Isogenic hiPSC models of Turner syndrome reveal shared roles of inactive X and Y in the human cranial neural crest network

Modeling the developmental etiology of viable human aneuploidies can be challenging in rodent models, where synteny with human chromosomes is affected, or primate-specific biology is implicated. In humans, monosomy-X (45,X) causes Turner syndrome (TS), altering craniofacial, skeletal, endocrine, and...

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Detalles Bibliográficos
Autores principales: Ahern, Darcy T., Bansal, Prakhar, Faustino, Isaac V., Glatt-Deeley, Heather R., Kondaveeti, Yuvabharath, Banda, Erin C., Pinter, Stefan F.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Cold Spring Harbor Laboratory 2023
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC10028916/
https://www.ncbi.nlm.nih.gov/pubmed/36945647
http://dx.doi.org/10.1101/2023.03.08.531747

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC10028916/
https://www.ncbi.nlm.nih.gov/pubmed/36945647
http://dx.doi.org/10.1101/2023.03.08.531747

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