influence of electrolyte velocity over the ion-exchange membrane surface on ion and vanadium redox batteries’ conductivity was formalized and quantified. …”
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  16. 4336
    “…OBJECTIVES: This study explored the ability of human epicardial adipose tissue (EAT)-derived microRNAs (miRNAs) to regulate the myocardial redox state and clinical outcomes. METHODS: This study screened for miRNAs expressed and released from human EAT and tested for correlations with the redox state in the adjacent myocardium in paired EAT/atrial biopsy specimens from patients undergoing cardiac surgery. …”
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  17. 4337
    “…The Au@AgNPs coated with carbimazole showed a highly selective reaction to Cr(6+) in tea samples through a redox reaction between Cr(6+) and carbimazole. The Cr(6+) in the contaminated tea sample reacted with methimazole—the hydrolysate of carbimazole—to form disulfide, which led to the decrease in the Raman intensity of the peak at 595 cm(−1). …”
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  18. 4338
    “…Besides demonstrating the technical feasibility of isolating plastids in a tissue-specific manner, our work provides strong evidence that plastids from vascular tissue have a higher redox turnover to ensure optimal functioning, notably under high solute strength as encountered in vascular cells.…”
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  19. 4339
    “…NADPH provides the indispensable reducing power to regenerate the reduced glutathione to maintain cellular redox equilibria. In mammalian cells, pentose phosphate pathway (PPP) is the major route to produce NADPH by using glycolytic intermediates, and the rate-limiting step of PPP is controlled by glucose-6-phosphate dehydrogenase (G6PD). …”
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  20. 4340
    “…We were the first to evaluate the effect of hard-anodized (type II) Ti-6Al-4V alloy discs on the cytotoxicity, mitochondrial function, and redox balance of fibroblasts mitochondria compared to standard-anodized (type III) and non-anodized discs. …”
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