Mostrando 1,561 - 1,580 Resultados de 13,225 Para Buscar 'Tranqueras~', tiempo de consulta: 4.42s Limitar resultados
  1. 1561
  2. 1562
  3. 1563
    “…Strain differences in susceptibility to promotion by the liver carcinogens 2‐acetylaminofluorene (2‐AAF) and phenobarbital (PB) were examined in the medium‐term bioassay system initially developed in our laboratory using male F344 rats as the test animal and glutathione S‐transferase placental form (GST‐P)‐positive foci as the lesion end‐point. …”
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  4. 1564
    “…The expression of glutathione S‐transferase (GST)‐π and four oncogene products, c‐Jun, c‐Fos, c‐H‐Ras, and c‐Myc, in human squamons cell carcinomas of the head and neck was investigated immunohistochemically before and after radiation therapy, to examine whether these oncogene products might be involved in GST‐π expression, and also to examine the relationship between their expression and therapeutic response. …”
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  5. 1565
  6. 1566
  7. 1567
  8. 1568
    “…Functional paralogous ABO, GBGT1, A3GALT2, and GGTA1 genes encode blood group A and B transferases (AT and BT), Forssman glycolipid synthase (FS), isoglobotriaosylceramide synthase (iGb3S), and α1,3-galactosyltransferase (GT), respectively. …”
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  9. 1569
    “…In this sense, Glutathione-S-transferase pi (GSTpi), an enzyme traditionally linked to phase II detoxification, is highly expressed in human lung epithelial cells, which represent the first line of defence against aeroallergens. …”
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  10. 1570
  11. 1571
  12. 1572
    “…We postulate that up-regulation of glutathione-S-transferase P1 (GSTP1) mediated by the transcription factor Nrf2 could be associated with the antioxidant effect of SDX on vascular endothelial cells. …”
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  13. 1573
    “…Background and Aims: Increased O-GlcNAc transferase (OGT)–induced O-linked N-acetylglucosamine (O-GlcNAc) post-translational modification is linked with diabetic complications. …”
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  14. 1574
  15. 1575
    “…A cluster of glutathione-S-transferase from epsilon group were found to be overexpressed in resistant populations of Anopheles funestus across Africa including GSTe1 [Cameroon (fold change, FC: 2.54), Ghana (4.20), Malawi (2.51)], GSTe2 [Cameroon (4.47), Ghana (7.52), Malawi (2.13)], GSTe3 [Cameroon (2.49), Uganda (2.60)], GSTe4 in Ghana (3.47), GSTe5 [Ghana (2.94), Malawi (2.26)], GSTe6 [Cameroun (3.0), Ghana (3.11), Malawi (3.07), Uganda (3.78)] and GSTe7 (2.39) in Ghana. …”
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  16. 1576
    “…Succinyl‐CoA:3‐ketoacid CoA transferase (SCOT) deficiency is an inherited metabolic disease caused by mutated OXCT1 gene resulting in recurrent ketoacidosis. …”
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  17. 1577
    “…The aim of our study was to assess the role of bilirubin, and the heme oxygenase 1 (HMOX1) and bilirubin UDP-glucuronosyl transferase (UGT1A1) promoter gene variants, which are involved in bilirubin homeostasis, in the NAFLD development in adult patients. …”
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  18. 1578
    “…PtrERF9 was further verified to directly and specifically bind with the promoters of glutathione S‐transferase U17 (PtrGSTU17) and ACC synthase1 (PtrACS1). …”
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  19. 1579
    “…The polymorphic loci of folate cycle genes including rs1805087 5-methylenetetrahydrofolate (MTR) and rs1979277 serine hydroxymethyl transferase (SHMT1) were examined. The study of polymorphisms was carried out through the TaqMan probe detection method using polymerase chain reaction. …”
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  20. 1580
    “…Succinyl-CoA:3-ketoacid CoA transferase (SCOT) deficiency is a rare autosomal recessive disorder that results in severe ketoacidosis due to a defect in ketone utilization. …”
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