Cardiolipin externalization mediates prion protein (PrP) peptide 106–126-associated mitophagy and mitochondrial dysfunction

Proper mitochondrial performance is imperative for the maintenance of normal neuronal function to prevent the development of neurodegenerative diseases. Persistent accumulation of damaged mitochondria plays a role in prion disease pathogenesis, which involves a chain of events that culminate in the...

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Detalles Bibliográficos
Autores principales: Yang, Dongming, Li, Jie, Li, Zhiping, Zhao, Mengyang, Wang, Dongdong, Sun, Zhixin, Wen, Pei, Gou, Fengting, Dai, Yuexin, Ji, Yilan, Li, Wen, Zhao, Deming, Yang, Lifeng
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Frontiers Media S.A. 2023
Materias:
Molecular Neuroscience
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC10272765/
https://www.ncbi.nlm.nih.gov/pubmed/37333615
http://dx.doi.org/10.3389/fnmol.2023.1163981

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC10272765/
https://www.ncbi.nlm.nih.gov/pubmed/37333615
http://dx.doi.org/10.3389/fnmol.2023.1163981

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