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  1. 1461
    “…Our objective was to identify the determinants of risk perception (RP) in RA patients and predictors of their willingness to take a proposed DMARD (DMARD willingness). …”
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    Influence of patient characteristics on perceived risks and willingness to take a proposed anti-rheumatic drug
  2. 1462
    “…BACKGROUND: Maladaptive behavior has been reported as a phenotypical feature in Prader–Willi syndrome (PWS). It severely limits social adaptation and the quality of life of children and adults with the syndrome. …”
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    Behavioral profile of adults with Prader-Willi syndrome: correlations with individual and environmental variables
  3. 1463
    por Emerick, Jill E, Vogt, Karen S
    Publicado 2013
    “…Prader-Willi syndrome (PWS) is a complex genetic disorder, caused by lack of expression of genes on the paternally inherited chromosome 15q11.2-q13. …”
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    Endocrine manifestations and management of Prader-Willi syndrome
  4. 1464
    “…A more common TMA is thrombotic thrombocytopenic purpura, which is caused by the lack of normal ADAMTS-13-mediated cleavage of von Willebrand factor (VWF). We investigated whether fH interacts with VWF and affects cleavage of VWF. …”
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    The Interaction between Factor H and Von Willebrand Factor
  5. 1465
    “…In the present study, the developmental relationships between non numerical and numerical magnitude processing are examined in people with Williams syndrome (WS), a genetic disorder known to associate visuo-spatial and math learning disabilities. …”
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    Magnitude Representations in Williams Syndrome: Differential Acuity in Time, Space and Number Processing
  6. 1466
    por Kuntz-Melcavage, Kara L.
    Publicado 2009
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    Precancer: The Beginning and the End of Cancer. Edited by Jules J. Berman, MD, PhD (E-Mail: jjberman1@comcast.net) with G. William Moore, MD, PhD. Jones & Bartlett Publishers: Sudbury, MA, USA, 2009; 186 pp; 42.95$; ISBN: 9780763777845
  7. 1467
    “…In this context, low level of expression of the normally silent alleles has been considered as genetic noise although such expression has never been further studied. Prader-Willi Syndrome (PWS) is a neurodevelopmental disease involving imprinted genes, including NDN, which are only expressed from the paternally inherited allele, with the maternally inherited allele silent. …”
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    Stochastic Loss of Silencing of the Imprinted Ndn/NDN Allele, in a Mouse Model and Humans with Prader-Willi Syndrome, Has Functional Consequences
  8. 1468
    “…Launaea sarmentosa (Willd) Schultz-Bip.ex Kuntze (Asteraceae), locally known as Kulhafila in the Maldives, is a creeping herb, native to tropical Indian coastlines. …”
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    Pharmacognostical evaluation of Launaea sarmentosa (Willd.) schultz-bip.ex Kuntze root
  9. 1469
    por Begley, David J, Jones, Hazel C
    Publicado 2013
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    Michael William Blackburn Bradbury 1930–2013
  10. 1470
    “…BACKGROUND: Deletion of the Williams-Beuren syndrome (WBS) critical region (WBSCR), at 7q11.23, causes a developmental disorder commonly characterized by hypersociability and excessive talkativeness and often considered the opposite behavioral phenotype to autism. …”
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    Presence of autism, hyperserotonemia, and severe expressive language impairment in Williams-Beuren syndrome
  11. 1471
    “…As such, research has attempted to identify the developmental processes that are derailed in some neuro-developmental disorders that impact upon social functioning. Williams syndrome (WS) and autism are disorders of development that are characterized by atypical yet divergent social phenotypes and atypicalities of attention to people. …”
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    Spontaneous and cued gaze-following in autism and Williams syndrome
  12. 1472
    “…Of all participants, 94.6% were willing to pay for the vaccine with a median WTP of US$1.94. …”
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    Public Acceptance and Willingness-to-Pay for a Future Dengue Vaccine: A Community-Based Survey in Bandung, Indonesia
  13. 1473
    “…Von Willebrand factor (vWF), a hemostatic protein normally synthesized and stored by endothelial cells and platelets, has been localized beyond the endothelium in vascular disease states. …”
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    Von Willebrand Factor Inhibits Mature Smooth Muscle Gene Expression through Impairment of Notch Signaling
  14. 1474
    por Berber, Ergül
    Publicado 2012
    “…Quantitative and/or qualitative deficiency of von Willebrand factor (vWF) is associated with the most common inherited bleeding disease von Willebrand disease (vWD). vWD is a complex disease with clinical and genetic heterogeneity. …”
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    The Molecular Genetics of von Willebrand Disease
  15. 1475
    “…Objective: von Willebrand disease (vWD) is the most common hereditary bleeding disorder. …”
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    The Prevalence of von Willebrand Disease and Significance of in Vitro Bleeding Time (PFA-100) in von Willebrand Disease Screening in the İzmir Region
  16. 1476
    “…BACKGROUND AND PURPOSE: Previous studies have linked procoagulant factor VIII (F VIII) to an increased risk of venous thrombosis, whereas the relation between plasma von Willebrand factor (VWF) and venous thrombosis remains poorly understood. …”
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    Cerebral Sinus and Venous Thrombosis Associated with von Willebrand Factor, Independently of Factor VIII
  17. 1477
    “…CONCLUSION: Patients with T2DM were willing to pay for improvements in efficacy, side effects, and dosing. …”
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    Effect of pill burden on dosing preferences, willingness to pay, and likely adherence among patients with type 2 diabetes
  18. 1478
    “…This protease specifically hydrolyzes von Willebrand factor (VWF) multimers, thus causing VWF size reduction. …”
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    Proteolytic Processing of Von Willebrand Factor by Adamts13 and Leukocyte Proteases
  19. 1479
    “…The large multimeric glycoprotein Von Willebrand factor (VWF) is best known for its role in haemostasis; however in recent years other functions of VWF have been identified, indicating that this protein is involved in multiple vascular processes. …”
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    Von Willebrand Factor, Angiodysplasia and Angiogenesis
  20. 1480
    “…CONTEXT: Recombinant human GH (rhGH) therapy in Prader-Willi syndrome (PWS) has been used by the medical community and advocated by parental support groups since its approval in the United States in 2000 and in Europe in 2001. …”
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    Growth Hormone Research Society Workshop Summary: Consensus Guidelines for Recombinant Human Growth Hormone Therapy in Prader-Willi Syndrome
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