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  1. 561
    “…An extracellular, paired theta-burst stimulation paradigm induced a robust GluA1-independent form of LTP lacking the early, rapidly decaying component characteristic of LTP in wild-type mice. …”
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    Induction and expression of GluA1 (GluR-A)-independent LTP in the hippocampus
  2. 562
    “…Late-phase long term potentiation (L-LTP) is thought to be the cellular basis for long-term memory (LTM). …”
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    BDNF Facilitates L-LTP Maintenance in the Absence of Protein Synthesis through PKMζ
  3. 563
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    Hypersensitivity to fennel is frequent in peach allergic patients and LTP is a major fennel allergen
  4. 564
    “…Both fear learning and activity-induced long-term potentiation (LTP) of cortico-basolateral amygdala (BLA) synapses are impaired in ASIC1a-null mice, suggesting a critical role of ASICs in fear memory formation. …”
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    ASIC-dependent LTP at multiple glutamatergic synapses in amygdala network is required for fear memory
  5. 565
    “…Paired associative stimulation (PAS(LTP)) of the human primary motor cortex (M1) can induce LTP-like plasticity by increasing corticospinal excitability beyond the stimulation period. …”
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    Augmenting LTP-Like Plasticity in Human Motor Cortex by Spaced Paired Associative Stimulation
  6. 566
    “…Lipophorin receptor variants that do not interact with LTP cannot mediate lipid uptake, revealing an essential role of LTP in the process. …”
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    Drosophila Lipophorin Receptors Recruit the Lipoprotein LTP to the Plasma Membrane to Mediate Lipid Uptake
  7. 567
    “…We found that modulation of actin dynamics has different roles in the induction and maintenance of LTD. Inhibition of either actin depolymerisation or polymerization blocks LTD induction whereas only the inhibition of actin depolymerisation blocks LTD maintenance. …”
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    The interplay between neuronal activity and actin dynamics mimic the setting of an LTD synaptic tag
  8. 568
    por Feegrade, E. S.
    Publicado 1929
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    Results of Experiments with Crude Oil Products of the Burma Oil Co., Ltd., Rangoon: As Larvicide
  9. 569
    “…One and 4 weeks after acute systemic treatment of adult rats with MK801, a potent loss of hippocampal in vivo LTP was evident compared to vehicle-treated controls. …”
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    Changes in Neuronal Oscillations Accompany the Loss of Hippocampal LTP that Occurs in an Animal Model of Psychosis
  10. 570
    “…In GluA1 knockout mice LTP and SWM deficits could be partially recovered by transgenic re-installation of full-length GluA1 in principle forebrain neurons. …”
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    Different Forms of AMPA Receptor Mediated LTP and Their Correlation to the Spatial Working Memory Formation
  11. 571
    “…Finally, the toxic effect of both extracellular oAβ and oTau on memory and LTP is dependent upon APP since APP-KO mice were resistant to oAβ- and oTau-induced defects in spatial/associative memory and LTP. …”
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    LTP and memory impairment caused by extracellular Aβ and Tau oligomers is APP-dependent
  12. 572
    “…The polarity of excitability changes associated with induction of Long-Term synaptic Depression (LTD) in CA1 pyramidal neurons is a contentious issue. …”
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    Plasticity of intrinsic excitability during LTD is mediated by bidirectional changes in h-channel activity
  13. 573
    “…Calcium-calmodulin/dependent protein kinase II (CaMKII) plays an essential role in LTP induction, but since it has the capacity to remain persistently activated even after the decay of external stimuli it has been proposed that it can also be necessary for LTP maintenance and therefore for memory persistence. …”
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    CaMKII Requirement for in Vivo Insular Cortex LTP Maintenance and CTA Memory Persistence
  14. 574
    “…Here, we report that drebrin regulates NMDAR-dependent and group 1 metabotropic glutamate receptor (mGluR)-dependent LTD induction in the hippocampus. While LFS induced NMDAR-dependent LTD in the developing hippocampus in wild-type (WT) mice, it did not induce LTD in developing drebrin E and A double knockout (DXKO) mice, indicating that drebrin is required for NMDAR-dependent LTD. …”
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    Drebrin Isoforms Critically Regulate NMDAR- and mGluR-Dependent LTD Induction
  15. 575
    “…INTRODUCTION: Amyloid-β oligomers (AβOs) are assumed to impair the ability of learning and memory by suppressing the induction of synaptic plasticity, such as long-term potentiation (LTP) in the early stage of Alzheimer's disease. …”
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    Amyloid-β oligomers suppress subunit-specific glutamate receptor increase during LTP
  16. 576
    “…Current evidence suggests that AMPARs are inserted into the postsynaptic membrane during long-term potentiation (LTP) and are removed from the membrane during long-term depression (LTD). …”
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    Proteasomal-Mediated Degradation of AKAP150 Accompanies AMPAR Endocytosis during cLTD
  17. 577
    “…Electrophysiological findings demonstrate that superfusion of methamphetamine reduced basal synaptic transmission and HFS-induced LTP in dorsal striatal slices, and SCH23390 prevented this effect. …”
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    SCH23390 Reduces Methamphetamine Self-Administration and Prevents Methamphetamine-Induced Striatal LTD
  18. 578
    “…Hippocampal synaptic plasticity includes both long-term potentiation (LTP) and long-term depression (LTD) of synaptic strength, and has been implicated in shaping place field representations that form upon initial exposure to a novel environment. …”
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    LTD is involved in the formation and maintenance of rat hippocampal CA1 place-cell fields
  19. 579
    “…Pharmacological activation of the muscarinic receptor M1R potentiates NMDA receptor activity and induces short-term potentiation at the synapses called muscarinic LTP, mLTP. Dysfunction of cholinergic transmission has been detected in the settings of cognitive impairment and dementia. …”
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    Prolonged Systemic Inflammation Alters Muscarinic Long-Term Potentiation (mLTP) in the Hippocampus
  20. 580
    “…BACKGROUND: Status epilepticus (SE) leads to memory impairment following a seizure, attributed to long-term potentiation (LTP) reduction. Thrombin, a coagulation factor that activates protease-activated receptor 1 (PAR1) is involved in cognitive impairment following traumatic brain injury by reducing hippocampal LTP and in seizures as seen in a SE pilocarpine-induced mice model. …”
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    Modulation of the Thrombin Pathway Restores LTP in a Pilocarpine Mice Model of Status Epilepticus
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