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ATM–dependent phosphorylation of CHD7 regulates morphogenesis-coupled DSB stress response in fetal radiation exposure

Following radiation exposure, unrepaired DNA double-strand breaks (DSBs) persist to some extent in a subset of cells as residual damage; they can exert adverse effects, including late-onset diseases. In search of the factor(s) that characterize(s) cells bearing such damage, we discovered ataxia-tela...

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Detalles Bibliográficos
Autores principales: Noda, Asao, Muramoto, Kaori, Mishima, Shuji
Formato: Online Artículo Texto
Lenguaje:English
Publicado: The American Society for Cell Biology 2023
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC10162424/
https://www.ncbi.nlm.nih.gov/pubmed/36884301
http://dx.doi.org/10.1091/mbc.E22-10-0450