AT 1 inhibition mediated neuroprotection after experimental traumatic brain injury is dependent on neutrophils in male mice

After traumatic brain injury (TBI) cerebral inflammation with invasion of neutrophils and lymphocytes is a crucial factor in the process of secondary brain damage. In TBI the intrinsic renin-angiotensin system is an important mediator of cerebral inflammation, as inhibition of the angiotensin II rec...

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Detalles Bibliográficos
Autores principales: Timaru-Kast, Ralph, Coronel-Castello, Shila P., Krämer, Tobias J., Hugonnet, André V., Schäfer, Michael K. E., Sebastiani, Anne, Thal, Serge C.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Nature Publishing Group UK 2023
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC10164737/
https://www.ncbi.nlm.nih.gov/pubmed/37150755
http://dx.doi.org/10.1038/s41598-023-33797-6

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC10164737/
https://www.ncbi.nlm.nih.gov/pubmed/37150755
http://dx.doi.org/10.1038/s41598-023-33797-6

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