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  1. 1581
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    Quantitative High-Throughput Screen Identifies Inhibitors of the Schistosoma mansoni Redox Cascade
  2. 1582
    por Gu, Chunmei, Shi, Yonghui, Le, Guowei
    Publicado 2008
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    Effect of Dietary Protein Level and Origin on the Redox Status in the Digestive Tract of Mice
  3. 1583
    “…These results define a novel redox-based mechanism to propagate Fas-dependent apoptosis.…”
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    Redox amplification of apoptosis by caspase-dependent cleavage of glutaredoxin 1 and S-glutathionylation of Fas
  4. 1584
    “…BACKGROUND: Cysteine (Cys) and its disulfide, cystine (CySS) represent the major extracellular thiol/disulfide redox control system. The redox potential (E(h)) of Cys/CySS is centered at approximately −80 mV in the plasma of healthy adults, and oxidation of E(h) Cys/CySS is implicated in inflammation associated with various diseases. …”
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    Cysteine Redox Potential Determines Pro-Inflammatory IL-1β Levels
  5. 1585
    “…Nrf2 plays pivotal roles in coordinating the antioxidant response and maintaining redox homeostasis. Nrf2 expression is exquisitely regulated; Nrf2 expression is suppressed under unstressed conditions but strikingly induced under oxidative stress. …”
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    An internal ribosomal entry site mediates redox-sensitive translation of Nrf2
  6. 1586
    “…We show that intraperoxisomal redox status is strongly influenced by environmental growth conditions. …”
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    Intraperoxisomal redox balance in mammalian cells: oxidative stress and interorganellar cross-talk
  7. 1587
    “…Our data suggests AtNUDT7 plays an important role in maintaining redox homeostasis, particularly for maintaining NADH:NAD(+ )balance for normal growth and development. …”
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    Modulation of redox homeostasis under suboptimal conditions by Arabidopsis nudix hydrolase 7
  8. 1588
    “…This proof-of-principle study applied and evaluated redox lipidomic approaches for the characterization and profiling of selected lipids and their oxidation products in human hair. …”
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    MALDI-MS Redox Lipidomics Applied to Human Hair: A First Look
  9. 1589
    “…Redox status affects various cellular activities, such as proliferation, differentiation, and death. …”
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    Unveiling the roles of the glutathione redox system in vivo by analyzing genetically modified mice
  10. 1590
    “…A central focus of research on PNSB dealt with the elucidation of mechanisms by which they manage to balance cellular redox under diverse conditions, in particular under photoheterotrophic growth. …”
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    Metabolic network modeling of redox balancing and biohydrogen production in purple nonsulfur bacteria
  11. 1591
    “…Here we report the formation of supramolecular hydrogels and their redox-responsive and self-healing properties due to host–guest interactions. …”
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    Redox-responsive self-healing materials formed from host–guest polymers
  12. 1592
    “…BACKGROUND: Redox cycling compounds have been reported to cause false positive inhibition of proteases in drug discovery studies. …”
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    Redox-Based Inactivation of Cysteine Cathepsins by Compounds Containing the 4-Aminophenol Moiety
  13. 1593
    “…We concluded that redox-potent compounds, which target the antioxidant system in fungi, possess a chemosensitizing capacity to enhance efficacy of conventional drugs.…”
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    Targeting the Oxidative Stress Response System of Fungi with Redox-Potent Chemosensitizing Agents
  14. 1594
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    Redox Transformations of Iron at Extremely Low pH: Fundamental and Applied Aspects
  15. 1595
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    Redox regulation of responses to hypoxia and NO-cGMP signaling in pulmonary vascular pathophysiology
  16. 1596
    “…Our results indicate TS covalently adducts PRX3, thereby disabling a major mitochondrial antioxidant network that counters chronic mitochondrial oxidative stress. Redox-active compounds like GV that modify the TR2/TRX2 network may significantly enhance the efficacy of TS, thereby providing a combinatorial approach for exploiting redox-dependent perturbations in mitochondrial function as a therapeutic approach in mesothelioma.…”
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    Peroxiredoxin 3 Is a Redox-Dependent Target of Thiostrepton in Malignant Mesothelioma Cells
  17. 1597
    “…This paper presents a nonivasive approach to study redox state of reduced cytochromes [Image: see text], [Image: see text] and [Image: see text] of complexes II and III in mitochondria of live cardiomyocytes by means of Raman microspectroscopy. …”
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    Mapping of Redox State of Mitochondrial Cytochromes in Live Cardiomyocytes Using Raman Microspectroscopy
  18. 1598
    por Soupene, Eric, Kuypers, Frans A
    Publicado 2012
    “…CONCLUSION: Mutant forms of LPCAT1 that are not inhibited by Ca(2+) and sulfhydryl-alkylating and –oxidizing agents will provide a better understanding of the physiological function of a mechanism that places the formation of PC, and the disposal of the bioactive species lysoPC, under the control of the redox status and Ca(2+) concentration of the cell.…”
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    Phosphatidylcholine formation by LPCAT1 is regulated by Ca(2+) and the redox status of the cell
  19. 1599
    “…The amino terminal PAS1 domain of NifL from Azotobacter vinelandii accommodates a redox-active FAD group; elevation of cytosolic oxygen concentrations result in FAD oxidation and a concomitant conformational re-arrangement that is relayed via a short downstream linker to a second PAS domain, PAS2. …”
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    Influence of PAS Domain Flanking Regions on Oligomerisation and Redox Signalling By NifL
  20. 1600
    “…Current evidence indicates the therapeutic potential of targeting the prooxidant shift in redox homeostasis for the treatment of age-related diseases associated with myofibroblast dysregulation.…”
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    Therapeutic Targeting of Redox Signaling in Myofibroblast Differentiation and Age-Related Fibrotic Disease
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