SHIP1 modulates antimalarial immunity by bridging the crosstalk between type I IFN signaling and autophagy

Stringent control of the type I interferon (IFN-I) signaling is critical for host immune defense against infectious diseases, yet the molecular mechanisms that regulate this pathway remain elusive. Here, we show that Src homology 2 containing inositol phosphatase 1 (SHIP1) suppresses IFN-I signaling...

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Detalles Bibliográficos
Autores principales: Li, Hongyu, Yang, Shuai, Zeng, Ke, Guo, Jiayin, Wu, Jian, Jiang, Huaji, Xie, Yingchao, Hu, Zhiqiang, Lu, Jiansen, Yang, Jianwu, Su, Xin-zhuan, Cui, Jun, Yu, Xiao
Formato: Online Artículo Texto
Lenguaje:English
Publicado: American Society for Microbiology 2023
Materias:
Research Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC10470592/
https://www.ncbi.nlm.nih.gov/pubmed/37366613
http://dx.doi.org/10.1128/mbio.03512-22

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC10470592/
https://www.ncbi.nlm.nih.gov/pubmed/37366613
http://dx.doi.org/10.1128/mbio.03512-22

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